Constitutive activation of c-kit in FMA3 murine mastocytoma cells caused by deletion of seven amino acids at the juxtamembrane domain.
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Y. Matsuzawa | H. Kitayama | Y. Kitamura | M. Morimoto | K. Hashimoto | Y. Kanakura | Y. Moriyama | T. Tsujimura
[1] Y. Matsuzawa,et al. Substitution of an aspartic acid results in constitutive activation of c-kit receptor tyrosine kinase in a rat tumor mast cell line RBL-2H3. , 1995, International archives of allergy and immunology.
[2] H. Kitayama,et al. Constitutively activating mutations of c-kit receptor tyrosine kinase confer factor-independent growth and tumorigenicity of factor-dependent hematopoietic cell lines. , 1995, Blood.
[3] al. et,et al. Activation of RET as a dominant transforming gene by germline mutations of MEN2A and MEN2B , 1995, Science.
[4] A. Ullrich,et al. Oncogenic activation of v-kit involves deletion of a putative tyrosine-substrate interaction site. , 1995, Oncogene.
[5] W. Muller,et al. Novel activating mutations in the neu proto-oncogene involved in induction of mammary tumors. , 1994, Molecular and Cellular Biology.
[6] L. Rohrschneider,et al. Tyrosine 569 in the c-Fms juxtamembrane domain is essential for kinase activity and macrophage colony-stimulating factor-dependent internalization , 1994, Molecular and cellular biology.
[7] Y. Matsuzawa,et al. Ligand-independent activation of c-kit receptor tyrosine kinase in a murine mastocytoma cell line P-815 generated by a point mutation , 1994 .
[8] H. Serve,et al. Tyrosine residue 719 of the c-kit receptor is essential for binding of the P85 subunit of phosphatidylinositol (PI) 3-kinase and for c-kit-associated PI 3-kinase activity in COS-1 cells. , 1994, The Journal of biological chemistry.
[9] R. Hofstra,et al. A mutation in the RET proto-oncogene associated with multiple endocrine neoplasia type 2B and sporadic medullary thyroid carcinoma , 1994, Nature.
[10] T. Kubota,et al. Sequence of a bovine c-kit proto-oncogene cDNA. , 1994, Gene.
[11] Y. Matsuzawa,et al. Ligand-independent activation of c-kit receptor tyrosine kinase in a murine mastocytoma cell line P-815 generated by a point mutation. , 1994, Blood.
[12] L. Ashman,et al. Identification of mutations in the coding sequence of the proto-oncogene c-kit in a human mast cell leukemia cell line causing ligand-independent activation of c-kit product. , 1993, The Journal of clinical investigation.
[13] B. Ponder,et al. Germ-line mutations of the RET proto-oncogene in multiple endocrine neoplasia type 2A , 1993, Nature.
[14] J. Ihle,et al. Association of hematopoietic cell phosphatase with c-Kit after stimulation with c-Kit ligand , 1993, Molecular and cellular biology.
[15] Y. Nishimune,et al. Mast cell number in the skin of heterozygotes reflects the molecular nature of c-kit mutation. , 1993, Blood.
[16] C. Michnoff,et al. Multiple B29 containing complexes on murine B lymphocytes. Common and stage-restricted Ig-associated polypeptide chains. , 1993, Journal of immunology.
[17] W. Fantl,et al. Signalling by receptor tyrosine kinases. , 1993, Annual review of biochemistry.
[18] M. Naito,et al. Cloning and expression of the chicken c-kit proto-oncogene. , 1993, Gene.
[19] L. Larue,et al. Spontaneous malignant transformation of melanocytes explanted from Wf/Wf mice with a Kit kinase-domain mutation. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[20] C. Fitzer-Attas,et al. Mouse platelet-derived growth factor alpha receptor: sequence, tissue-specific expression and correlation with metastatic phenotype. , 1992, Oncogene.
[21] Y. Yarden,et al. Structural organization of the murine c-kit proto-oncogene. , 1992, Oncogene.
[22] S. Lev,et al. Interkinase domain of kit contains the binding site for phosphatidylinositol 3' kinase. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[23] C. Heldin,et al. Activation of the human c‐kit product by ligand‐induced dimerization mediates circular actin reorganization and chemotaxis. , 1991, The EMBO journal.
[24] H. Kitayama,et al. Expression and functional role of the proto-oncogene c-kit in acute myeloblastic leukemia cells. , 1991, Blood.
[25] E. Morii,et al. Characterization of Ws mutant allele of rats: a 12-base deletion in tyrosine kinase domain of c-kit gene. , 1991, Blood.
[26] Alastair,et al. Signal transduction by normal isoforms and W mutant variants of the Kit receptor tyrosine kinase. , 1991, The EMBO journal.
[27] T. Pawson,et al. The Steel/W transduction pathway: kit autophosphorylation and its association with a unique subset of cytoplasmic signaling proteins is induced by the Steel factor , 1991, Molecular and cellular biology.
[28] K. Davies,et al. Molecular biology of the W and steel loci. , 1991 .
[29] H. Kitayama,et al. Proliferation of human myeloid leukemia cell line associated with the tyrosine-phosphorylation and activation of the proto-oncogene c-kit product. , 1991, Blood.
[30] P. Leder,et al. The hematopoietic growth factor KL is encoded by the SI locus and is the ligand of the c-kit receptor, the gene product of the W locus , 1990, Cell.
[31] David A. Williams,et al. Stem cell factor is encoded at the SI locus of the mouse and is the ligand for the c-kit tyrosine kinase receptor , 1990, Cell.
