Expression of alternatively spliced sodium channel alpha-subunit genes. Unique splicing patterns are observed in dorsal root ganglia.
暂无分享,去创建一个
Zhengyan Kan | John Castle | J. Castle | P. Garrett-engele | Jason M. Johnson | C. Raymond | C. Armour | Z. Kan | N. Tsinoremas | Christopher D Armour | Jason M Johnson | Christopher K Raymond | Philip Garrett-Engele | Nicholas Tsinoremas | Zhengyan Kan
[1] M. Zimmermann,et al. Ethical guidelines for investigations of experimental pain in conscious animals , 1983, Pain.
[2] R. Sarao,et al. Developmentally regulated alternative RNA splicing of rat brain sodium channel mRNAs. , 1991, Nucleic acids research.
[3] S. Dib-Hajj,et al. Down-Regulation of Transcripts for Na Channel α -SNS in Spinal Sensory Neurons Following Axotomy , 1996 .
[4] R. Eglen,et al. Functional Analysis of a Voltage‐Gated Sodium Channel and Its Splice Variant from Rat Dorsal Root Ganglia , 1998, Journal of neurochemistry.
[5] M. Meisler,et al. Exon organization, coding sequence, physical mapping, and polymorphic intragenic markers for the human neuronal sodium channel gene SCN8A. , 1998, Genomics.
[6] A. L. Goldin,et al. Alternative Splicing of an Insect Sodium Channel Gene Generates Pharmacologically Distinct Sodium Channels , 2002, The Journal of Neuroscience.
[7] William A Catterall,et al. Overview of the voltage-gated sodium channel family , 2003, Genome Biology.
[8] A. J. Castiglioni,et al. Cell-Specific Alternative Splicing Increases Calcium Channel Current Density in the Pain Pathway , 2004, Neuron.
[9] Juha Muilu,et al. Conservation of human alternative splice events in mouse. , 2003, Nucleic acids research.
[10] Stephen W. Edwards,et al. Microarray Standard Data Set and Figures of Merit for Comparing Data Processing Methods and Experiment Designs , 2003, Bioinform..
[11] S. Dib-Hajj,et al. Voltage-gated sodium channels and the molecular pathogenesis of pain: a review. , 2000, Journal of rehabilitation research and development.
[12] David States,et al. Selecting for functional alternative splices in ESTs. , 2002, Genome research.
[13] J. M. Ritchie,et al. Cloning of a sodium channel alpha subunit from rabbit Schwann cells. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[14] Yudong D. He,et al. Expression profiling using microarrays fabricated by an ink-jet oligonucleotide synthesizer , 2001, Nature Biotechnology.
[15] A. L. Goldin,et al. A neutral amino acid change in segment IIS4 dramatically alters the gating properties of the voltage-dependent sodium channel. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[16] D. Gerhold,et al. Chronic neuropathic pain is accompanied by global changes in gene expression and shares pathobiology with neurodegenerative diseases , 2002, Neuroscience.
[17] R. Quatrano. Genomics , 1998, Plant Cell.
[18] K. Schaller,et al. Alternatively spliced sodium channel transcripts in brain and muscle , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[19] 김삼묘,et al. “Bioinformatics” 특집을 내면서 , 2000 .
[20] P. Yarowsky,et al. Mutually exclusive exon splicing of type III brain sodium channel alpha subunit RNA generates developmentally regulated isoforms in rat brain. , 1993, The Journal of biological chemistry.
[21] R. LaMotte,et al. Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain , 1999, PAIN®.
[22] M. Akimoto,et al. Gene microarray analysis of experimental glaucomatous retina from cynomologous monkey. , 2003, Investigative ophthalmology & visual science.
[23] Gail Mandel,et al. Nomenclature of Voltage-Gated Sodium Channels , 2000, Neuron.
[24] G. Stephanopoulos,et al. A compendium of gene expression in normal human tissues. , 2001, Physiological genomics.
[25] T. Jessell. PAIN , 1982, The Lancet.
[26] M. Meisler,et al. Alternative Splicing of the Sodium Channel SCN8A Predicts a Truncated Two-domain Protein in Fetal Brain and Non-neuronal Cells* , 1997, The Journal of Biological Chemistry.
[27] J. Castle,et al. Genome-Wide Survey of Human Alternative Pre-mRNA Splicing with Exon Junction Microarrays , 2003, Science.
[28] K. Wong,et al. A Novel Tetrodotoxin-sensitive, Voltage-gated Sodium Channel Expressed in Rat and Human Dorsal Root Ganglia* , 1997, The Journal of Biological Chemistry.
[29] 宁北芳,et al. 疟原虫var基因转换速率变化导致抗原变异[英]/Paul H, Robert P, Christodoulou Z, et al//Proc Natl Acad Sci U S A , 2005 .
[30] Jin Mo Chung,et al. An experimental model for peripheral neuropathy produced by segmental spinal nerve ligation in the rat , 1992, PAIN.
[31] C. Bollensdorff,et al. Mouse heart Na+ channels: primary structure and function of two isoforms and alternatively spliced variants. , 2002, American journal of physiology. Heart and circulatory physiology.
[32] W. Catterall,et al. From Ionic Currents to Molecular Mechanisms The Structure and Function of Voltage-Gated Sodium Channels , 2000, Neuron.
[33] A. Orth,et al. Large-scale analysis of the human and mouse transcriptomes , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[34] Christopher J. Lee,et al. Alternative splicing in the human, mouse and rat genomes is associated with an increased frequency of exon creation and/or loss , 2003, Nature Genetics.
[35] Eric E Schadt,et al. Optimization of oligonucleotide arrays and RNA amplification protocols for analysis of transcript structure and alternative splicing , 2003, Genome Biology.
[36] M. Meisler,et al. Evolution and diversity of mammalian sodium channel genes. , 1999, Genomics.
[37] D. Wilkin,et al. Neuron , 2001, Brain Research.
[38] S. Dib-Hajj,et al. Changes in Expression of Two Tetrodotoxin-Resistant Sodium Channels and Their Currents in Dorsal Root Ganglion Neurons after Sciatic Nerve Injury But Not Rhizotomy , 2000, The Journal of Neuroscience.