Significant Role for Fas in the Pathogenesis of Autoimmune Diabetes
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L. Matis | C. Costa | Yi Wang | Xiao Su | Yamin Shen | Qile Hu | J. Kristan | Demokos Gero
[1] A. Cross,et al. Dual Role for Fas Ligand in the Initiation of and Recovery from Experimental Allergic Encephalomyelitis , 1999, The Journal of experimental medicine.
[2] T. Mak,et al. TNF receptor 1-dependent beta cell toxicity as an effector pathway in autoimmune diabetes. , 1999, Journal of immunology.
[3] T. Utsugi,et al. Perforin-independent beta-cell destruction by diabetogenic CD8(+) T lymphocytes in transgenic nonobese diabetic mice. , 1999, The Journal of clinical investigation.
[4] J. Katz,et al. In Autoimmune Diabetes the Transition from Benign to Pernicious Insulitis Requires an Islet Cell Response to Tumor Necrosis Factor α , 1999, The Journal of experimental medicine.
[5] R. Rajotte,et al. Beta-cell destruction in NOD mice correlates with Fas (CD95) expression on beta-cells and proinflammatory cytokine expression in islets. , 1999, Diabetes.
[6] Z. Wang,et al. Bisindolylmaleimide VIII facilitates Fas-mediated apoptosis and inhibits T cell-mediated autoimmune diseases , 1999, Nature Medicine.
[7] A. Strasser,et al. Mechanisms of beta cell death in diabetes: a minor role for CD95. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[8] F. Marumo,et al. The Fas/Fas ligand system is involved in the pathogenesis of autoimmune myocarditis in rats. , 1998, Journal of immunology.
[9] R. Flavell,et al. Local expression of TNFalpha in neonatal NOD mice promotes diabetes by enhancing presentation of islet antigens. , 1998, Immunity.
[10] F. Shanahan,et al. Fas ligand expression in primary colon adenocarcinomas: evidence that the Fas counterattack is a prevalent mechanism of immune evasion in human colon cancer , 1998, The Journal of pathology.
[11] P. Galle,et al. Involvement of the CD95 (APO-1/Fas) receptor and ligand system in Helicobacter pylori-induced gastric epithelial apoptosis. , 1998, The Journal of clinical investigation.
[12] W. Suarez-Pinzon,et al. Cytokines and their roles in pancreatic islet beta-cell destruction and insulin-dependent diabetes mellitus. , 1998, Biochemical pharmacology.
[13] S. Sakoda,et al. Fas has a crucial role in the progression of experimental autoimmune encephalomyelitis. , 1998, Molecular immunology.
[14] M. Monden,et al. Protection of islet allografts transplanted together with Fas ligand expressing testicular allografts , 1998, Diabetologia.
[15] H. Jun,et al. Cellular and molecular mechanisms for the initiation and progression of beta cell destruction resulting from the collaboration between macrophages and T cells. , 1998, Autoimmunity.
[16] C. Benoist,et al. Interferon-gamma impacts at multiple points during the progression of autoimmune diabetes. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[17] G. Stassi,et al. Nitric Oxide Primes Pancreatic β Cells for Fas-mediated Destruction in Insulin-dependent Diabetes Mellitus , 1997, The Journal of experimental medicine.
[18] R. Zinkernagel,et al. Reduced Incidence and Delayed Onset of Diabetes in Perforin-deficient Nonobese Diabetic Mice , 1997, The Journal of experimental medicine.
[19] A. Cross,et al. Fas and Fas ligand enhance the pathogenesis of experimental allergic encephalomyelitis, but are not essential for immune privilege in the central nervous system. , 1997, Journal of immunology.
[20] V. Kuchroo,et al. Fas- and FasL-deficient mice are resistant to induction of autoimmune encephalomyelitis. , 1997, Journal of immunology.
[21] Y. Matsuzawa,et al. Requirement of Fas for the Development of Autoimmune Diabetes in Nonobese Diabetic Mice , 1997, The Journal of experimental medicine.