[32] N. Copeland,et al. Mast cell growth factor maps near the steel locus on mouse chromosome 10 and is deleted in a number of steel alleles , 1990, Cell.
[33] P. Leder,et al. The kit ligand: A cell surface molecule altered in steel mutant fibroblasts , 1990, Cell.
[34] C. March,et al. Identification of a ligand for the c-kit proto-oncogene , 1990, Cell.
[35] S. Cannistra,et al. Signal transduction of the human granulocyte-macrophage colony-stimulating factor and interleukin-3 receptors involves tyrosine phosphorylation of a common set of cytoplasmic proteins. , 1990, Blood.
[36] K. Nocka,et al. Molecular bases of dominant negative and loss of function mutations at the murine c‐kit/white spotting locus: W37, Wv, W41 and W. , 1990, The EMBO journal.
[37] Joseph Schlessinger,et al. Signal transduction by receptors with tyrosine kinase activity , 1990, Cell.
[38] A. Reith,et al. W mutant mice with mild or severe developmental defects contain distinct point mutations in the kinase domain of the c-kit receptor. , 1990, Genes & development.
[39] L. Rohrschneider,et al. Activation of the feline c-fms proto-oncogene: Multiple alterations are required to generate a fully transformed phenotype , 1988, Cell.
[40] J. Downing,et al. A point mutation in the extracellular domain of the human CSF-1 receptor (c-fms proto-oncogene product) activates its transforming potential , 1988, Cell.
[41] S. Majumder,et al. c-kit protein, a transmembrane kinase: identification in tissues and characterization , 1988, Molecular and cellular biology.
[42] D. Housman,et al. The dominant-white spotting (W) locus of the mouse encodes the c-kit proto-oncogene , 1988, Cell.
[43] V. Chapman,et al. The proto-oncogene c-kit encoding a transmembrane tyrosine kinase receptor maps to the mouse W locus , 1988, Nature.
[44] Cori Bargmann,et al. Oncogenic activation of the neu‐encoded receptor protein by point mutation and deletion. , 1988, The EMBO journal.
[45] F. Ruddle,et al. Primary structure of c‐kit: relationship with the CSF‐1/PDGF receptor kinase family–oncogenic activation of v‐kit involves deletion of extracellular domain and C terminus. , 1988, The EMBO journal.
[46] C. Stocking,et al. Autocrine stimulation after transfer of the granulocyte/macrophage colony-stimulating factor gene and autonomous growth are distinct but interdependent steps in the oncogenic pathway. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[47] A. Ullrich,et al. Human proto‐oncogene c‐kit: a new cell surface receptor tyrosine kinase for an unidentified ligand. , 1987, The EMBO journal.
[48] M. Roussel,et al. Transforming potential of the c-fms proto-oncogene (CSF-1 receptor) , 1987, Nature.
[49] L. Rohrschneider,et al. Murine c-fms cDNA: cloning, sequence analysis and retroviral expression. , 1987, Oncogene research.
[50] Cori Bargmann,et al. Multiple independent activations of the neu oncogene by a point mutation altering the transmembrane domain of p185 , 1986, Cell.
[51] John E. Murphy,et al. A new acute transforming feline retrovirus and relationship of its oncogene v-kit with the protein kinase gene family , 1986, Nature.
[52] S. Galli,et al. Fate of bone marrow-derived cultured mast cells after intracutaneous, intraperitoneal, and intravenous transfer into genetically mast cell- deficient W/Wv mice. Evidence that cultured mast cells can give rise to both connective tissue type and mucosal mast cells , 1985, The Journal of experimental medicine.
[53] Mary Anne Wheeler,et al. Stem , 1985 .
[54] S. Hosoda,et al. COMPARISON OF SEROTONIN-PRODUCING MURINE MASTOCYTOMAS, P-815 AND FMA3: DETERMINATION OF TRYPTOPHAN HYDROXYLASE, AROMATIC L-AMINO ACID DECARBOXYLASE, AND CELLULAR CONCENTRATION OF TRYPTOPHAN, 5-HYDROXYTRYPTOPHAN, 5-HYDROXYTRYPTAMINE AND 5-HYDROXYINDOLEACETIC ACID , 1984 .
[55] J. Sambrook,et al. Molecular Cloning: A Laboratory Manual , 2001 .
[56] D. Baltimore,et al. Construction of a retrovirus packaging mutant and its use to produce helper-free defective retrovirus , 1983, Cell.
[57] P Berg,et al. Transformation of mammalian cells to antibiotic resistance with a bacterial gene under control of the SV40 early region promoter. , 1982, Journal of molecular and applied genetics.
[58] Makio,et al. Clonal assay of mouse mast cell colonies in methylcellulose culture. , 1982, Blood.
[59] C. Auffray,et al. Purification of mouse immunoglobulin heavy-chain messenger RNAs from total myeloma tumor RNA. , 2005, European journal of biochemistry.
[60] M. Wigler,et al. DNA-mediated transfer of the adenine phosphoribosyltransferase locus into mammalian cells. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[61] E. Russell. Hereditary anemias of the mouse: a review for geneticists. , 1979, Advances in genetics.
[62] F. Sanger,et al. DNA sequencing with chain-terminating inhibitors. , 1977, Proceedings of the National Academy of Sciences of the United States of America.
[63] F. Graham,et al. Characteristics of a human cell line transformed by DNA from human adenovirus type 5. , 1977, The Journal of general virology.
[64] J. Furth,et al. Transplantable Mastocytoma in the Mouse Containing Histamine, Heparin, 5-Hydroxytryptamine.∗ , 1957, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.