The environment, geoepidemiology and ANCA-associated vasculitides.

[1]  C. Kallenberg,et al.  Membrane-bound proteinase 3 and its receptors: relevance for the pathogenesis of Wegener's Granulomatosis. , 2009, Autoimmunity reviews.

[2]  W. Gross,et al.  ANCA-associated vasculitides: pathogenetic aspects and current evidence-based therapy. , 2009, Journal of autoimmunity.

[3]  D. Scott,et al.  Renal vasculitis in Japan and the UK--are there differences in epidemiology and clinical phenotype? , 2008, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[4]  N. Basu,et al.  Assessment of systemic vasculitis. , 2008, Autoimmunity reviews.

[5]  Minoru Fukuda,et al.  Molecular mimicry in pauci-immune focal necrotizing glomerulonephritis , 2008, Nature Medicine.

[6]  C. Kallenberg,et al.  Vasculitis and infections: Contribution to the issue of autoimmunity reviews devoted to “autoimmunity and infection” , 2008, Autoimmunity Reviews.

[7]  Kazuo Suzuki,et al.  MPO-ANCA induces IL-17 production by activated neutrophils in vitro via classical complement pathway-dependent manner. , 2008, Journal of autoimmunity.

[8]  Min Chen,et al.  Antineutrophil Cytoplasmic Autoantibody-Associated Vasculitis in Older Patients , 2008, Medicine.

[9]  F. Yu,et al.  Evaluation of a new algorithm in classification of systemic vasculitis. , 2008, Rheumatology.

[10]  C. Kallenberg,et al.  Pathogenesis of PR3-ANCA associated vasculitis. , 2008, Journal of autoimmunity.

[11]  Min Chen,et al.  Follow‐up of avidity and titre of anti‐myeloperoxidase antibodies in sera from patients with propylthiouracil‐induced vasculitis , 2007, Clinical endocrinology.

[12]  D. Savitz,et al.  Association of silica exposure with anti-neutrophil cytoplasmic autoantibody small-vessel vasculitis: a population-based, case-control study. , 2007, Clinical journal of the American Society of Nephrology : CJASN.

[13]  Min Chen,et al.  Inhibition of oxidation activity of myeloperoxidase (MPO) by propylthiouracil (PTU) and anti-MPO antibodies from patients with PTU-induced vasculitis. , 2007, Clinical immunology.

[14]  C. Kallenberg,et al.  Mechanisms of Disease: pathogenesis and treatment of ANCA-associated vasculitides , 2006, Nature Clinical Practice Rheumatology.

[15]  Su-xia Wang,et al.  Renal histology in Chinese patients with anti-myeloperoxidase autoantibody-positive Wegener's granulomatosis. , 2006, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[16]  Kazuo Suzuki,et al.  Incidence of ANCA-associated primary renal vasculitis in the Miyazaki Prefecture: the first population-based, retrospective, epidemiologic survey in Japan. , 2006, Clinical journal of the American Society of Nephrology : CJASN.

[17]  D. Scott,et al.  Development and validation of a consensus methodology for the classification of the ANCA-associated vasculitides and polyarteritis nodosa for epidemiological studies , 2006, Annals of the rheumatic diseases.

[18]  P. Chapman,et al.  The epidemiology of Wegener's granulomatosis and microscopic polyangiitis in a Southern Hemisphere region. , 2006, Rheumatology.

[19]  H-y Wang,et al.  Propylthiouracil-induced anti-neutrophil cytoplasmic antibody-associated vasculitis. , 2006, Kidney international.

[20]  Z. Sachetto,et al.  No evidence of parvovirus B19 in tissue samples from patients with polyarteritis nodosa and microscopic polyangiitis as assessed by the polymerase chain reaction , 2006, Annals of the rheumatic diseases.

[21]  F. Yu,et al.  Cinical and pathological characteristics of Chinese patients with antineutrophil cytoplasmic autoantibody associated systemic vasculitides: a study of 426 patients from a single centre , 2005, Postgraduate Medical Journal.

[22]  Ying Zhang,et al.  Characteristics of Chinese patients with Wegener's granulomatosis with anti-myeloperoxidase autoantibodies. , 2005, Kidney international.

[23]  F. Fourquet,et al.  Occupational exposure in ANCA-positive patients: a case-control study. , 2005, Kidney international.

[24]  T. Links,et al.  Occurrence of antineutrophil cytoplasmic antibodies and associated vasculitis in patients with hyperthyroidism treated with antithyroid drugs: A long-term followup study. , 2005, Arthritis and rheumatism.

[25]  W. Graninger,et al.  Clinical interpretation of antineutrophil cytoplasmic antibodies: parvovirus B19 infection as a pitfall , 2004, Annals of the rheumatic diseases.

