Programmed Cell Death in Animal Development and Disease
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Richard L. Frock | F. Alt | S. Monti | D. Neuberg | H. Steller | V. Choi | Yu Zhang | M. Gostissa | R. Chiarle | S. Lewis | B. Molinie | C. Giallourakis | M. Compagno | Y. Fuchs | Daniel J. Malkin | Yu-jui Ho | D. Myers
[1] A. D. De Marzo,et al. Transcription-Induced DNA Double Strand Breaks: Both Oncogenic Force and Potential Therapeutic Target? , 2011, Clinical Cancer Research.
[2] F. Alt,et al. Mechanisms that promote and suppress chromosomal translocations in lymphocytes. , 2011, Annual review of immunology.
[3] T. Cathomen,et al. Zinc-finger nuclease based genome surgery: it's all about specificity. , 2011, Current gene therapy.
[4] S. Grizot,et al. The I-CreI meganuclease and its engineered derivatives: applications from cell modification to gene therapy. , 2011, Protein engineering, design & selection : PEDS.
[5] M. Nussenzweig,et al. AID targeting in antibody diversity. , 2011, Advances in immunology.
[6] M. Nussenzweig,et al. Deep-sequencing identification of the genomic targets of the cytidine deaminase AID and its cofactor RPA in B lymphocytes , 2011, Nature Immunology.
[7] F. Alt,et al. Elements between the IgH variable (V) and diversity (D) clusters influence antisense transcription and lineage-specific V(D)J recombination , 2010, Proceedings of the National Academy of Sciences.
[8] H. Steller,et al. Coupling of Apoptosis and L/R Patterning Controls Stepwise Organ Looping , 2010, Current Biology.
[9] Erin L. Doyle,et al. Targeting DNA Double-Strand Breaks with TAL Effector Nucleases , 2010, Genetics.
[10] M. Lieber,et al. The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway. , 2010, Annual review of biochemistry.
[11] Daniel G. Miller,et al. Frequent endonuclease cleavage at off-target locations in vivo. , 2010, Molecular therapy : the journal of the American Society of Gene Therapy.
[12] M. Nussenzweig,et al. Origin of Chromosomal Translocations in Lymphoid Cancer , 2010, Cell.
[13] Ira M. Hall,et al. Genome-wide mapping and assembly of structural variant breakpoints in the mouse genome. , 2010, Genome research.
[14] M. Jasin,et al. Alternative end-joining is suppressed by the canonical NHEJ component Xrcc4/ligase IV during chromosomal translocation formation , 2010, Nature Structural &Molecular Biology.
[15] Michael S. Becker,et al. The role of mechanistic factors in promoting chromosomal translocations found in lymphoid and other cancers. , 2010, Advances in immunology.
[16] Jie Zhang,et al. Nuclear Receptor-Induced Chromosomal Proximity and DNA Breaks Underlie Specific Translocations in Cancer , 2009, Cell.
[17] S. Kulkarni,et al. Aberrantly resolved RAG-mediated DNA breaks in Atm-deficient lymphocytes target chromosomal breakpoints in cis , 2009, Proceedings of the National Academy of Sciences.
[18] I. Amit,et al. Comprehensive mapping of long range interactions reveals folding principles of the human genome , 2011 .
[19] Steven J. M. Jones,et al. Circos: an information aesthetic for comparative genomics. , 2009, Genome research.
[20] F. Alt,et al. Mechanisms promoting translocations in editing and switching peripheral B cells , 2009, Nature.
[21] M. Stratton,et al. The cancer genome , 2009, Nature.
[22] D. Schatz,et al. Balancing AID and DNA repair during somatic hypermutation. , 2009, Trends in immunology.
[23] F. Alt,et al. Chromosomal location targets different MYC family gene members for oncogenic translocations , 2009, Proceedings of the National Academy of Sciences.
[24] Leighton J. Core,et al. Nascent RNA Sequencing Reveals Widespread Pausing and Divergent Initiation at Human Promoters , 2008, Science.
