Gene ecology: Acis-acting gene-to-gene interaction due to the spatial arrangement of genes in chromosomes affects neighbouring transfected c-H-ras expression

[1]  W. Krzyzosiak,et al.  Isolation of genes specifically expressed in flat revertant cells derived from activated ras-transformed NIH 3T3 cells by treatment with azatyrosine. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[2]  M. Tykocinski,et al.  Loss of tumorigenicity of rat glioblastoma directed by episome-based antisense cDNA transcription of insulin-like growth factor I. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[3]  H. Naora,et al.  An optimum dose of c-H-ras is a prerequisite for hormone-dependent conversion of a cell between cancerous and normal states in tissue culture. , 1991, Cell biology international reports.

[4]  H. Naora,et al.  Reversible Conversion of Cells Between Cancerous and Normal States , 1991 .

[5]  R. Friedman,et al.  Expression of gene rrg is associated with reversion of NIH 3T3 transformed by LTR-c-H-ras. , 1990, Science.

[6]  Samuel H. Wilson,et al.  Deregulation of DNA polymerase β by sense and antisense RNA expression in mouse 3T3 cells alters cell growth , 1990, Somatic cell and molecular genetics.

[7]  P. Lengyel,et al.  Isolation of recessive (mediator-) revertants from NIH 3T3 cells transformed with a c-H-ras oncogene , 1990, Molecular and cellular biology.

[8]  T. Sugimura,et al.  Flat reversion by okadaic acid of raf and ret-II transformants. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[9]  I. G. Young,et al.  Fine-structure mapping of the murine IL-3 and GM-CSF genes by pulsed-field gel electrophoresis and molecular cloning. , 1989, Genomics.

[10]  J. Shay,et al.  Reversible cellular senescence: implications for immortalization of normal human diploid fibroblasts , 1989, Molecular and cellular biology.

[11]  H. Rubin,et al.  Evidence for the progressive and adaptive nature of spontaneous transformation in the NIH 3T3 cell line. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[12]  H. Kitayama,et al.  A ras-related gene with transformation suppressor activity , 1989, Cell.

[13]  P. L. Chen,et al.  Suppression of the neoplastic phenotype by replacement of the RB gene in human cancer cells. , 1988, Science.

[14]  T. Sugimura,et al.  Deletion of transfected oncogenes from NIH 3T3 transformants by inhibitors of poly(ADP-ribose) polymerase. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[15]  C. Y. Wang,et al.  Production of tumor necrosis factor/cachectin by human T cell lines and peripheral blood T lymphocytes stimulated by phorbol myristate acetate and anti-CD3 antibody , 1988, The Journal of experimental medicine.

[16]  G. Trinchieri,et al.  Independent regulation of tumor necrosis factor and lymphotoxin production by human peripheral blood lymphocytes , 1987, The Journal of experimental medicine.

[17]  M. Emerman,et al.  Comparison of promoter suppression in avian and murine retrovirus vectors. , 1986, Nucleic acids research.

[18]  B. Groner,et al.  The v‐mos and H‐ras oncogene expression represses glucocorticoid hormone‐dependent transcription from the mouse mammary tumor virus LTR. , 1986, The EMBO journal.

[19]  R. King,et al.  Androgen regulation by the long terminal repeat of mouse mammary tumor virus , 1986, Molecular and cellular biology.

[20]  A. Kelso,et al.  Independent regulation of granulocyte-macrophage colony-stimulating factor and multi-lineage colony-stimulating factor production in T lymphocyte clones. , 1986, Journal of Immunology.

[21]  M. Emerman,et al.  Quantitative analysis of gene suppression in integrated retrovirus vectors , 1986, Molecular and cellular biology.

[22]  D. Goeddel,et al.  Tumor necrosis factors: gene structure and biological activities. , 1986, Cold Spring Harbor symposia on quantitative biology.

[23]  H. Naora.  Gene ecology. A novel regulatory system in the gene expression network. , 1986, Rivista di biologia.

[24]  M. Barbacid,et al.  ras gene Amplification and malignant transformation , 1985, Molecular and cellular biology.

[25]  M. Emerman,et al.  Genes with promoters in retrovirus vectors can be independently suppressed by an epigenetic mechanism , 1984, Cell.

[26]  D. Spandidos,et al.  Elevated expression of the human ras oncogene family in premalignant and malignant tumours of the colorectum. , 1984, British Journal of Cancer.

[27]  B. Nadal-Ginard,et al.  Expression of the cardiac ventricular alpha- and beta-myosin heavy chain genes is developmentally and hormonally regulated. , 1984, The Journal of biological chemistry.

[28]  B. Nadal-Ginard,et al.  Cardiac alpha- and beta-myosin heavy chain genes are organized in tandem. , 1984, Proceedings of the National Academy of Sciences of the United States of America.

[29]  I. Verma,et al.  Expression of cellular oncogenes in human malignancies. , 1984, Science.

[30]  H. Blau,et al.  Isolation and characterization of full-length cDNA clones for human alpha-, beta-, and gamma-actin mRNAs: skeletal but not cytoplasmic actins have an amino-terminal cysteine that is subsequently removed , 1983, Molecular and cellular biology.

[31]  E. Chen,et al.  Complete nucleotide sequences of the T24 human bladder carcinoma oncogene and its normal homologue , 1983, Nature.

[32]  J. Bishop.  Cellular oncogenes and retroviruses. , 1983, Annual review of biochemistry.

[33]  N. Deacon,et al.  Clustered genes require extragenic territorial DNA sequences. , 1982, Differentiation; research in biological diversity.

[34]  R. Mulligan,et al.  Selection for animal cells that express the Escherichia coli gene coding for xanthine-guanine phosphoribosyltransferase. , 1981, Proceedings of the National Academy of Sciences of the United States of America.

[35]  R. Mulligan,et al.  Expression of a bacterial gene in mammalian cells. , 1980, Science.

[36]  K. Illmensee,et al.  Normal genetically mosaic mice produced from malignant teratocarcinoma cells. , 1975, Proceedings of the National Academy of Sciences of the United States of America.

[37]  H. Blau,et al.  Isolation and characterization of full-length cDNA clones for human alpha-, beta-, and gamma-actin mRNAs: skeletal but not cytoplasmic actins have an amino-terminal cysteine that is subsequently removed , 1983, Molecular and cellular biology.