[22] P. Cohen,et al. Radiation and stress-induced apoptosis: a role for Fas/Fas ligand interactions. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[23] J. Corbett,et al. Interferon-γ Increases the Sensitivity of Islets of Langerhans for Inducible Nitric-oxide Synthase Expression Induced by Interleukin 1* , 1997, The Journal of Biological Chemistry.
[24] M. Zupancic,et al. Kinetics of response in lymphoid tissues to antiretroviral therapy of HIV-1 infection. , 1997, Science.
[25] D. Allan,et al. Apoptosis Is the Mode of β-Cell Death Responsible for the Development of IDDM in the Nonobese Diabetic (NOD) Mouse , 1997, Diabetes.
[26] H. Maeda,et al. Polymorphism of murine Fas ligand that affects the biological activity. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[27] S. Nagata,et al. Essential roles of the Fas ligand in the development of hepatitis , 1997, Nature Medicine.
[28] C. Janeway,et al. The Role of Fas in Autoimmune Diabetes , 1997, Cell.
[29] C. Benoist,et al. Cell Death Mediators in Autoimmune Diabetes—No Shortage of Suspects , 1997, Cell.
[30] T. Saito,et al. Nonsense mutation at Tyr-4046 in the DNA-dependent protein kinase catalytic subunit of severe combined immune deficiency mice. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[31] M. Todaro,et al. Potential Involvement of Fas and Its Ligand in the Pathogenesis of Hashimoto's Thyroiditis , 1997, Science.
[32] M. V. von Herrath,et al. Interferon-γ Is Essential for Destruction of β Cells and Development of Insulin-dependent Diabetes Mellitus , 1997, The Journal of experimental medicine.
[33] L. Notarangelo,et al. Missense mutations in the Fas gene resulting in autoimmune lymphoproliferative syndrome: a molecular and immunological analysis. , 1997, Blood.
[34] P. Vassalli,et al. Prevention of autoimmune diabetes mellitus in NOD mice by transgenic expression of soluble tumor necrosis factor receptor p55 , 1997, European journal of immunology.
[35] M. Oldstone,et al. Interferon-g Is Essential for Destruction of b Cells and Development of Insulin-dependent Diabetes Mellitus , 1997 .
[36] S. Nagata. Fas ligand and immune evasion , 1996, Nature Medicine.
[37] K. Sullivan,et al. Fas gene mutations in the Canale-Smith syndrome, an inherited lymphoproliferative disorder associated with autoimmunity. , 1996, The New England journal of medicine.
[38] J. Tschopp,et al. Melanoma Cell Expression of Fas(Apo-1/CD95) Ligand: Implications for Tumor Immune Escape , 1996, Science.
[39] C. Janeway,et al. Local expression of transgene encoded TNF alpha in islets prevents autoimmune diabetes in nonobese diabetic (NOD) mice by preventing the development of auto-reactive islet-specific T cells , 1996, The Journal of experimental medicine.
[40] K. Yamada,et al. Mouse islet cell lysis mediated by interleukin-1-induced Fas , 1996, Diabetologia.
[41] L. Matis,et al. Treatment of experimental encephalomyelitis with a novel chimeric fusion protein of myelin basic protein and proteolipid protein. , 1996, The Journal of clinical investigation.
[42] P. Schur,et al. Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease. , 1996, The Journal of clinical investigation.
[43] F. Shanahan,et al. The Fas counterattack: Fas-mediated T cell killing by colon cancer cells expressing Fas ligand , 1996, The Journal of experimental medicine.
[44] John A Todd,et al. Genetic Analysis of Autoimmune Disease , 1996, Cell.
[45] R. Tisch,et al. Insulin-Dependent Diabetes Mellitus , 1996, Cell.
