Anti-E. coli Immunoglobulin Yolk (IgY): Reduction of pathogen receptors and inflammation factors could be caused by decrease in E. coli load

[1]  J. Quevedo,et al.  Inhibition of indoleamine 2,3-dioxygenase 1/2 prevented cognitive impairment and energetic metabolism changes in the hippocampus of adult rats subjected to polymicrobial sepsis , 2017, Journal of Neuroimmunology.

[2]  E. Holler,et al.  Reduction of aGVHD using chicken antibodies directed against intestinal pathogens in a murine model. , 2017, Blood.

[3]  Daniel Wolff,et al.  Low urinary indoxyl sulfate levels early after transplantation reflect a disrupted microbiome and are associated with poor outcome. , 2015, Blood.

[4]  Y. Taur,et al.  Intestinal Blautia Is Associated with Reduced Death from Graft-versus-Host Disease. , 2015, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

[5]  J. Kagan,et al.  The Dorsoventral Regulatory Gene Cassette spätzle / Toll / cactus Controls the Potent Antifungal Response in Drosophila Adults , 2015 .

[6]  A. Viale,et al.  The effects of intestinal tract bacterial diversity on mortality following allogeneic hematopoietic stem cell transplantation. , 2014, Blood.

[7]  E. Holler,et al.  Cytostatic conditioning in experimental allogeneic bone marrow transplantation: Busulfan causes less early gastrointestinal toxicity but Treosulfan results in improved immune reconstitution , 2014, Immunopharmacology and immunotoxicology.

[8]  R. Fernandez,et al.  Recognition of lipid A variants by the TLR4-MD-2 receptor complex , 2012, Front. Cell. Inf. Microbio..

[9]  S. Akira,et al.  Muramyl Dipeptide Induces NOD2-Dependent Ly6Chigh Monocyte Recruitment to the Lungs and Protects Against Influenza Virus Infection , 2012, PloS one.

[10]  R. Khanin,et al.  Regulation of intestinal inflammation by microbiota following allogeneic bone marrow transplantation , 2012, The Journal of experimental medicine.

[11]  P. Rosenstiel,et al.  MIP-3α Expression in Macrophages Is NOD Dependent , 2012, Digestion.

[12]  P. Oefner,et al.  Tryptophan catabolism is associated with acute GVHD after human allogeneic stem cell transplantation and indicates activation of indoleamine 2,3-dioxygenase. , 2011, Blood.

[13]  J. Hansen,et al.  Sensing disease and danger: a survey of vertebrate PRRs and their origins. , 2011, Developmental and comparative immunology.

[14]  N. Warner,et al.  The Nod2 sensor promotes intestinal pathogen eradication via the chemokine CCL2-dependent recruitment of inflammatory monocytes. , 2011, Immunity.

[15]  E. Holler,et al.  Steroid treatment alters adhesion molecule and chemokine expression in experimental acute graft-vs.-host disease of the intestinal tract. , 2011, Experimental hematology.

[16]  D. Stevens,et al.  Innate immune recognition of, and response to, Clostridium sordellii. , 2010, Anaerobe.

[17]  S. Choi,et al.  Acute graft-versus-host disease: new treatment strategies , 2009, Current opinion in hematology.

[18]  E. Holler,et al.  Chemokine and chemokine receptor expression analysis in target organs of acute graft-versus-host disease , 2009, Genes and Immunity.

[19]  G. Gerken,et al.  Colitis-associated variant of TLR2 causes impaired mucosal repair because of TFF3 deficiency. , 2009, Gastroenterology.

[20]  J. Blanco,et al.  Molecular diversity of Escherichia coli in the human gut: New ecological evidence supporting the role of adherent‐invasive E. coli (AIEC) in Crohn's disease , 2009, Inflammatory bowel diseases.

[21]  Jun Sik Lee,et al.  Blockade of Indoleamine 2,3-Dioxygenase Protects Mice against Lipopolysaccharide-Induced Endotoxin Shock1 , 2009, The Journal of Immunology.

[22]  E. Holler,et al.  Graft-versus-host disease , 2009, The Lancet.

[23]  S. Akira,et al.  TLR agonists regulate alloresponses and uncover a critical role for donor APCs in allogeneic bone marrow rejection. , 2008, Blood.

[24]  S. Akira,et al.  Deletion of TLR5 results in spontaneous colitis in mice. , 2007, The Journal of clinical investigation.

[25]  U. Göbel,et al.  Shift Towards Pro-inflammatory Intestinal Bacteria Aggravates Acute Murine Colitis via Toll-like Receptors 2 and 4 , 2007, PloS one.

[26]  A. Gewirtz,et al.  Activation of toll‐like receptor 3 protects against DSS‐induced acute colitis , 2007, Inflammatory bowel diseases.

[27]  R. Coffman,et al.  Therapeutic targeting of innate immunity with Toll-like receptor agonists and antagonists , 2007, Nature Medicine.

[28]  D. Fuchs,et al.  HIV inhibits CD4+ T-cell proliferation by inducing indoleamine 2,3-dioxygenase in plasmacytoid dendritic cells. , 2007, Blood.

