HMGB1: A critical mediator for oxidized-low density lipoproteins induced atherosclerosis.
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Xinqun Hu | Zhaowei Zhu | C. Shu | Qin Zhou
[1] F. Romeo,et al. Oxidized LDL attenuates protective autophagy and induces apoptotic cell death of endothelial cells: Role of oxidative stress and LOX-1 receptor expression. , 2015, International journal of cardiology.
[2] Jingjing Cai,et al. Hypoxic Trophoblast HMGB1 Induces Endothelial Cell Hyperpermeability via the TRL-4/Caveolin-1 Pathway , 2014, The Journal of Immunology.
[3] F. van Petegem,et al. Deciphering the Binding of Caveolin-1 to Client Protein Endothelial Nitric-oxide Synthase (eNOS) , 2014, The Journal of Biological Chemistry.
[4] Xiaofeng Yu,et al. IGF-1 alleviates ox-LDL-induced inflammation via reducing HMGB1 release in HAECs. , 2012, Acta biochimica et biophysica Sinica.
[5] F. Antohe,et al. Hyperlipidemia stimulates the extracellular release of the nuclear high mobility group box 1 protein , 2011, Cell and Tissue Research.
[6] K. Peter,et al. High-Mobility Group Box Protein 1 Neutralization Reduces Development of Diet-Induced Atherosclerosis in Apolipoprotein E–Deficient Mice , 2011, Arteriosclerosis, thrombosis, and vascular biology.
[7] David S. Park,et al. Caveolin, Caveolae, and Endothelial Cell Function , 2003, Arteriosclerosis, thrombosis, and vascular biology.