Synaptic NMDA receptor activity boosts intrinsic antioxidant defenses
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P. Ghazal | G. McKenzie | R. Corriveau | K. Dakin | Francesc X. Soriano | G. Hardingham | B. Yankner | A. Kaindl | D. Wyllie | J. Fowler | K. Horsburgh | C. Ikonomidou | S. Papadia | F. Léveillé | M. Craigon | V. Stefovska | M. Sifringer | H. H. Hansen | Marc‐André Martel | Jill H. Fowler
[1] G. Hardingham,et al. The Dichotomy of NMDA Receptor Signaling , 2007, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.
[2] S. Lipton,et al. S-nitrosylation of peroxiredoxin 2 promotes oxidative stress-induced neuronal cell death in Parkinson's disease , 2007, Proceedings of the National Academy of Sciences.
[3] N. Knuckey,et al. Peroxiredoxin 2 overexpression protects cortical neuronal cultures from ischemic and oxidative injury but not glutamate excitotoxicity, whereas Cu/Zn superoxide dismutase 1 overexpression protects only against oxidative injury , 2007, Journal of neuroscience research.
[4] S. Rhee,et al. Sulfiredoxin, the cysteine sulfinic acid reductase specific to 2-Cys peroxiredoxin: its discovery, mechanism of action, and biological significance. , 2007, Kidney international. Supplement.
[5] David S. Park,et al. Role of Cdk5-Mediated Phosphorylation of Prx2 in MPTP Toxicity and Parkinson's Disease , 2007, Neuron.
[6] Margaret A. Taylor,et al. Interaction of amyloid binding alcohol dehydrogenase/Aβ mediates up-regulation of peroxiredoxin II in the brains of Alzheimer’s disease patients and a transgenic Alzheimer’s disease mouse model , 2007, Molecular and Cellular Neuroscience.
[7] C. Winterbourn,et al. The High Reactivity of Peroxiredoxin 2 with H2O2 Is Not Reflected in Its Reaction with Other Oxidants and Thiol Reagents* , 2007, Journal of Biological Chemistry.
[8] Ann Marie Craig,et al. NMDA Receptor Subunits Have Differential Roles in Mediating Excitotoxic Neuronal Death Both In Vitro and In Vivo , 2007, The Journal of Neuroscience.
[9] Hilmar Bading,et al. Decoding NMDA Receptor Signaling: Identification of Genomic Programs Specifying Neuronal Survival and Death , 2007, Neuron.
[10] L. Netto,et al. Reactions of yeast thioredoxin peroxidases I and II with hydrogen peroxide and peroxynitrite: rate constants by competitive kinetics. , 2007, Free radical biology & medicine.
[11] Fred H. Gage,et al. NMDA-receptor-mediated, cell-specific integration of new neurons in adult dentate gyrus , 2006, Nature.
[12] J. Klose,et al. Acute and long-term proteome changes induced by oxidative stress in the developing brain , 2006, Cell Death and Differentiation.
[13] S. Lipton,et al. The chemical biology of clinically tolerated NMDA receptor antagonists , 2006, Journal of neurochemistry.
[14] Barry Halliwell,et al. Oxidative stress and neurodegeneration: where are we now? , 2006, Journal of neurochemistry.
[15] Changlian Zhu,et al. Neuroprotective properties of memantine in different in vitro and in vivo models of excitotoxicity , 2006, The European journal of neuroscience.
[16] N. Hardingham,et al. Preconditioning Doses of NMDA Promote Neuroprotection by Enhancing Neuronal Excitability , 2006, The Journal of Neuroscience.
[17] K. Muir. Glutamate-based therapeutic approaches: clinical trials with NMDA antagonists. , 2006, Current opinion in pharmacology.
[18] H. Masutani,et al. The Involvement of Thioredoxin and Thioredoxin Binding Protein‐2 on Cellular Proliferation and Aging Process , 2005, Annals of the New York Academy of Sciences.
[19] P. Mecocci,et al. Oxidative stress in brain aging, neurodegenerative and vascular diseases: an overview. , 2005, Journal of chromatography. B, Analytical technologies in the biomedical and life sciences.
[20] Dae-Yeul Yu,et al. Characterization of neural cell types expressing peroxiredoxins in mouse brain , 2005, Neuroscience Letters.