[29]  J. Ravetch,et al.  Opposing effects of Toll-like receptor stimulation induce autoimmunity or tolerance. , 2007, Trends in immunology.

[30]  Yu-Tseung Liu,et al.  Maintenance of colonic homeostasis by distinctive apical TLR9 signalling in intestinal epithelial cells , 2006, Nature Cell Biology.

[31]  Charles P. Lin,et al.  An inflammatory checkpoint regulates recruitment of graft-versus-host reactive T cells to peripheral tissues , 2006, The Journal of experimental medicine.

[32]  G. Rogler,et al.  Prognostic significance of NOD2/CARD15 variants in HLA-identical sibling hematopoietic stem cell transplantation: effect on long-term outcome is confirmed in 2 independent cohorts and may be modulated by the type of gastrointestinal decontamination. , 2006, Blood.

[33]  F. Martinon,et al.  NLRs join TLRs as innate sensors of pathogens. , 2005, Trends in immunology.

[34]  D. Fuchs,et al.  ENHANCED ENZYMATIC DEGRADATION OF TRYPTOPHAN BY INDOLEAMINE 2,3-DIOXYGENASE CONTRIBUTES TO THE TRYPTOPHAN-DEFICIENT STATE SEEN AFTER MAJOR TRAUMA , 2005, Shock.

[35]  Richard A. Flavell,et al.  Nod2-Dependent Regulation of Innate and Adaptive Immunity in the Intestinal Tract , 2005, Science.

[36]  Michael Karin,et al.  Nod2 Mutation in Crohn's Disease Potentiates NF-κB Activity and IL-1ß Processing , 2005, Science.

[37]  D. Schwartz,et al.  Toll-like receptors in the pathogenesis of human disease , 2004, Nature Immunology.

[38]  D. Munn,et al.  Ido expression by dendritic cells: tolerance and tryptophan catabolism , 2004, Nature Reviews Immunology.

[39]  Laurent Beaugerie,et al.  High prevalence of adherent-invasive Escherichia coli associated with ileal mucosa in Crohn's disease. , 2004, Gastroenterology.

[40]  G. Rogler,et al.  Both donor and recipient NOD2/CARD15 mutations associate with transplant-related mortality and GvHD following allogeneic stem cell transplantation. , 2004, Blood.

[41]  J. Schölmerich,et al.  Probiotic effects on experimental graft-versus-host disease: let them eat yogurt. , 2004, Blood.

[42]  M. Murray.  Tryptophan depletion and HIV infection: a metabolic link to pathogenesis. , 2003, The Lancet. Infectious diseases.

[43]  S. Janssens,et al.  Role of Toll-Like Receptors in Pathogen Recognition , 2003, Clinical Microbiology Reviews.

[44]  S. Akira,et al.  Toll-like receptors control activation of adaptive immune responses , 2001, Nature Immunology.

[45]  M. Seishima,et al.  Lipopolysaccharide induction of indoleamine 2,3‐dioxygenase is mediated dominantly by an IFN‐γ‐independent mechanism , 2001, European journal of immunology.

[46]  Zvi Fuks,et al.  Endothelial Apoptosis as the Primary Lesion Initiating Intestinal Radiation Damage in Mice , 2001, Science.

[47]  Y. Ogura,et al.  Human Nod1 Confers Responsiveness to Bacterial Lipopolysaccharides* , 2001, The Journal of Biological Chemistry.

[48]  W. Hammes,et al.  Characterization of Reutericyclin Produced by Lactobacillus reuteri LTH2584 , 2000, Applied and Environmental Microbiology.

[49]  C. Janeway,et al.  A human homologue of the Drosophila Toll protein signals activation of adaptive immunity , 1997, Nature.

[50]  B. Lemaître,et al.  The Dorsoventral Regulatory Gene Cassette spätzle/Toll/cactus Controls the Potent Antifungal Response in Drosophila Adults , 1996, Cell.

[51]  John S. Thompson,et al.  Effect of total body irradiation, busulfan-cyclophosphamide, or cyclophosphamide conditioning on inflammatory cytokine release and development of acute and chronic graft-versus-host disease in H-2- incompatible transplanted SCID mice , 1994 .

[52]  J. Armitage.  Bone marrow transplantation. , 1994, The New England journal of medicine.

[53]  U. Graeven,et al.  Evidence that sustained growth suppression of intestinal anaerobic bacteria reduces the risk of acute graft-versus-host disease after sibling marrow transplantation. , 1992, Blood.

[54]  F. P. Nestel,et al.  Macrophage priming and lipopolysaccharide-triggered release of tumor necrosis factor alpha during graft-versus-host disease , 1992, The Journal of experimental medicine.

[55]  I. Casas,et al.  Production and isolation of reuterin, a growth inhibitor produced by Lactobacillus reuteri , 1988, Antimicrobial Agents and Chemotherapy.

[56]  D. V. van Bekkum,et al.  Role of bacterial microflora in development of intestinal lesions from graft-versus-host reaction. , 1977, Journal of the National Cancer Institute.

[57]  D. van der Waaij,et al.  Mitigation of secondary disease of allogeneic mouse radiation chimeras by modification of the intestinal microflora. , 1974, Journal of the National Cancer Institute.