Clinical benefits of TNF‐α inhibitors in Chinese adult patients with NLRP3‐associated autoinflammatory disease
暂无分享,去创建一个
M. Shen | Yi Wang | Mengzhu Zhao | Di Wu | Weihong Yu | N. Wu | Junke Miao
[1] R. Manna,et al. Current Therapeutic Options for the Main Monogenic Autoinflammatory Diseases and PFAPA Syndrome: Evidence-Based Approach and Proposal of a Practical Guide , 2020, Frontiers in Immunology.
[2] O. Karadag,et al. Management of Behcet's syndrome. , 2020, Rheumatology.
[3] Jing Yang,et al. USP15 potentiates NF-κB activation by differentially stabilizing TAB2 and TAB3. , 2020, The FEBS journal.
[4] A. Renieri,et al. Hints for Genetic and Clinical Differentiation of Adult-Onset Monogenic Autoinflammatory Diseases , 2019, Mediators of inflammation.
[5] Wen Zhang,et al. Phenotypes and genotypes of Chinese adult patients with systemic autoinflammatory diseases. , 2019, Seminars in arthritis and rheumatism.
[6] J. Ting,et al. The NLRP3 inflammasome: molecular activation and regulation to therapeutics , 2019, Nature Reviews Immunology.
[7] G. Konstantopoulou,et al. Biologics in SAPHO syndrome: A systematic review. , 2019, Seminars in arthritis and rheumatism.
[8] I. Meyts,et al. Deficiency of Adenosine Deaminase 2 (DADA2): Updates on the Phenotype, Genetics, Pathogenesis, and Treatment , 2018, Journal of Clinical Immunology.
[9] I. Touitou,et al. ‘A20 haploinsufficiency (HA20): clinical phenotypes and disease course of patients with a newly recognised NF-kB-mediated autoinflammatory disease’ , 2018, Annals of the rheumatic diseases.
[10] L. Joosten,et al. OLT1177, a β-sulfonyl nitrile compound, safe in humans, inhibits the NLRP3 inflammasome and reverses the metabolic cost of inflammation , 2018, Proceedings of the National Academy of Sciences.
[11] D. Kastner,et al. A20 haploinsufficiency (HA20): clinical phenotypes and disease course of patients with a newly recognised NF-kB-mediated autoinflammatory disease , 2018, Annals of the rheumatic diseases.
[12] A. Wree,et al. TNF regulates transcription of NLRP3 inflammasome components and inflammatory molecules in cryopyrinopathies , 2017, The Journal of clinical investigation.
[13] R. Goldbach-Mansky,et al. Brief Report: Clinical and Molecular Phenotypes of Low‐Penetrance Variants of NLRP3: Diagnostic and Therapeutic Challenges , 2017, Arthritis & rheumatology.
[14] M. Shen,et al. Muckle-Wells syndrome in Chinese patients: a single center case series , 2017, Clinical Rheumatology.
[15] G. Núñez,et al. Mechanism and Regulation of NLRP3 Inflammasome Activation. , 2016, Trends in biochemical sciences.
[16] S. Ozen,et al. Diagnostic criteria for cryopyrin-associated periodic syndrome (CAPS) , 2016, Annals of the rheumatic diseases.
[17] N. Blank,et al. Real-life effectiveness of canakinumab in cryopyrin-associated periodic syndrome. , 2016, Rheumatology.
[18] I. Koné-Paut,et al. Current treatment recommendations and considerations for cryopyrin-associated periodic syndrome , 2015, Expert review of clinical immunology.
[19] J. Kuemmerle-Deschner. CAPS — pathogenesis, presentation and treatment of an autoinflammatory disease , 2015, Seminars in Immunopathology.
[20] S. Özen,et al. Treatment of autoinflammatory diseases: results from the Eurofever Registry and a literature review , 2012, Annals of the rheumatic diseases.
[21] C. Wouters,et al. An International registry on Autoinflammatory diseases: the Eurofever experience , 2012, Annals of the rheumatic diseases.
[22] A. Fabre,et al. Mutations in the autoinflammatory cryopyrin-associated periodic syndrome gene: epidemiological study and lessons from eight years of genetic analysis in France , 2010, Annals of the rheumatic diseases.
[23] A. Martini,et al. Follow-up and quality of life of patients with cryopyrin-associated periodic syndromes treated with Anakinra. , 2010, The Journal of pediatrics.
[24] P. Hawkins,et al. Use of canakinumab in the cryopyrin-associated periodic syndrome. , 2009, The New England journal of medicine.
[25] W. Dixon,et al. Drug-specific risk of tuberculosis in patients with rheumatoid arthritis treated with anti-TNF therapy: results from the British Society for Rheumatology Biologics Register (BSRBR) , 2009, Annals of the rheumatic diseases.
[26] G. Núñez,et al. Cutting Edge: TNF-α Mediates Sensitization to ATP and Silica via the NLRP3 Inflammasome in the Absence of Microbial Stimulation1 , 2009, The Journal of Immunology.
[27] G. Valesini,et al. Biological and clinical effects of anti-TNFalpha treatment. , 2007, Autoimmunity reviews.
[28] Takeo Tanaka,et al. A severe case of chronic infantile neurologic, cutaneous, articular syndrome treated with biologic agents. , 2006, Arthritis and rheumatism.
[29] H. Takada,et al. Long-term safety and efficacy of canakinumab in cryopyrin-associated periodic syndrome: results from an open-label, phase III pivotal study in Japanese patients. , 2017, Clinical and experimental rheumatology.
[30] S. Al Jumaah,et al. A retrospective review of autoinflammatory diseases in Saudi children at a rheumatology clinic , 2012, Annals of Saudi medicine.