Deep phenotyping of miRNAs in exercise-induced cardiac hypertrophy and fibrosis
暂无分享,去创建一个
O. Sonmez | M. Eşrefoğlu | F. Akbaş | M. Mengi | M. Altan | Ş. Dinçer | F. Tecellioglu | G. Metin | M. Pala | Şenay Gorucu Yilmaz | A. Karabulut | Mustafa Yildiz | Meltem Kumas Kulualp
[1] C. E. Condrat,et al. miRNAs as Biomarkers in Disease: Latest Findings Regarding Their Role in Diagnosis and Prognosis , 2020, Cells.
[2] Xiaowei Wang,et al. miRDB: an online database for prediction of functional microRNA targets , 2019, Nucleic Acids Res..
[3] A. Doxakis,et al. Targeting metalloproteinases in cardiac remodeling , 2019, Journal of Cardiovascular Medicine and Cardiology.
[4] M. Care,et al. Evaluation of cardiac hypertrophy in the setting of sudden cardiac death , 2019, Forensic sciences research.
[5] C. Bouchard,et al. The Effects of Regular Exercise on Circulating Cardiovascular-related MicroRNAs , 2019, Scientific Reports.
[6] Xiaowei Wang,et al. Prediction of functional microRNA targets by integrative modeling of microRNA binding and target expression data , 2019, Genome Biology.
[7] Junjie Xiao,et al. MicroRNAs in heart and circulation during physical exercise , 2018, Journal of sport and health science.
[8] J. Sadoshima,et al. Mechanisms of physiological and pathological cardiac hypertrophy , 2018, Nature Reviews Cardiology.
[9] S. Ellefsen,et al. Adaptations to Endurance and Strength Training. , 2018, Cold Spring Harbor perspectives in medicine.
[10] L. Burrell,et al. Kruppel-Like Factor 15 Is Critical for the Development of Left Ventricular Hypertrophy , 2018, International journal of molecular sciences.
[11] P. Kohl,et al. Cardiac fibroblasts , 2018, Herzschrittmachertherapie + Elektrophysiologie.
[12] Peifeng Li,et al. MicroRNA as a Therapeutic Target in Cardiac Remodeling , 2017, BioMed research international.
[13] M. Masè,et al. Selection of reference genes is critical for miRNA expression analysis in human cardiac tissue. A focus on atrial fibrillation , 2017, Scientific Reports.
[14] Manuela Malatesta,et al. Histological and Histochemical Methods - Theory and Practice , 2016, European journal of histochemistry : EJH.
[15] M. Mohammadi,et al. Phosphorylation of GSK-3β and reduction of apoptosis as targets of troxerutin effect on reperfusion injury of diabetic myocardium. , 2015, European journal of pharmacology.
[16] A. Dávalos,et al. Circulating inflammatory miRNA signature in response to different doses of aerobic exercise. , 2015, Journal of applied physiology.
[17] B. Dieter,et al. Evidence for distinct effects of exercise in different cardiac hypertrophic disorders. , 2015, Life sciences.
[18] F. Reis,et al. Sitagliptin prevents aggravation of endocrine and exocrine pancreatic damage in the Zucker Diabetic Fatty rat - focus on amelioration of metabolic profile and tissue cytoprotective properties , 2014, Diabetology & Metabolic Syndrome.
[19] P. Thompson,et al. Rapid upregulation and clearance of distinct circulating microRNAs after prolonged aerobic exercise. , 2014, Journal of applied physiology.
[20] Andreas Krämer,et al. Causal analysis approaches in Ingenuity Pathway Analysis , 2013, Bioinform..
[21] Y. Pinto,et al. The Therapeutic Potential of miRNAs in Cardiac Fibrosis: Where Do We Stand? , 2013, Journal of Cardiovascular Translational Research.
[22] Yingtong Zeng,et al. MicroRNA-132 may play a role in coexistence of depression and cardiovascular disease: A hypothesis , 2013, Medical science monitor : international medical journal of experimental and clinical research.
[23] Howard Y. Chang,et al. Long Noncoding RNAs: Cellular Address Codes in Development and Disease , 2013, Cell.
[24] Z. Kassiri,et al. Cardiac fibroblasts, fibrosis and extracellular matrix remodeling in heart disease , 2012, Fibrogenesis & tissue repair.
[25] J. Leenders,et al. Repression of Cardiac Hypertrophy by KLF15: Underlying Mechanisms and Therapeutic Implications , 2012, PloS one.
[26] K. Chowdhury,et al. The miRNA-212/132 family regulates both cardiac hypertrophy and cardiomyocyte autophagy , 2012, Nature Communications.
[27] S. Gammeltoft,et al. Angiotensin II type 1 receptor signalling regulates microRNA differentially in cardiac fibroblasts and myocytes , 2011, British journal of pharmacology.
[28] J. Krieger,et al. Aerobic Exercise Training–Induced Left Ventricular Hypertrophy Involves Regulatory MicroRNAs, Decreased Angiotensin-Converting Enzyme-Angiotensin II, and Synergistic Regulation of Angiotensin-Converting Enzyme 2-Angiotensin (1-7) , 2011, Hypertension.
