Role of CSBP/p38/RK stress response kinase in LPS and cytokine signaling mechanisms
暂无分享,去创建一个
[1] A. Dilella,et al. Localization of the human stress responsive MAP kinase-like CSAIDs binding protein (CSBP) gene to chromosome 6q21.3/21.2. , 1995, Genomics.
[2] E. Goldsmith,et al. How MAP Kinases Are Regulated (*) , 1995, The Journal of Biological Chemistry.
[3] P. Cohen,et al. Activation of the MAP kinase homologue RK requires the phosphorylation of Thr‐180 and Tyr‐182 and both residues are phosphorylated in chemically stressed KB cells , 1995, FEBS letters.
[4] Philip R. Cohen,et al. SB 203580 is a specific inhibitor of a MAP kinase homologue which is stimulated by cellular stresses and interleukin‐1 , 1995, FEBS letters.
[5] Jiahuai Han,et al. Pro-inflammatory Cytokines and Environmental Stress Cause p38 Mitogen-activated Protein Kinase Activation by Dual Phosphorylation on Tyrosine and Threonine (*) , 1995, The Journal of Biological Chemistry.
[6] Jiahuai Han,et al. Independent human MAP-kinase signal transduction pathways defined by MEK and MKK isoforms , 1995, Science.
[7] A. Hand,et al. Mechanism of action of bicyclic imidazoles defines a translational regulatory pathway for tumor necrosis factor alpha. , 1995, Journal of inflammation.
[8] L. Zon,et al. Role of SAPK/ERK kinase-1 in the stress-activated pathway regulating transcription factor c-Jun , 1994, Nature.
[9] Jerry L. Adams,et al. A protein kinase involved in the regulation of inflammatory cytokine biosynthesis , 1994, Nature.
[10] J. Hsuan,et al. Interleukin-1 activates a novel protein kinase cascade that results in the phosphorylation of hsp27 , 1994, Cell.
[11] D. Bertolini,et al. Cytokine suppressive anti-inflammatory compounds inhibit bone resorption in vitro. , 1994, Bone.
[12] A. Levitzki,et al. Prevention of lipopolysaccharide-induced lethal toxicity by tyrosine kinase inhibitors. , 1994, Science.
[13] J. Woodgett,et al. The stress-activated protein kinase subfamily of c-Jun kinases , 1994, Nature.
[14] M. Karin,et al. JNK1: A protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain , 1994, Cell.
[15] Cord Sunderkötter,et al. Macrophages and angiogenesis , 1994, Journal of leukocyte biology.
[16] R. Ulevitch,et al. Recognition of endotoxin by cells leading to transmembrane signaling. , 1994, Current opinion in immunology.
[17] C. Manthey,et al. Interactions of lipopolysaccharide with macrophages. , 1994, Immunology series.
[18] Jiahuai Han,et al. Endotoxin induces rapid protein tyrosine phosphorylation in 70Z/3 cells expressing CD14. , 1993, The Journal of biological chemistry.
[19] M. Kracht,et al. Interleukin 1 and tumour necrosis factor activate the mitogen‐activated protein (MAP) kinase kinase in cultured cells , 1993, FEBS letters.
[20] P. Bender,et al. Bicyclic Imidazoles as a Novel Class of Cytokine Biosynthesis Inhibitors , 1993, Annals of the New York Academy of Sciences.
[21] C. June,et al. Lipopolysaccharide-induced protein tyrosine phosphorylation in human macrophages is mediated by CD14. , 1993, Journal of immunology.
[22] I. Fidler,et al. Tyrosine phosphorylation of mitogen-activated protein kinases is necessary for activation of murine macrophages by natural and synthetic bacterial products , 1993, The Journal of experimental medicine.
[23] N. Freshney,et al. Interleukin 1 and tumor necrosis factor stimulate two novel protein kinases that phosphorylate the heat shock protein hsp27 and beta-casein. , 1993, The Journal of biological chemistry.
[24] S. Dower. Cytokine receptor families. , 1993, Advances in second messenger and phosphoprotein research.
[25] C. Dinarello,et al. Inflammatory cytokines: interleukin-1 and tumor necrosis factor as effector molecules in autoimmune diseases , 1991, Current Biology.
[26] D. Kioussis,et al. Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis. , 1991, The EMBO journal.
[27] Y. Tan,et al. Interleukin 1 and tumor necrosis factor activate common multiple protein kinases in human fibroblasts. , 1991, The Journal of biological chemistry.
[28] Nancy Y. Ip,et al. ERKs: A family of protein-serine/threonine kinases that are activated and tyrosine phosphorylated in response to insulin and NGF , 1991, Cell.
[29] B. Beutler,et al. Interactive effects of the tumor necrosis factor promoter and 3'-untranslated regions. , 1991, Journal of immunology.
[30] R. Ulevitch,et al. CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein. , 1990, Science.
[31] B. Beutler,et al. The essential role of the UA-rich sequence in endotoxin-induced cachectin/TNF synthesis. , 1990, European cytokine network.
[32] C V Jongeneel,et al. Kappa B-type enhancers are involved in lipopolysaccharide-mediated transcriptional activation of the tumor necrosis factor alpha gene in primary macrophages , 1990, The Journal of experimental medicine.
[33] John C. Lee,et al. Inhibition of human monocyte IL-1 production by SK&F 86002 , 1990 .
[34] K. Matsushima,et al. Enhanced phosphorylation of 65 and 74 kDa proteins by tumor necrosis factor and interleukin-1 in human peripheral blood mononuclear cells. , 1990, Cytokine.
[35] B. Beutler,et al. Translational control mediated by UA-rich sequences. , 1990, Enzyme.
[36] G. Shaw,et al. Translational blockade imposed by cytokine-derived UA-rich sequences. , 1989, Science.
[37] J. Lee,et al. Inhibition of monocyte IL-1 production by the anti-inflammatory compound, SK&F 86002. , 1988, International journal of immunopharmacology.
[38] B. Beutler,et al. Identification of a common nucleotide sequence in the 3'-untranslated region of mRNA molecules specifying inflammatory mediators. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[39] P. Bender,et al. Antiinflammatory activity of 5,6-diaryl-2,3-dihydroimidazo[2,1-b]thiazoles. Isomeric 4-pyridyl and 4-substituted phenyl derivatives. , 1984, Journal of medicinal chemistry.