Direct, activating interaction between glycogen synthase kinase-3β and p53 after DNA damage
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Ling Song | Richard S. Jope | Xinbin Chen | A. Zmijewska | R. Jope | G. Johnson | G. Bijur | Ling Song | J. Zmijewski | Gail V. W. Johnson | Xinbin Chen | Piyajit Watcharasit | Piyajit Watcharasit | Gautam N. Bijur | Jaroslaw W. Zmijewski | Anna Zmijewska | G. N. Bijur | Anna A. Zmijewska
[1] J Mao,et al. Axin and Frat1 interact with Dvl and GSK, bridging Dvl to GSK in Wnt‐mediated regulation of LEF‐1 , 1999, The EMBO journal.
[2] Lithium attenuates p53 levels in human neuroblastoma SH-SY5Y cells. , 1999, Neuroreport.
[3] D. Chuang,et al. Long Term Lithium Treatment Suppresses p53 and Bax Expression but Increases Bcl-2 Expression , 1999, The Journal of Biological Chemistry.
[4] J C Adair,et al. Is it Alzheimer's? , 1998, Hospital practice.
[5] D. Chuang,et al. Neuroprotective effects of chronic lithium on focal cerebral ischemia in rats , 1998, Neuroreport.
[6] R. Jope,et al. Glycogen Synthase Kinase-3β Facilitates Staurosporine- and Heat Shock-induced Apoptosis , 2000, The Journal of Biological Chemistry.
[7] D. Kimelman,et al. Role of Glycogen Synthase Kinase-3β in Neuronal Apoptosis Induced by Trophic Withdrawal , 2000, The Journal of Neuroscience.
[8] J. Testa,et al. The phosphatidylinositol 3-kinase/AKT signal transduction pathway plays a critical role in the expression of p21WAF1/CIP1/SDI1 induced by cisplatin and paclitaxel. , 2000, Cancer research.
[9] D. M. Ferkey,et al. GSK-3: new thoughts on an old enzyme. , 2000, Developmental biology.
[10] Y. Kinoshita,et al. The role of p53 in neuronal cell death , 2000, Cell Death and Differentiation.
[11] J. Woodgett,et al. Modulation of the glycogen synthase kinase‐3 family by tyrosine phosphorylation. , 1993, The EMBO journal.
[12] Xinbin Chen,et al. Definition of the p53 Functional Domains Necessary for Inducing Apoptosis* , 2000, The Journal of Biological Chemistry.
[13] A. Levine,et al. Surfing the p53 network , 2000, Nature.
[14] M. Vitek,et al. Akt Activation Protects Hippocampal Neurons from Apoptosis by Inhibiting Transcriptional Activity of p53* , 2001, The Journal of Biological Chemistry.
[15] P. Cohen,et al. Specificity and mechanism of action of some commonly used protein kinase inhibitors , 2000 .
[16] F. McCormick,et al. Phosphoinositide 3-OH Kinase (PI3K) and PKB/Akt Delay the Onset of p53-mediated, Transcriptionally Dependent Apoptosis* , 1999, The Journal of Biological Chemistry.
[17] R. Jope,et al. The multifaceted roles of glycogen synthase kinase 3β in cellular signaling , 2001, Progress in Neurobiology.
[18] G. Cooper,et al. Role of Glycogen Synthase Kinase-3 in the Phosphatidylinositol 3-Kinase/Akt Cell Survival Pathway* , 1998, The Journal of Biological Chemistry.
[19] Benjamin Geiger,et al. Down-Regulation of β-Catenin by Activated p53 , 2001, Molecular and Cellular Biology.
[20] P. Cohen,et al. Inactivation of glycogen synthase kinase-3 beta by phosphorylation: new kinase connections in insulin and growth-factor signalling. , 1993, The Biochemical journal.
[21] C. Phiel,et al. Molecular targets of lithium action. , 2003, Annual review of pharmacology and toxicology.
[22] D. M. Ferkey,et al. Interaction among Gsk-3, Gbp, Axin, and APC in Xenopus Axis Specification , 2000, The Journal of cell biology.
[23] James R. Woodgett,et al. Lithium inhibits glycogen synthase kinase-3 activity and mimics Wingless signalling in intact cells , 1996, Current Biology.
[24] H. Manji,et al. The Mood‐Stabilizing Agent Valproate Inhibits the Activity of Glycogen Synthase Kinase‐3 , 2000, Journal of neurochemistry.
[25] Rachel L. Allen,et al. Defying death after DNA damage , 2000, Nature.
[26] C. Pozniak,et al. Neuronal life and death: an essential role for the p53 family , 2000, Cell Death and Differentiation.
[27] D. Melton,et al. A molecular mechanism for the effect of lithium on development. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[28] R. Jope,et al. Proapoptotic Stimuli Induce Nuclear Accumulation of Glycogen Synthase Kinase-3β* , 2001, The Journal of Biological Chemistry.
[29] K. Imahori,et al. Tau protein kinase I is essential for amyloid beta-protein-induced neurotoxicity. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[30] S. Elledge,et al. The DNA damage response: putting checkpoints in perspective , 2000, Nature.
[31] J. Bartek,et al. Pathways governing G1/S transition and their response to DNA damage , 2001, FEBS letters.
[32] Akira Kikuchi,et al. Axin, a negative regulator of the Wnt signaling pathway, forms a complex with GSK‐3β and β‐catenin and promotes GSK‐3β‐dependent phosphorylation of β‐catenin , 1998 .
[33] D. M. Ferkey,et al. GBP, an Inhibitor of GSK-3, Is Implicated in Xenopus Development and Oncogenesis , 1998, Cell.