Modified autonomic regulation in mice mutated in the β4 subunit of the lh/lh calcium channel.

[1]  Hiroyuki Watanabe,et al.  Inhalation anesthesia is preferable for recording rat cardiac function using an electrocardiogram. , 2014, Biological & pharmaceutical bulletin.

[2]  William A. Catterall,et al.  Signaling complexes of voltage-gated sodium and calcium channels , 2010, Neuroscience Letters.

[3]  A. Dolphin.  Calcium channel diversity: multiple roles of calcium channel subunits , 2009, Current Opinion in Neurobiology.

[4]  M. Murakami,et al.  Modified autonomic regulation in mice with a P/Q-type calcium channel mutation. , 2009, Biochemical and biophysical research communications.

[5]  Takashi Suzuki,et al.  Modified Sympathetic Nerve System Activity with Overexpression of the Voltage-dependent Calcium Channel β3 Subunit* , 2008, Journal of Biological Chemistry.

[6]  Takashi Suzuki,et al.  Modified sympathetic regulation in N-type calcium channel null-mouse. , 2007, Biochemical and biophysical research communications.

[7]  Ming-hui Li,et al.  Structural basis of the α1–β subunit interaction of voltage-gated Ca2+ channels , 2004, Nature.

[8]  A. Hudspeth,et al.  Direct interaction with a nuclear protein and regulation of gene silencing by a variant of the Ca2+-channel β4 subunit , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[9]  C. Fletcher,et al.  Reduced Voltage Sensitivity of Activation of P/Q-Type Ca2+ Channels is Associated with the Ataxic Mouse MutationRolling Nagoya (tgrol ) , 2000, The Journal of Neuroscience.

[10]  R. Tsien,et al.  Ablation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunit. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[11]  Denise S Walker,et al.  A New β Subtype-specific Interaction in α1ASubunit Controls P/Q-type Ca2+ Channel Activation* , 1999, The Journal of Biological Chemistry.

[12]  K. Campbell,et al.  β Subunit Reshuffling Modifies N- and P/Q-Type Ca2+Channel Subunit Compositions in Lethargic Mouse Brain , 1999, Molecular and Cellular Neuroscience.

[13]  A. Dolphin,et al.  Modelling of a voltage‐dependent Ca2+ channel β subunit as a basis for understanding its functional properties , 1999, FEBS letters.

[14]  T. Copeland,et al.  Altered Expression and Assembly of N-type Calcium Channel α1B and β Subunits in Epileptic lethargic(lh/lh) Mouse* , 1998, The Journal of Biological Chemistry.

[15]  M. Meisler,et al.  Mutation of the Ca2+ Channel β Subunit Gene Cchb4 Is Associated with Ataxia and Seizures in the Lethargic (lh) Mouse , 1997, Cell.

[16]  Richard Hawkes,et al.  Absence Epilepsy in Tottering Mutant Mice Is Associated with Calcium Channel Defects , 1996, Cell.

[17]  Dennis E Bulman,et al.  Familial Hemiplegic Migraine and Episodic Ataxia Type-2 Are Caused by Mutations in the Ca2+ Channel Gene CACNL1A4 , 1996, Cell.

[18]  K. Campbell,et al.  Identification of critical amino acids involved in α 1‐β interaction in voltage‐dependent Ca2+ channels , 1996 .

[19]  E. Perez-Reyes,et al.  Molecular diversity of Ca2+ channel beta subunits. , 1994, Biochemical Society transactions.

[20]  M. Adams,et al.  Calcium channels coupled to glutamate release identified by omega-Aga-IVA. , 1992, Science.

[21]  A. Brown,et al.  Normalization of current kinetics by interaction between the α1and β subunits of the skeletal muscle dihydropyridine-sensitive Ca2+ channel , 1991, Nature.

[22]  S. Oda.  [The observation of rolling mouse Nagoya (rol), a new neurological mutant, and its maintenance (author's transl)]. , 1973, Jikken dobutsu. Experimental animals.

[23]  R Y Tsien,et al.  Calcium channels, stores, and oscillations. , 1990, Annual review of cell biology.

[24]  R. Tsien,et al.  Dominant role of N-type Ca2+ channels in evoked release of norepinephrine from sympathetic neurons. , 1988, Science.