A comparative gene expression matrix in Apoe-deficient mice identifies unique and atherosclerotic disease stage-specific gene regulation patterns in monocytes and macrophages.

[1]  M. Bader,et al.  Role of Gpnmb in atherosclerosis of female mice. , 2022, Biochemical and biophysical research communications.

[2]  M. Reilly,et al.  Long Noncoding RNA MIAT Controls Advanced Atherosclerotic Lesion Formation and Plaque Destabilization , 2021, Circulation.

[3]  K. Ley,et al.  Myeloid cell-specific Irf5 deficiency stabilizes atherosclerotic plaques in Apoe–/– mice , 2021, Molecular metabolism.

[4]  C. Scavone,et al.  The Role of GPNMB in Inflammation , 2021, Frontiers in Immunology.

[5]  M. Kiss,et al.  Haematopoetic TREM2 deficiency modulates atherosclerosis and lipid metabolism , 2020 .

[6]  P. Kohl,et al.  Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering , 2020, Basic Research in Cardiology.

[7]  C. Glass,et al.  Microanatomy of the Human Atherosclerotic Plaque by Single-Cell Transcriptomics , 2020, Circulation research.

[8]  J. Molkentin,et al.  Ontogeny of arterial macrophages defines their functions in homeostasis and inflammation , 2020, Nature Communications.

[9]  Maxim N. Artyomov,et al.  Limited proliferation capacity of aorta intima resident macrophages requires monocyte recruitment for atherosclerotic plaque progression , 2020, Nature Immunology.

[10]  K. Moore,et al.  Regulatory T Cells License Macrophage Pro-Resolving Functions During Atherosclerosis Regression , 2020, Circulation research.

[11]  L. Willemsen,et al.  Macrophage subsets in atherosclerosis as defined by single‐cell technologies , 2020, The Journal of pathology.

[12]  R. Diaz,et al.  Efficacy and Safety of Low-Dose Colchicine after Myocardial Infarction. , 2019, The New England journal of medicine.

[13]  Clint L. Miller,et al.  Atheroprotective roles of smooth muscle cell phenotypic modulation and the TCF21 disease gene as revealed by single-cell analysis , 2019, Nature Medicine.

[14]  Qingbo Xu,et al.  Adventitial Cell Atlas of wt (Wild Type) and ApoE (Apolipoprotein E)-Deficient Mice Defined by Single-Cell RNA Sequencing , 2019, Arteriosclerosis, thrombosis, and vascular biology.

[15]  S. Ramsey,et al.  Single-cell analysis of fate-mapped macrophages reveals heterogeneity, including stem-like properties, during atherosclerosis progression and regression. , 2019, JCI insight.

[16]  Maxim N. Artyomov,et al.  Transcriptome Analysis Reveals Nonfoamy Rather Than Foamy Plaque Macrophages Are Proinflammatory in Atherosclerotic Murine Models , 2018, Circulation research.

[17]  W. Ling,et al.  Apoptotic cell induction of miR-10b in macrophages contributes to advanced atherosclerosis progression in ApoE−/− mice , 2018, Cardiovascular research.

[18]  Dennis Wolf,et al.  Atlas of the Immune Cell Repertoire in Mouse Atherosclerosis Defined by Single-Cell RNA-Sequencing and Mass Cytometry , 2018, Circulation research.

[19]  Dennis Wolf,et al.  Single-Cell RNA-Seq Reveals the Transcriptional Landscape and Heterogeneity of Aortic Macrophages in Murine Atherosclerosis , 2018, Circulation research.

[20]  P. Libby,et al.  Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease , 2017, The New England journal of medicine.

[21]  T. Renné,et al.  MicroRNA-210 Enhances Fibrous Cap Stability in Advanced Atherosclerotic Lesions , 2017, Circulation research.

[22]  K. Bornfeldt,et al.  Macrophage Phenotype and Function in Different Stages of Atherosclerosis. , 2016, Circulation research.

[23]  P. Libby,et al.  Atheroprotection through SYK inhibition fails in established disease when local macrophage proliferation dominates lesion progression , 2016, Basic Research in Cardiology.

[24]  Clinton S. Robbins,et al.  Monocyte fate in atherosclerosis. , 2015, Arteriosclerosis, thrombosis, and vascular biology.

[25]  Christian Stolte,et al.  COMPARTMENTS: unification and visualization of protein subcellular localization evidence , 2014, Database J. Biol. Databases Curation.

[26]  P. Libby,et al.  Local proliferation dominates lesional macrophage accumulation in atherosclerosis , 2013, Nature Medicine.

[27]  F. Ginhoux,et al.  Tissue-resident macrophages self-maintain locally throughout adult life with minimal contribution from circulating monocytes. , 2013, Immunity.

[28]  A. Mildner,et al.  Fate mapping reveals origins and dynamics of monocytes and tissue macrophages under homeostasis. , 2013, Immunity.

[29]  Andrew C. Li,et al.  Regulated Accumulation of Desmosterol Integrates Macrophage Lipid Metabolism and Inflammatory Responses , 2012, Cell.

[30]  P. Libby Inflammation in Atherosclerosis , 2012, Arteriosclerosis, thrombosis, and vascular biology.

[31]  F. Geissmann,et al.  Monocytes in atherosclerosis: subsets and functions , 2010, Nature Reviews Cardiology.

[32]  H. Itabe Oxidative Modification of LDL: Its Pathological Role in Atherosclerosis , 2009, Clinical reviews in allergy & immunology.

[33]  P. Libby,et al.  Identification of Splenic Reservoir Monocytes and Their Deployment to Inflammatory Sites , 2009, Science.

[34]  M. Febbraio,et al.  Absence of CD36 protects against atherosclerosis in ApoE knock-out mice with no additional protection provided by absence of scavenger receptor A I/II. , 2008, Cardiovascular research.

[35]  P. Libby,et al.  Monocyte accumulation in mouse atherogenesis is progressive and proportional to extent of disease , 2006, Proceedings of the National Academy of Sciences.

[36]  G. Hansson,et al.  Inflammation and atherosclerosis. , 2006, Annual review of pathology.

[37]  J Fernando Bazan,et al.  IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. , 2005, Immunity.

[38]  K. Moore,et al.  Loss of receptor-mediated lipid uptake via scavenger receptor A or CD36 pathways does not ameliorate atherosclerosis in hyperlipidemic mice. , 2005, The Journal of clinical investigation.

[39]  P. Libby Inflammation in atherosclerosis , 2002, Nature.

[40]  Z. Galis,et al.  Atherosclerotic lesions grow through recruitment and proliferation of circulating monocytes in a murine model. , 2002, The American journal of pathology.

[41]  M. Jaye,et al.  PPAR-α and PPAR-γ activators induce cholesterol removal from human macrophage foam cells through stimulation of the ABCA1 pathway , 2001, Nature Medicine.

[42]  Andrew C. Li,et al.  Polymerase Chain Reaction–Based Method for Quantifying Recruitment of Monocytes to Mouse Atherosclerotic Lesions In Vivo Enhancement by Tumor Necrosis Factor-α and Interleukin-1β , 2000 .

[43]  P. Casellas,et al.  lnterleukin-13 is a new human lymphokine regulating inflammatory and immune responses , 1993, Nature.

[44]  H. Aburatani,et al.  Human macrophage scavenger receptors: primary structure, expression, and localization in atherosclerotic lesions. , 1990, Proceedings of the National Academy of Sciences of the United States of America.