The environment, geoepidemiology and ANCA-associated vasculitides.
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[1] C. Kallenberg,et al. Membrane-bound proteinase 3 and its receptors: relevance for the pathogenesis of Wegener's Granulomatosis. , 2009, Autoimmunity reviews.
[2] W. Gross,et al. ANCA-associated vasculitides: pathogenetic aspects and current evidence-based therapy. , 2009, Journal of autoimmunity.
[3] D. Scott,et al. Renal vasculitis in Japan and the UK--are there differences in epidemiology and clinical phenotype? , 2008, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[4] N. Basu,et al. Assessment of systemic vasculitis. , 2008, Autoimmunity reviews.
[5] Minoru Fukuda,et al. Molecular mimicry in pauci-immune focal necrotizing glomerulonephritis , 2008, Nature Medicine.
[6] C. Kallenberg,et al. Vasculitis and infections: Contribution to the issue of autoimmunity reviews devoted to “autoimmunity and infection” , 2008, Autoimmunity Reviews.
[7] Kazuo Suzuki,et al. MPO-ANCA induces IL-17 production by activated neutrophils in vitro via classical complement pathway-dependent manner. , 2008, Journal of autoimmunity.
[8] Min Chen,et al. Antineutrophil Cytoplasmic Autoantibody-Associated Vasculitis in Older Patients , 2008, Medicine.
[9] F. Yu,et al. Evaluation of a new algorithm in classification of systemic vasculitis. , 2008, Rheumatology.
[10] C. Kallenberg,et al. Pathogenesis of PR3-ANCA associated vasculitis. , 2008, Journal of autoimmunity.
[11] Min Chen,et al. Follow‐up of avidity and titre of anti‐myeloperoxidase antibodies in sera from patients with propylthiouracil‐induced vasculitis , 2007, Clinical endocrinology.
[12] D. Savitz,et al. Association of silica exposure with anti-neutrophil cytoplasmic autoantibody small-vessel vasculitis: a population-based, case-control study. , 2007, Clinical journal of the American Society of Nephrology : CJASN.
[13] Min Chen,et al. Inhibition of oxidation activity of myeloperoxidase (MPO) by propylthiouracil (PTU) and anti-MPO antibodies from patients with PTU-induced vasculitis. , 2007, Clinical immunology.
[14] C. Kallenberg,et al. Mechanisms of Disease: pathogenesis and treatment of ANCA-associated vasculitides , 2006, Nature Clinical Practice Rheumatology.
[15] Su-xia Wang,et al. Renal histology in Chinese patients with anti-myeloperoxidase autoantibody-positive Wegener's granulomatosis. , 2006, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[16] Kazuo Suzuki,et al. Incidence of ANCA-associated primary renal vasculitis in the Miyazaki Prefecture: the first population-based, retrospective, epidemiologic survey in Japan. , 2006, Clinical journal of the American Society of Nephrology : CJASN.
[17] D. Scott,et al. Development and validation of a consensus methodology for the classification of the ANCA-associated vasculitides and polyarteritis nodosa for epidemiological studies , 2006, Annals of the rheumatic diseases.
[18] P. Chapman,et al. The epidemiology of Wegener's granulomatosis and microscopic polyangiitis in a Southern Hemisphere region. , 2006, Rheumatology.
[19] H-y Wang,et al. Propylthiouracil-induced anti-neutrophil cytoplasmic antibody-associated vasculitis. , 2006, Kidney international.
[20] Z. Sachetto,et al. No evidence of parvovirus B19 in tissue samples from patients with polyarteritis nodosa and microscopic polyangiitis as assessed by the polymerase chain reaction , 2006, Annals of the rheumatic diseases.
[21] F. Yu,et al. Cinical and pathological characteristics of Chinese patients with antineutrophil cytoplasmic autoantibody associated systemic vasculitides: a study of 426 patients from a single centre , 2005, Postgraduate Medical Journal.
[22] Ying Zhang,et al. Characteristics of Chinese patients with Wegener's granulomatosis with anti-myeloperoxidase autoantibodies. , 2005, Kidney international.
[23] F. Fourquet,et al. Occupational exposure in ANCA-positive patients: a case-control study. , 2005, Kidney international.
[24] T. Links,et al. Occurrence of antineutrophil cytoplasmic antibodies and associated vasculitis in patients with hyperthyroidism treated with antithyroid drugs: A long-term followup study. , 2005, Arthritis and rheumatism.
[25] W. Graninger,et al. Clinical interpretation of antineutrophil cytoplasmic antibodies: parvovirus B19 infection as a pitfall , 2004, Annals of the rheumatic diseases.