[46] M. Todaro,et al. Expression of apoptosis-inducing CD95 (Fas/Apo-1) on human beta-cells sorted by flow-cytometry and cultured in vitro. , 1995, Transplantation proceedings.
[47] D. Green,et al. Fas Ligand-Induced Apoptosis as a Mechanism of Immune Privilege , 1995, Science.
[48] P. Galle,et al. Involvement of the CD95 (APO-1/Fas) receptor and ligand in liver damage , 1995, The Journal of experimental medicine.
[49] D. Bellgrau,et al. A role for CD95 ligand in preventing graft rejection , 1995, Nature.
[50] T. Ogihara,et al. Identification of a new susceptibility locus for insulin-dependent diabetes mellitus by ancestral haplotype congenic mapping. , 1995, The Journal of clinical investigation.
[51] Warren Strober,et al. Dominant interfering fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome , 1995, Cell.
[52] S. Nagata,et al. The Fas death factor , 1995, Science.
[53] E. Lacy,et al. Massive upregulation of the Fas ligand in lpr and gld mice: implications for Fas regulation and the graft-versus-host disease-like wasting syndrome , 1995, The Journal of experimental medicine.
[54] Melinda Fitzgerald,et al. Immunol. Cell Biol. , 1995 .
[55] J. Todd,et al. Genetic control of autoimmune diabetes in the NOD mouse. , 1995, Annual review of immunology.
[56] J. Inazawa,et al. Human Fas ligand: gene structure, chromosomal location and species specificity. , 1994, International immunology.
[57] R. Tisch,et al. Effect of tumor necrosis factor alpha on insulin-dependent diabetes mellitus in NOD mice. I. The early development of autoimmunity and the diabetogenic process , 1994, The Journal of experimental medicine.
[58] D. Eizirik,et al. TNF-α and IFN-γ potentiate the deleterious effects of IL-1β on mouse pancreatic islets mainly via generation of nitric oxide , 1994 .
[59] M R Alderson,et al. The mouse Fas-ligand gene is mutated in gld mice and is part of a TNF family gene cluster. , 1994, Immunity.
[60] N. Jenkins,et al. Generalized lymphoproliferative disease in mice, caused by a point mutation in the fas ligand , 1994, Cell.
[61] M. A. Bowman,et al. Prevention of diabetes in the NOD mouse: implications for therapeutic intervention in human disease. , 1994, Immunology today.
[62] N. Copeland,et al. Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis , 1992, Nature.
[63] N. Copeland,et al. The cDNA structure, expression, and chromosomal assignment of the mouse Fas antigen. , 1992, Journal of immunology.
[64] V. Fadok,et al. Apoptosis and programmed cell death in immunity. , 1992, Annual review of immunology.
[65] K. Lafferty,et al. Involvement of O2 radicals in ‘autoimmune’ diabetes , 1989, Immunology and cell biology.
[66] L. Harrison,et al. IFN-gamma and tumor necrosis factor-alpha. Cytotoxicity to murine islets of Langerhans. , 1988, Journal of immunology.
[67] N. Sarvetnick,et al. Insulin-dependent diabetes mellitus induced in transgenic mice by ectopic expression of class II MHC and interferon-gamma , 1988, Cell.
[68] J. Nerup,et al. Human tumor necrosis factor potentiates human interleukin 1-mediated rat pancreatic beta-cell cytotoxicity. , 1987, Journal of immunology.
[69] K. Lafferty,et al. Combined Treatment With Nicotinamide and Desferrioxamine Prevents Islet Allograft Destruction in NOD Mice , 1986, Diabetes.
[70] L. Wicker,et al. Transfer of Autoimmune Diabetes Mellitus with Splenocytes from Nonobese Diabetic (NOD) Mice , 1986, Diabetes.
[71] J. Nerup,et al. Cytotoxicity of human pI 7 interleukin-1 for pancreatic islets of Langerhans. , 1986, Science.
[72] P. Kelleher,et al. Insulin-dependent? , 1985, The Lancet.