Human Insulin Resistance Is Associated With Increased Plasma Levels of 12α-Hydroxylated Bile Acids
暂无分享,去创建一个
[1] Gabi Kastenmüller,et al. Early Metabolic Markers of the Development of Dysglycemia and Type 2 Diabetes and Their Physiological Significance , 2013, Diabetes.
[2] F. Schütz,et al. Hepatic glucose sensing is required to preserve β cell glucose competence. , 2013, The Journal of clinical investigation.
[3] Barbara Gross,et al. Bile acid receptors as targets for the treatment of dyslipidemia and cardiovascular disease , 2012, Journal of Lipid Research.
[4] F. Gonzalez,et al. Bile Acids Acutely Stimulate Insulin Secretion of Mouse β-Cells via Farnesoid X Receptor Activation and KATP Channel Inhibition , 2012, Diabetes.
[5] A. Groen,et al. Bile acid sequestrants: more than simple resins , 2012, Current opinion in lipidology.
[6] D. Accili,et al. Impaired generation of 12-hydroxylated bile acids links hepatic insulin signaling with dyslipidemia. , 2012, Cell metabolism.
[7] E. Oetjen. Body Weight, Not Insulin Sensitivity or Secretion, May Predict Spontaneous Weight Changes in Nondiabetic and Prediabetic Subjects: The RISC Study , 2012 .
[8] M. Walker,et al. Influence of Hyperinsulinemia and Insulin Resistance on In Vivo β-Cell Function , 2011, Diabetes.
[9] M. Walker,et al. In fl uence of Hyperinsulinemia and Insulin Resistance on In Vivo b -Cell Function Their Role in Human b -Cell Dysfunction , 2011 .
[10] Christian Gieger,et al. Metabolic Footprint of Diabetes: A Multiplatform Metabolomics Study in an Epidemiological Setting , 2010, PloS one.
[11] F. Kuipers,et al. Improved glycemic control with colesevelam treatment in patients with type 2 diabetes is not directly associated with changes in bile acid metabolism , 2010, Hepatology.
[12] S. Woods,et al. Intestinal adaptation after ileal interposition surgery increases bile acid recycling and protects against obesity-related comorbidities. , 2010, American journal of physiology. Gastrointestinal and liver physiology.
[13] D. Accili,et al. Hepatic FoxO1 Ablation Exacerbates Lipid Abnormalities during Hyperglycemia* , 2010, The Journal of Biological Chemistry.
[14] S. Fiorucci,et al. The bile acid sensor FXR regulates insulin transcription and secretion. , 2010, Biochimica et biophysica acta.
[15] A. Moschetta,et al. Deciphering the nuclear bile acid receptor FXR paradigm , 2010, Nuclear receptor signaling.
[16] J. Chiang,et al. Bile acids: regulation of synthesis , 2009, Journal of Lipid Research.
[17] J. Holst,et al. Serum Bile Acids Are Higher in Humans With Prior Gastric Bypass: Potential Contribution to Improved Glucose and Lipid Metabolism , 2009, Obesity.
[18] P. Dent,et al. Bile acids as regulatory molecules , 2009, Journal of Lipid Research.
[19] Beverley Balkau,et al. Fatty liver is associated with insulin resistance, risk of coronary heart disease, and early atherosclerosis in a large European population , 2009, Hepatology.
[20] T. Osborne,et al. FGF15/FGFR4 Integrates Growth Factor Signaling with Hepatic Bile Acid Metabolism and Insulin Action* , 2009, Journal of Biological Chemistry.
[21] B. Staels,et al. Role of bile acids and bile acid receptors in metabolic regulation. , 2009, Physiological reviews.
[22] Johan Auwerx,et al. Targeting bile-acid signalling for metabolic diseases , 2008, Nature Reviews Drug Discovery.
[23] C. Kahn,et al. Hepatic insulin resistance directly promotes formation of cholesterol gallstones , 2008, Nature Medicine.
[24] Margaret F. Gregor,et al. Thematic review series: Adipocyte Biology. Adipocyte stress: the endoplasmic reticulum and metabolic disease Published, JLR Papers in Press, May 9, 2007. , 2007, Journal of Lipid Research.
[25] Margaret F. Gregor,et al. Adipocyte stress: the endoplasmic reticulum and metabolic disease , 2007 .
[26] D. Accili,et al. FoxO1 protects against pancreatic beta cell failure through NeuroD and MafA induction. , 2005, Cell metabolism.
[27] Jesse D. Martinez,et al. Bile acid induces hydrophobicity-dependent membrane alterations. , 2005, Biochimica et biophysica acta.
[28] B. Staels,et al. Glucose regulates the expression of the farnesoid X receptor in liver. , 2004, Diabetes.
[29] N. de Pedro,et al. The Farnesoid X Receptor Controls Gene Expression in a Ligand- and Promoter-selective Fashion* , 2004, Journal of Biological Chemistry.
[30] M. Walker,et al. The EGIR-RISC STUDY (The European group for the study of insulin resistance: relationship between insulin sensitivity and cardiovascular disease risk): I. Methodology and Objectives , 2004, Diabetologia.
[31] S. Tazuma,et al. Feeding natural hydrophilic bile acids inhibits intestinal cholesterol absorption: studies in the gallstone-susceptible mouse. , 2003, American journal of physiology. Gastrointestinal and liver physiology.
[32] U. Beuers,et al. Ursodeoxycholic acid in cholestatic liver disease: Mechanisms of action and therapeutic use revisited , 2002, Hepatology.
[33] B. Aronow,et al. Independent and overlapping transcriptional activation during liver development and regeneration in mice , 2002, Hepatology.
[34] A. Hofmann,et al. The continuing importance of bile acids in liver and intestinal disease. , 1999, Archives of internal medicine.
[35] M. Makishima,et al. Identification of a nuclear receptor for bile acids. , 1999, Science.
[36] J. Lehmann,et al. Bile acids: natural ligands for an orphan nuclear receptor. , 1999, Science.
[37] A Mari,et al. How to measure insulin sensitivity , 1998, Journal of hypertension.
[38] H. Kurihara,et al. Altered bile acid metabolism related to atherosclerosis in alloxan diabetic rats. , 1996, Journal of atherosclerosis and thrombosis.
[39] D. Heuman. Quantitative estimation of the hydrophilic-hydrophobic balance of mixed bile salt solutions. , 1989, Journal of lipid research.
[40] T. Akiyoshi,et al. Cholesterol gallstones in alloxan-diabetic mice. , 1986, Journal of lipid research.
[41] T. Akiyoshi,et al. Altered Bile Acid Metabolism in Nonobese, Spontaneously Diabetic (NOD) Mice , 1985, Diabetes.
[42] A. Hofmann,et al. Physicochemical properties of bile acids and their relationship to biological properties: an overview of the problem. , 1984, Journal of lipid research.
[43] D. Billington,et al. Effects of bile salts on the plasma membranes of isolated rat hepatocytes. , 1980, The Biochemical journal.
[44] R. Coleman,et al. Effects of different bile salts upon the composition and morphology of a liver plasma membrane preparation. Deoxycholate is more membrane damaging than cholate and its conjugates. , 1977, Biochimica et biophysica acta.