S. aureus blocks efferocytosis of neutrophils by macrophages through the activity of its virulence factor alpha toxin
暂无分享,去创建一个
J. Suzich | B. Sellman | L. Miller | C. Kendall Stover | Lily I. Cheng | T. Cohen | O. Jones-Nelson | Taylor S. Cohen | Omari Jones-Nelson | Meghan Hotz | Lily Cheng | Lloyd S. Miller | JoAnn Suzich | Bret R. Sellman | Meghan J. Hotz | C. Stover | L. S. Miller
[1] Sam W. Lee,et al. Control of signaling-mediated clearance of apoptotic cells by the tumor suppressor p53 , 2015, Science.
[2] J. Suzich,et al. Staphylococcus aureus α toxin potentiates opportunistic bacterial lung infections , 2016, Science Translational Medicine.
[3] K. Ravichandran,et al. Phagocytosis of apoptotic cells in homeostasis , 2015, Nature Immunology.
[4] P. Buckley,et al. Antibody-Based Biologics and Their Promise to Combat Staphylococcus aureus Infections. , 2016, Trends in pharmacological sciences.
[5] L. Lau,et al. Matricellular Protein CCN1 Activates a Proinflammatory Genetic Program in Murine Macrophages , 2010, The Journal of Immunology.
[6] J. Cinatl,et al. Mcl-1-Mediated Impairment of the Intrinsic Apoptosis Pathway in Circulating Neutrophils from Critically Ill Patients Can Be Overcome by Fas Stimulation1 , 2009, The Journal of Immunology.
[7] Paul M. Orwin,et al. Exotoxins of Staphylococcus aureus. , 2000, Clinical microbiology reviews.
[8] N. Palaniyar,et al. NET balancing: a problem in inflammatory lung diseases , 2013, Front. Immun..
[9] A. Prince,et al. Toxin-Induced Necroptosis Is a Major Mechanism of Staphylococcus aureus Lung Damage , 2015, PLoS pathogens.
[10] Xiaodong Wang,et al. RIP3-mediated necrotic cell death accelerates systematic inflammation and mortality , 2015, Proceedings of the National Academy of Sciences.
[11] Yoon-Keun Kim,et al. CCN1 Secretion Induced by Cigarette Smoking Extracts Augments IL-8 Release from Bronchial Epithelial Cells , 2013, PloS one.
[12] Y. Tesfaigzi,et al. CCN1 secretion and cleavage regulate the lung epithelial cell functions after cigarette smoke. , 2014, American journal of physiology. Lung cellular and molecular physiology.
[13] A. Prince,et al. Activation of inflammasome signaling mediates pathology of acute P. aeruginosa pneumonia. , 2013, The Journal of clinical investigation.
[14] A. Prince,et al. Cystic fibrosis: a mucosal immunodeficiency syndrome , 2012, Nature Medicine.
[15] T. V. Kolesnikova,et al. CYR61, a product of a growth factor-inducible immediate early gene, promotes angiogenesis and tumor growth. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[16] Ki-Hyun Kim,et al. The Matricellular Protein CCN1 Mediates Neutrophil Efferocytosis in Cutaneous Wound Healing , 2015, Nature Communications.
[17] Naftali Kaminski,et al. Comprehensive gene expression profiles reveal pathways related to the pathogenesis of chronic obstructive pulmonary disease. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[18] K. Dalhoff,et al. Impaired neutrophil exocytosis in patients with severe pneumonia , 1999, Intensive Care Medicine.
[19] Robin R. Chamberland,et al. Staphylococcus aureus α-hemolysin mediates virulence in a murine model of severe pneumonia through activation of the NLRP3 inflammasome. , 2012, The Journal of infectious diseases.
[20] Jun Xu,et al. Extracellular histones are major mediators of death in sepsis , 2009, Nature Medicine.
[21] A. Prince,et al. Bacterial Pathogens Activate a Common Inflammatory Pathway through IFNλ Regulation of PDCD4 , 2013, PLoS pathogens.
[22] Mallary C Greenlee-Wacker,et al. Phagocytosis of Staphylococcus aureus by Human Neutrophils Prevents Macrophage Efferocytosis and Induces Programmed Necrosis , 2014, The Journal of Immunology.
[23] Seamus J. Martin,et al. Inflammatory outcomes of apoptosis, necrosis and necroptosis , 2014, Biological chemistry.
[24] T. Standiford,et al. Extracellular histones are essential effectors of C5aR‐ and C5L2‐mediated tissue damage and inflammation in acute lung injury , 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[25] K. Moore,et al. Response to Staphylococcus aureus requires CD36-mediated phagocytosis triggered by the COOH-terminal cytoplasmic domain , 2005, The Journal of cell biology.
[26] A. Prince,et al. CD4+ T cells promote the pathogenesis of Staphylococcus aureus pneumonia. , 2015, The Journal of infectious diseases.
[27] S. Fortune,et al. Efferocytosis is an innate antibacterial mechanism. , 2012, Cell host & microbe.
[28] Simon C Watkins,et al. CYR61 (CCN1) Is Essential for Placental Development and Vascular Integrity , 2002, Molecular and Cellular Biology.
[29] M. Matthay,et al. The acute respiratory distress syndrome. , 1996, The New England journal of medicine.
[30] J. Hudcová,et al. α-Hemolysin activity of methicillin-susceptible Staphylococcus aureus predicts ventilator-associated pneumonia. , 2014, American journal of respiratory and critical care medicine.
[31] L. Lau,et al. Cyr61, a product of a growth factor-inducible immediate-early gene, promotes cell proliferation, migration, and adhesion , 1996, Molecular and cellular biology.
[32] K. Preissner,et al. Neutrophil Extracellular Traps Directly Induce Epithelial and Endothelial Cell Death: A Predominant Role of Histones , 2012, PloS one.
[33] M. Febbraio,et al. CD36 Is Essential for Regulation of the Host Innate Response to Staphylococcus aureus α-Toxin–Mediated Dermonecrosis , 2015, The Journal of Immunology.
[34] A. Condliffe,et al. The neutrophil in chronic obstructive pulmonary disease. , 2013, American journal of respiratory cell and molecular biology.
[35] I. Autenrieth,et al. Bacteria induce CTGF and CYR61 expression in epithelial cells in a lysophosphatidic acid receptor-dependent manner. , 2008, International journal of medical microbiology : IJMM.
[36] I. Inoshima,et al. A Staphylococcus aureus Pore-Forming Toxin Subverts the Activity of ADAM10 to Cause Lethal Infection , 2011, Nature Medicine.
[37] J. Suzich,et al. Assessment of an Anti-Alpha-Toxin Monoclonal Antibody for Prevention and Treatment of Staphylococcus aureus-Induced Pneumonia , 2013, Antimicrobial Agents and Chemotherapy.
[38] B. Wilson,et al. Survival of Staphylococcus aureus Inside Neutrophils Contributes to Infection1 , 2000, The Journal of Immunology.