[26] L. Mouthon,et al. Lack of association between B19 or V9 erythrovirus infection and ANCA-positive vasculitides: a case-control study. , 2003, Rheumatology.
[27] V. Esnault. Apoptosis: the central actor in the three hits that trigger anti-neutrophil cytoplasmic antibody-related systemic vasculitis. , 2002, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[28] P. Heeringa,et al. Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice. , 2002, The Journal of clinical investigation.
[29] H. Raspe,et al. No difference in the incidences of vasculitides between north and south Germany: first results of the German vasculitis register. , 2002, Rheumatology.
[30] A. Edefonti,et al. Anti-neutrophil cytoplasmic autoantibody positive glomerulonephritis in monozygotic twins , 2002, Archives of disease in childhood.
[31] D. Scott,et al. Epidemiology of vasculitis in Europe , 2001, Annals of the rheumatic diseases.
[32] G. Tsay,et al. Parvovirus B19 infection associated with the production of antineutrophil cytoplasmic antibody (ANCA) and anticardiolipin antibody (aCL) , 2000, Lupus.
[33] D. Scott,et al. Epidemiology of systemic vasculitis: a ten-year study in the United Kingdom. , 2000, Arthritis and rheumatism.
[34] A. Ueki,et al. Activation-induced cell death in human peripheral blood lymphocytes after stimulation with silicate in vitro. , 1998, International journal of oncology.
[35] W. El-Reshaid,et al. The spectrum of renal disease associated with microscopic polyangiitis and classic polyarteritis nodosa in Kuwait. , 1997, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[36] J. Leigh,et al. Silica-induced apoptosis in alveolar and granulomatous cells in vivo. , 1997, Environmental health perspectives.
[37] J. Levine,et al. Antineutrophil Cytoplasmic Autoantibodies Interact with Primary Granule Constituents on the Surface of Apoptotic Neutrophils in the Absence of Neutrophil Priming , 1996, The Journal of experimental medicine.
[38] C. Kallenberg,et al. Trimethoprim-sulfamethoxazole (co-trimoxazole) for the prevention of relapses of Wegener's granulomatosis , 1996 .
[39] J. Sánchez-Román,et al. Antimyeloperoxidase antibodies in individuals with occupational exposure to silica. , 1996, Annals of the rheumatic diseases.
[40] L. Corman,et al. Association of Wegener's granulomatosis with parvovirus B19 infection: comment on the concise communication by Nikkari et al. , 1995, Arthritis and rheumatism.
[41] C. Goebel,et al. Neutrophils and drug metabolism. , 1995, Science.
[42] G. Schaffner,et al. A novel class of autoantigens of anti-neutrophil cytoplasmic antibodies in necrotizing and crescentic glomerulonephritis: the lysosomal membrane glycoprotein h-lamp-2 in neutrophil granulocytes and a related membrane protein in glomerular endothelial cells , 1995, The Journal of experimental medicine.
[43] G. Khursigara,et al. Transformation of lupus-inducing drugs to cytotoxic products by activated neutrophils. , 1994, Science.
[44] R J Falk,et al. Nomenclature of systemic vasculitides. Proposal of an international consensus conference. , 1994, Arthritis and rheumatism.
[45] C. Kallenberg. Overlapping syndromes, undifferentiated connective tissue disease, and other fibrosing conditions , 1992, Current opinion in rheumatology.
[46] M. Kohno,et al. Inactivation of peroxidases of rat bone marrow by repeated administration of propylthiouracil is accompanied by a change in the heme structure. , 1988, Biochemical pharmacology.
[47] Emile de Heer,et al. Hypotheses on the etiology of antineutrophil cytoplasmic autoantibody associated vasculitis: the cause is hidden, but the result is known. , 2008, Clinical journal of the American Society of Nephrology : CJASN.
[48] Min Chen,et al. The target antigens of antineutrophil cytoplasmic antibodies (ANCA) induced by propylthiouracil. , 2007, International immunopharmacology.
[49] A. Tropsha,et al. Autoimmunity is triggered by cPR-3(105–201), a protein complementary to human autoantigen proteinase-3 , 2004, Nature Medicine.
[50] D. Scott,et al. Epidemiology of the vasculitides. , 2003, Current opinion in rheumatology.
[51] R. Falk,et al. Silica exposure in anti-neutrophil cytoplasmic autoantibody-associated glomerulonephritis and lupus nephritis. , 2001, Journal of the American Society of Nephrology : JASN.
[52] C. Kallenberg,et al. Association of Chronic Nasal Carriage of Staphylococcus aureus and Higher Relapse Rates in Wegener Granulomatosis , 1994, Annals of Internal Medicine.