Electrophysiologic properties of intravenous isradipine in persons with normal sinus node and atrioventricular nodal function

[1]  R. Hof.  Comparison of cardiodepressant and vasodilator effects of PN 200-110 (isradipine), nifedipine and diltiazem in anesthetized rabbits. , 1987, The American journal of cardiology.

[2]  N. Taira,et al.  Differences in cardiovascular profile among calcium antagonists. , 1987, The American journal of cardiology.

[3]  J. Somberg.  Calcium channel blockers that prolong the QT interval. , 1985, American heart journal.

[4]  Y. Wada,et al.  Separation of the Coronary Vasodilator from the Cardiac Effects of PN 200–110, a New Dihydropyridine Calcium Antagonist, in the Dog Heart , 1985, Journal of cardiovascular pharmacology.

[5]  H. Vuorela,et al.  PN 200–110, A New Calcium Antagonist: Electrophysiological, Inotropic, and Chronotropic Effects on Guinea Pig Myocardial Tissue and Effects on Contraction and Calcium Uptake of Rabbit Aorta , 1984, Journal of cardiovascular pharmacology.

[6]  G. Scholtysik,et al.  Effects of the New Calcium Antagonist PN 200–110 on the Myocardium and the Regional Peripheral Circulation in Anesthetized Cats and Dogs , 1984, Journal of cardiovascular pharmacology.

[7]  S. Wolf,et al.  The QT interval during reflex cardiovascular adaptation. , 1984, Circulation.

[8]  G. Scholtysik,et al.  Effects of the Calcium Antagonist PY 108–068 on Myocardial Tissues In Vitro and on Reflex Tachycardia In Vivo , 1983, Journal of cardiovascular pharmacology.

[9]  J. Mason,et al.  Comparative clinical electrophysiologic effects of diltiazem, verapamil and nifedipine: a review. , 1982, The American journal of cardiology.

[10]  J. Bigger,et al.  Premature Atrial Stimulation as a Key to the Understanding of Sinoatrial Conduction in Man: Presentation of Data and Critical Review of the Literature , 1973, Circulation.