[26]  L. Mouthon,et al.  Lack of association between B19 or V9 erythrovirus infection and ANCA-positive vasculitides: a case-control study. , 2003, Rheumatology.

[27]  V. Esnault Apoptosis: the central actor in the three hits that trigger anti-neutrophil cytoplasmic antibody-related systemic vasculitis. , 2002, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[28]  P. Heeringa,et al.  Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice. , 2002, The Journal of clinical investigation.

[29]  H. Raspe,et al.  No difference in the incidences of vasculitides between north and south Germany: first results of the German vasculitis register. , 2002, Rheumatology.

[30]  A. Edefonti,et al.  Anti-neutrophil cytoplasmic autoantibody positive glomerulonephritis in monozygotic twins , 2002, Archives of disease in childhood.

[31]  D. Scott,et al.  Epidemiology of vasculitis in Europe , 2001, Annals of the rheumatic diseases.

[32]  G. Tsay,et al.  Parvovirus B19 infection associated with the production of antineutrophil cytoplasmic antibody (ANCA) and anticardiolipin antibody (aCL) , 2000, Lupus.

[33]  D. Scott,et al.  Epidemiology of systemic vasculitis: a ten-year study in the United Kingdom. , 2000, Arthritis and rheumatism.

[34]  A. Ueki,et al.  Activation-induced cell death in human peripheral blood lymphocytes after stimulation with silicate in vitro. , 1998, International journal of oncology.

[35]  W. El-Reshaid,et al.  The spectrum of renal disease associated with microscopic polyangiitis and classic polyarteritis nodosa in Kuwait. , 1997, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[36]  J. Leigh,et al.  Silica-induced apoptosis in alveolar and granulomatous cells in vivo. , 1997, Environmental health perspectives.

[37]  J. Levine,et al.  Antineutrophil Cytoplasmic Autoantibodies Interact with Primary Granule Constituents on the Surface of Apoptotic Neutrophils in the Absence of Neutrophil Priming , 1996, The Journal of experimental medicine.

[38]  C. Kallenberg,et al.  Trimethoprim-sulfamethoxazole (co-trimoxazole) for the prevention of relapses of Wegener's granulomatosis , 1996 .

[39]  J. Sánchez-Román,et al.  Antimyeloperoxidase antibodies in individuals with occupational exposure to silica. , 1996, Annals of the rheumatic diseases.

[40]  L. Corman,et al.  Association of Wegener's granulomatosis with parvovirus B19 infection: comment on the concise communication by Nikkari et al. , 1995, Arthritis and rheumatism.

[41]  C. Goebel,et al.  Neutrophils and drug metabolism. , 1995, Science.

[42]  G. Schaffner,et al.  A novel class of autoantigens of anti-neutrophil cytoplasmic antibodies in necrotizing and crescentic glomerulonephritis: the lysosomal membrane glycoprotein h-lamp-2 in neutrophil granulocytes and a related membrane protein in glomerular endothelial cells , 1995, The Journal of experimental medicine.

[43]  G. Khursigara,et al.  Transformation of lupus-inducing drugs to cytotoxic products by activated neutrophils. , 1994, Science.

[44]  R J Falk,et al.  Nomenclature of systemic vasculitides. Proposal of an international consensus conference. , 1994, Arthritis and rheumatism.

[45]  C. Kallenberg Overlapping syndromes, undifferentiated connective tissue disease, and other fibrosing conditions , 1992, Current opinion in rheumatology.

[46]  M. Kohno,et al.  Inactivation of peroxidases of rat bone marrow by repeated administration of propylthiouracil is accompanied by a change in the heme structure. , 1988, Biochemical pharmacology.

[47]  Emile de Heer,et al.  Hypotheses on the etiology of antineutrophil cytoplasmic autoantibody associated vasculitis: the cause is hidden, but the result is known. , 2008, Clinical journal of the American Society of Nephrology : CJASN.

[48]  Min Chen,et al.  The target antigens of antineutrophil cytoplasmic antibodies (ANCA) induced by propylthiouracil. , 2007, International immunopharmacology.

[49]  A. Tropsha,et al.  Autoimmunity is triggered by cPR-3(105–201), a protein complementary to human autoantigen proteinase-3 , 2004, Nature Medicine.

[50]  D. Scott,et al.  Epidemiology of the vasculitides. , 2003, Current opinion in rheumatology.

[51]  R. Falk,et al.  Silica exposure in anti-neutrophil cytoplasmic autoantibody-associated glomerulonephritis and lupus nephritis. , 2001, Journal of the American Society of Nephrology : JASN.

[52]  C. Kallenberg,et al.  Association of Chronic Nasal Carriage of Staphylococcus aureus and Higher Relapse Rates in Wegener Granulomatosis , 1994, Annals of Internal Medicine.