[29] M. Irigoyen,et al. MicroRNAs 29 are involved in the improvement of ventricular compliance promoted by aerobic exercise training in rats. , 2011, Physiological genomics.
[30] J. McMullen,et al. Molecular distinction between physiological and pathological cardiac hypertrophy: experimental findings and therapeutic strategies. , 2010, Pharmacology & therapeutics.
[31] V. Yang,et al. Mammalian Krüppel-like factors in health and diseases. , 2010, Physiological reviews.
[32] D. Mann,et al. Clinical applications of miRNAs in cardiac remodeling and heart failure. , 2010, Personalized medicine.
[33] E. Olson,et al. Searching for miR-acles in cardiac fibrosis. , 2009, Circulation research.
[34] W. Dillmann. Cardiac hypertrophy and thyroid hormone signaling , 2009, Heart Failure Reviews.
[35] E. Olson,et al. Toward microRNA-based therapeutics for heart disease: the sense in antisense. , 2008, Circulation research.
[36] Jeffrey E. Thatcher,et al. Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis , 2008, Proceedings of the National Academy of Sciences.
[37] Shay Artzi,et al. miRNAminer: A tool for homologous microRNA gene search , 2008, BMC Bioinformatics.
[38] Jamie K. Scott,et al. Quantitation of DNA and RNA. , 2007, CSH protocols.
[39] M. Lindsay,et al. Expression profiling in vivo demonstrates rapid changes in lung microRNA levels following lipopolysaccharide-induced inflammation but not in the anti-inflammatory action of glucocorticoids , 2007, BMC Genomics.
[40] C. Croce,et al. MicroRNA-133 controls cardiac hypertrophy , 2007, Nature Medicine.
[41] Saptarsi M. Haldar,et al. Kruppel-like factor 15 is a regulator of cardiomyocyte hypertrophy , 2007, Proceedings of the National Academy of Sciences.
[42] T. Karlsen,et al. Aerobic high-intensity intervals improve VO2max more than moderate training. , 2007, Medicine and science in sports and exercise.
[43] Danish Sayed,et al. MicroRNAs Play an Essential Role in the Development of Cardiac Hypertrophy , 2007, Circulation research.
[44] Saptarsi M. Haldar,et al. The Kruppel-like factor KLF15 inhibits connective tissue growth factor (CTGF) expression in cardiac fibroblasts. , 2006, Journal of molecular and cellular cardiology.
[45] T. Murohara,et al. Noninvasive Evaluation of the Time Course of Change in Cardiac Function in Spontaneously Hypertensive Rats by Echocardiography , 2005, Hypertension Research.
[46] J. Krieger,et al. Duration-controlled swimming exercise training induces cardiac hypertrophy in mice. , 2003, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.
[47] C. Livi,et al. HAND1 and HAND2 are expressed in the adult-rodent heart and are modulated during cardiac hypertrophy. , 2002, Biochemical and biophysical research communications.
[48] C. Otto,et al. Recommendations for quantification of Doppler echocardiography: a report from the Doppler Quantification Task Force of the Nomenclature and Standards Committee of the American Society of Echocardiography. , 2002, Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography.
[49] D. Srivastava,et al. Human eHAND, but not dHAND, is down-regulated in cardiomyopathies. , 2001, Journal of molecular and cellular cardiology.
[50] W. Roberts,et al. Relation between extent of cardiac muscle cell disorganization and left ventricular wall thickness in hypertrophic cardiomyopathy. , 1992, The American journal of cardiology.
[51] G. de Simone,et al. Echocardiographic measurement of left ventricular mass and volume in normotensive and hypertensive rats. Necropsy validation. , 1990, American journal of hypertension.
[52] E. DeLong,et al. Comparing the areas under two or more correlated receiver operating characteristic curves: a nonparametric approach. , 1988, Biometrics.
[53] R. Stein,et al. The cardiac response to exercise training: echocardiographic analysis at rest and during exercise. , 1980, The American journal of cardiology.
[54] R. Jones,et al. Effects of exercise training on left ventricular function in normal subjects: a longitudinal study by radionuclide angiography. , 1980, The American journal of cardiology.
[55] David R. Heise,et al. Causal Analysis , 2018, Encyclopedia of Social Network Analysis and Mining. 2nd Ed..
[56] Avani A. Pendse,et al. The Pathophysiology of Cardiac Hypertrophy and Heart Failure , 2014 .
[57] N. Dhalla,et al. Differences in Concentric Cardiac Hypertrophy and Eccentric Hypertrophy , 2013 .
[58] U. Wisløff,et al. Animal models in the study of exercise-induced cardiac hypertrophy. , 2010, Physiological research.
[59] 沈維道. 草蝦Kruppel-Like Factor蛋白質的功能性分析 , 2010 .
[60] K. Tanonaka,et al. Preferable anesthetic conditions for echocardiographic determination of murine cardiac function. , 2005, Journal of pharmacological sciences.
[61] B. Maron. Structural features of the athlete heart as defined by echocardiography. , 1986, Journal of the American College of Cardiology.
[62] Thomas D. Schmittgen,et al. Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2 2 DD C T Method , 2022 .