[25] M. Nussenzweig,et al. AID Is Required for the Chromosomal Breaks in c-myc that Lead to c-myc/IgH Translocations , 2008, Cell.
[26] D. Spector,et al. 53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility , 2008, Nature.
[27] Mark Johnson,et al. NCBI BLAST: a better web interface , 2008, Nucleic Acids Res..
[28] D. Schatz,et al. Two levels of protection for the B cell genome during somatic hypermutation , 2008, Nature.
[29] T. Ried,et al. AID-deficient Bcl-xL transgenic mice develop delayed atypical plasma cell tumors with unusual Ig/Myc chromosomal rearrangements , 2007, The Journal of experimental medicine.
[30] Michael M. Murphy,et al. IgH class switching and translocations use a robust non-classical end-joining pathway , 2007, Nature.
[31] A. Aguilera,et al. Activation-induced cytidine deaminase action is strongly stimulated by mutations of the THO complex , 2007, Proceedings of the National Academy of Sciences.
[32] Tom Misteli,et al. Spatial genome organization in the formation of chromosomal translocations. , 2007, Seminars in cancer biology.
[33] Frederick W. Alt,et al. References and Notes Supporting Online Material Materials and Methods Figs. S1 to S3 Table S1 References Antibody Class Switching Mediated by Yeast Endonuclease–generated Dna Breaks , 2022 .
[34] F. Alt,et al. Evolution of the immunoglobulin heavy chain class switch recombination mechanism. , 2007, Advances in immunology.
[35] J. Manley,et al. Cotranscriptional processes and their influence on genome stability. , 2006, Genes & development.
[36] Andrew D Griffiths,et al. Amplification of complex gene libraries by emulsion PCR , 2006, Nature Methods.
[37] K. Rajewsky,et al. Canonical NF-kappaB activity, dispensable for B cell development, replaces BAFF-receptor signals and promotes B cell proliferation upon activation. , 2006, Immunity.
[38] Michel C. Nussenzweig,et al. Role of genomic instability and p53 in AID-induced c-myc–Igh translocations , 2006, Nature.
[39] Michael M. Murphy,et al. H2AX prevents DNA breaks from progressing to chromosome breaks and translocations. , 2006, Molecular cell.
[40] P. Pandolfi,et al. Breaking the rules of cancer , 2006, Nature Medicine.
[41] M. Cogné,et al. Interallelic Class Switch Recombination Contributes Significantly to Class Switching in Mouse B Cells 1 , 2005, The Journal of Immunology.
[42] D. Schatz,et al. Identification of an AID-independent pathway for chromosomal translocations between the Igh switch region and Myc , 2004, Nature Immunology.
[43] M. Lieber,et al. R-loops at immunoglobulin class switch regions in the chromosomes of stimulated B cells , 2003, Nature Immunology.
[44] F. Alt,et al. Internal IgH class switch region deletions are position-independent and enhanced by AID expression , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[45] F. Alt,et al. Unrepaired DNA Breaks in p53-Deficient Cells Lead to Oncogenic Gene Amplification Subsequent to Translocations , 2002, Cell.
[46] W. J. Kent,et al. BLAT--the BLAST-like alignment tool. , 2002, Genome research.
[47] A. Aguilera. The connection between transcription and genomic instability , 2002, The EMBO journal.
[48] Riccardo Dalla-Favera,et al. Mechanisms of chromosomal translocations in B cell lymphomas , 2001, Oncogene.
[49] Gouri Nanjangud,et al. Hypermutation of multiple proto-oncogenes in B-cell diffuse large-cell lymphomas , 2001, Nature.
[50] T. Honjo,et al. Class Switch Recombination and Hypermutation Require Activation-Induced Cytidine Deaminase (AID), a Potential RNA Editing Enzyme , 2000, Cell.
[51] F. Alt,et al. The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[52] M. Jasin,et al. Genetic manipulation of genomes with rare-cutting endonucleases. , 1996, Trends in genetics : TIG.
[53] S. Lukyanov,et al. An improved PCR method for walking in uncloned genomic DNA. , 1995, Nucleic acids research.