[21] A. Hannan,et al. Nature, nurture and neurology: gene–environment interactions in neurodegenerative disease , 2005, The FEBS journal.
[22] A. Shalev,et al. Thioredoxin-interacting protein is stimulated by glucose through a carbohydrate response element and induces beta-cell apoptosis. , 2005, Endocrinology.
[23] N. Hardingham,et al. Nuclear Ca2+ and the cAMP Response Element-Binding Protein Family Mediate a Late Phase of Activity-Dependent Neuroprotection , 2005, The Journal of Neuroscience.
[24] Takahisa Taguchi,et al. Diminished Neuronal Activity Increases Neuron-Neuron Connectivity Underlying Silent Synapse Formation and the Rapid Conversion of Silent to Functional Synapses , 2005, The Journal of Neuroscience.
[25] G. McKenzie,et al. Nuclear Ca2+ and CaM kinase IV specify hormonal‐ and Notch‐responsiveness , 2005, Journal of neurochemistry.
[26] S. Rhee,et al. Reduction of Cysteine Sulfinic Acid by Sulfiredoxin Is Specific to 2-Cys Peroxiredoxins* , 2005, Journal of Biological Chemistry.
[27] S. Rhee,et al. Characterization of Mammalian Sulfiredoxin and Its Reactivation of Hyperoxidized Peroxiredoxin through Reduction of Cysteine Sulfinic Acid in the Active Site to Cysteine* , 2004, Journal of Biological Chemistry.
[28] R. Corriveau,et al. Pronounced Cell Death in the Absence of NMDA Receptors in the Developing Somatosensory Thalamus , 2004, The Journal of Neuroscience.
[29] Richard T. Lee,et al. Hyperglycemia Promotes Oxidative Stress through Inhibition of Thioredoxin Function by Thioredoxin-interacting Protein* , 2004, Journal of Biological Chemistry.
[30] I. Kohane,et al. Gene regulation and DNA damage in the ageing human brain , 2004, Nature.
[31] N. Plesnila,et al. Combination Therapy in Ischemic Stroke: Synergistic Neuroprotective Effects of Memantine and Clenbuterol , 2004, Stroke.
[32] Joachim Klose,et al. Mitochondrial Dysfunction and Oxidative Damage in parkin-deficient Mice* , 2004, Journal of Biological Chemistry.
[33] E. Koonin,et al. Regeneration of Peroxiredoxins by p53-Regulated Sestrins, Homologs of Bacterial AhpD , 2004, Science.
[34] W. Markesbery,et al. Incipient Alzheimer's disease: Microarray correlation analyses reveal major transcriptional and tumor suppressor responses , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[35] M. Toledano,et al. ATP-dependent reduction of cysteine–sulphinic acid by S. cerevisiae sulphiredoxin , 2003, Nature.
[36] P. Coffer,et al. FOXO transcription factors directly activate bim gene expression and promote apoptosis in sympathetic neurons , 2003, The Journal of cell biology.
[37] S. Oikawa,et al. Mitochondrial peroxiredoxin‐3 protects hippocampal neurons from excitotoxic injury in vivo , 2003, Journal of neurochemistry.
[38] G. Marfany,et al. Peroxiredoxin 2 (PRDX2), an antioxidant enzyme, is underexpressed in Down syndrome fetal brains , 2003, Cellular and Molecular Life Sciences CMLS.
[39] T. Speed,et al. Summaries of Affymetrix GeneChip probe level data. , 2003, Nucleic acids research.
[40] Terence P. Speed,et al. A comparison of normalization methods for high density oligonucleotide array data based on variance and bias , 2003, Bioinform..
[41] J. Olney,et al. Drug‐induced Apoptotic Neurodegeneration in the Developing Brain , 2002, Brain pathology.
[42] H. Bading,et al. Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways , 2002, Nature Neuroscience.
[43] Kai Stühler,et al. Genetic analysis of the mouse brain proteome , 2002, Nature Genetics.
[44] Hilmar Bading,et al. Nuclear calcium signaling controls CREB-mediated gene expression triggered by synaptic activity , 2001, Nature Neuroscience.
[45] C. Ikonomidou,et al. Neuronal death enhanced by N-methyl-D-aspartate antagonists. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[46] C. Zorumski,et al. Neural activity and survival in the developing nervous system , 2000, Molecular Neurobiology.
