Nitric Oxide Modulates the CBF Response to Increased Extracellular Potassium

[1]  A Villringer,et al.  Coupling of cerebral blood flow to neuronal activation: role of adenosine and nitric oxide. , 1994, The American journal of physiology.

[2]  X. Xu,et al.  SIN-1 reverses attenuation of hypercapnic cerebrovasodilation by nitric oxide synthase inhibitors. , 1994, The American journal of physiology.

[3]  Y. Nakaya,et al.  Nonendothelial‐derived nitric oxide activates the ATP‐sensitive K+ channel of vascular smooth muscle cells , 1994, FEBS letters.

[4]  J. Kirsch,et al.  A cholinergic agonist induces cerebral hyperemia in isoflurane- but not pentobarbital-anesthetized dogs. , 1994, Anesthesia and analgesia.

[5]  R. Cohen,et al.  Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscle , 1994, Nature.

[6]  R. Dacey,et al.  N omega-nitro-L-arginine constricts cerebral arterioles without increasing intracellular calcium levels. , 1994, The American journal of physiology.

[7]  M. Moskowitz,et al.  Nitric Oxide Synthase Inhibition and Cerebrovascular Regulation , 1994, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[8]  M. Moskowitz,et al.  Importance of Nitric Oxide Synthase Inhibition to the Attenuated Vascular Responses Induced by Topical L-Nitroarginine during Vibrissal Stimulation , 1994, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[9]  F. Chabaud,et al.  A vascular smooth muscles nitric oxide relaxation by a mechanism distinct of calcium changes. , 1994, Life sciences.

[10]  W. Mathews,et al.  Role of calcium-activated K+ channels in vasodilation induced by nitroglycerine, acetylcholine and nitric oxide. , 1993, The Journal of pharmacology and experimental therapeutics.

[11]  B. E. Robertson,et al.  cGMP-dependent protein kinase activates Ca-activated K channels in cerebral artery smooth muscle cells. , 1993, The American journal of physiology.

[12]  C. Iadecola,et al.  Regulation of the cerebral microcirculation during neural activity: is nitric oxide the missing link? , 1993, Trends in Neurosciences.

[13]  Ulrich Dirnagl,et al.  Blockade of Nitric Oxide Synthesis in Rats Strongly Attenuates the CBF Response to Extracellular Acidosis , 1993, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[14]  A Villringer,et al.  Characterization of CBF response to somatosensory stimulation: model and influence of anesthetics. , 1993, The American journal of physiology.

[15]  A. Villringer,et al.  Role of nitric oxide in the coupling of cerebral blood flow to neuronal activation in rats , 1993, Neuroscience Letters.

[16]  C. Lajoie,et al.  Ca(2+)-independent isoforms of protein kinase C differentially translocate in smooth muscle. , 1992, The American journal of physiology.

[17]  C. Iadecola,et al.  Focal elevations in neocortical interstitial K+ produced by stimulation of the fastigial nucleus in rat , 1991, Brain Research.

[18]  K. Okumura,et al.  Prostaglandin H2 as an endothelium-derived contracting factor and its interaction with endothelium-derived nitric oxide. , 1991, Journal of hypertension.

[19]  A. A. Parsons,et al.  Analysis of Acetylcholine-Induced Relaxation of Rabbit Isolated Middle Cerebral Artery: Effects of Inhibitors of Nitric Oxide Synthesis, Na,K-ATPase, and ATP-Sensitive K Channels , 1991, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[20]  H. Brockerhoff,et al.  Interactionof Anesthetic Barbiturates with the Phosphoinositide‐Dependent Pathway of Signal Transduction a , 1991, Annals of the New York Academy of Sciences.

[21]  J. Angus,et al.  Evidence that acetylcholine‐mediated hyperpolarization of the rat small mesenteric artery does not involve the K+ channel opened by cromakalim , 1991, British journal of pharmacology.

[22]  W. Halpern,et al.  Potassium dilates rat cerebral arteries by two independent mechanisms. , 1990, The American journal of physiology.

[23]  T. Martin,et al.  Atrial natriuretic peptide-dependent phosphorylation of smooth muscle cell particulate fraction proteins is mediated by cGMP-dependent protein kinase. , 1989, The Journal of biological chemistry.

[24]  U. Dirnagl,et al.  Continuous Measurement of Cerebral Cortical Blood Flow by Laser—Doppler Flowmetry in a Rat Stroke Model , 1989, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[25]  T. Yanagisawa,et al.  Nitroglycerin relaxes canine coronary arterial smooth muscle without reducing intracellular Ca2+ concentrations measured with fura‐2 , 1989, British journal of pharmacology.

[26]  F. Edwards,et al.  Inward rectification in rat cerebral arterioles; involvement of potassium ions in autoregulation. , 1988, The Journal of physiology.

[27]  O B Paulson,et al.  Does the release of potassium from astrocyte endfeet regulate cerebral blood flow? , 1987, Science.

[28]  C. Taylor,et al.  Inhibitory effects of a synthetic atrial peptide on contractions and 45Ca fluxes in vascular smooth muscle. , 1986, The Journal of pharmacology and experimental therapeutics.

[29]  H. Karaki,et al.  Mechanism of inhibitory action of sodium nitroprusside in vascular smooth muscle of rabbit aorta. , 1986, Archives internationales de pharmacodynamie et de therapie.

[30]  R. Macdonald,et al.  Anticonvulsant drugs: mechanisms of action. , 1986, Advances in neurology.

[31]  R. Hester Effects of 2-nicotinamidoethyl nitrate on agonist-sensitive Ca++ release and Ca++ entry in rabbit aorta. , 1985, The Journal of pharmacology and experimental therapeutics.

[32]  R. Miller,et al.  Mechanisms of Smooth Muscle Relaxation , 1985 .

[33]  F. Murad,et al.  Effect of ouabain and alterations in potassium concentration on relaxation induced by sodium nitroprusside. , 1983, Blood vessels.

[34]  D. Lübbers,et al.  Capillary flow in the brain cortex during changes in oxygen supply and state of activation. , 1978, Ciba Foundation symposium.

[35]  D. Bohr,et al.  Potassium-induced relaxation as an indicator of Na+-K+ ATPase activity in vascular smooth muscle. , 1978, Blood vessels.

[36]  W. Kuschinsky,et al.  Perivascular Potassium and pH as Determinants of Local Pial Arterial Diameter in Cats: A MICROAPPLICATION STUDY , 1972, Circulation research.