[47] C. Sommer,et al. Temporary changes of the AP-1 transcription factor binding activity in the gerbil hippocampus after transient global ischemia, and ischemic tolerance induction 1 1 Published on the World Wide Web on 22 June 2000. , 2000, Brain Research.
[48] P. Seeburg,et al. C-Terminal Truncation of NR2A Subunits Impairs Synaptic But Not Extrasynaptic Localization of NMDA Receptors , 2000, The Journal of Neuroscience.
[49] D. Accili,et al. Differential regulation of gene expression by insulin and IGF‐1 receptors correlates with phosphorylation of a single amino acid residue in the forkhead transcription factor FKHR , 2000, The EMBO journal.
[50] S. Lipton,et al. Neuroprotection by the NMDA receptor-associated open-channel blocker memantine in a photothrombotic model of cerebral focal ischemia in neonatal rat. , 1999, European journal of pharmacology.
[51] G. Westbrook,et al. The Incorporation of NMDA Receptors with a Distinct Subunit Composition at Nascent Hippocampal Synapses In Vitro , 1999, The Journal of Neuroscience.
[52] M. Dragunow,et al. Environmental enrichment inhibits spontaneous apoptosis, prevents seizures and is neuroprotective , 1999, Nature Medicine.
[53] M. Greenberg,et al. Akt Promotes Cell Survival by Phosphorylating and Inhibiting a Forkhead Transcription Factor , 1999, Cell.
[54] M. Manns,et al. Copyright © 1997, American Society for Microbiology CREB Controls LAP/C/EBP � Transcription , 1997 .
[55] J. Inoue,et al. MEK Kinase Is Involved in Tumor Necrosis Factor α-Induced NF-κB Activation and Degradation of IκB-α* , 1996, The Journal of Biological Chemistry.
[56] A. Shevchenko,et al. Mass spectrometric sequencing of proteins silver-stained polyacrylamide gels. , 1996, Analytical chemistry.
[57] M. Imagawa,et al. DNA Binding Specificity of the CCAAT/Enhancer-binding Protein Transcription Factor Family (*) , 1996, The Journal of Biological Chemistry.
[58] M. Olive,et al. Design of a C/EBP-specific, Dominant-negative bZIP Protein with Both Inhibitory and Gain-of-function Properties * , 1996, The Journal of Biological Chemistry.
[59] M E Greenberg,et al. Regulation of gene expression in hippocampal neurons by distinct calcium signaling pathways. , 1993, Science.
[60] M E Greenberg,et al. Stimulation of protein tyrosine phosphorylation by NMDA receptor activation , 1991, Science.
[61] C. Rossberg,et al. Neuroprotective effect of memantine demonstrated in vivo and in vitro. , 1990, European journal of pharmacology.
[62] H. J. G. GUNDERSEN,et al. Some new, simple and efficient stereological methods and their use in pathological research and diagnosis , 1988, APMIS : acta pathologica, microbiologica, et immunologica Scandinavica.
[63] A. Holmgren,et al. Rat liver thioredoxin and thioredoxin reductase: purification and characterization. , 1982, Biochemistry.
[64] P. Mahadevan,et al. An overview , 2007, Journal of Biosciences.
[65] Z. A. Wood,et al. Structure, mechanism and regulation of peroxiredoxins. , 2003, Trends in biochemical sciences.
[66] M. F. Sánchez-Fonta,et al. Peroxiredoxin 2 ( PRDX 2 ) , an antioxidant enzyme , is under-expressed in Down syndrome fetal brains , 2003 .
[67] M. Küçük,et al. Reduction of Edema and Infarction by Memantine and MK-801 After Focal Cerebral Ischaemia and Reperfusion in Rat , 2000, Acta Neurochirurgica.
[68] J. Olney,et al. Blockade of NMDA receptors and apoptotic neurodegeneration in the developing brain. , 1999, Science.
[69] R. Dempsey,et al. Protective Effects of Memantine Against Ischemia-Reperfusion Injury in Spontaneously Hypertensive Rats , 1999, Acta Neurochirurgica.
[70] M. Manns,et al. CREB Controls LAP/C/EBP b Transcription , 1997 .
[71] W. Wesemann,et al. Distribution of memantine in brain, liver, and blood of the rat. , 1982, Arzneimittel-Forschung.