Modulation of Tumor Necrosis Factor-mediated Cell Death by Fullerenes

[1]  C. Hetz.  Apoptosis, necrosis and autophagy: from mechanisms to biomedical applications. , 2008, Current molecular medicine.

[2]  Zoran Markovic,et al.  The mechanism of cell-damaging reactive oxygen generation by colloidal fullerenes. , 2007, Biomaterials.

[3]  S. Mocellin,et al.  Towards the development of tumor necrosis factor (TNF) sensitizers: making TNF work against cancer. , 2007, Current pharmaceutical design.

[4]  S. Linder,et al.  Acute apoptosis by cisplatin requires induction of reactive oxygen species but is not associated with damage to nuclear DNA , 2007, International journal of cancer.

[5]  Miroslav D. Dramićanin,et al.  Inactivation of nanocrystalline C60 cytotoxicity by γ-irradiation , 2006 .

[6]  Zoran Markovic,et al.  Distinct cytotoxic mechanisms of pristine versus hydroxylated fullerene. , 2006, Toxicological sciences : an official journal of the Society of Toxicology.

[7]  Eva Oberdörster,et al.  Ecotoxicology of carbon-based engineered nanoparticles: Effects of fullerene (C60) on aquatic organisms , 2006 .

[8]  S. Sollott,et al.  Mitochondrial ROS-induced ROS release: an update and review. , 2006, Biochimica et biophysica acta.

[9]  V. Trajković,et al.  Acidosis affects tumor cell survival through modulation of nitric oxide release. , 2006, Free radical biology & medicine.

[10]  Nebojsa Romcevic,et al.  Inactivation of nanocrystalline C60 cytotoxicity by gamma-irradiation. , 2006, Biomaterials.

[11]  J. West,et al.  Nano-C60 cytotoxicity is due to lipid peroxidation. , 2005, Biomaterials.

[12]  Stephen R. Wilson,et al.  [60]fullerene is a powerful antioxidant in vivo with no acute or subacute toxicity. , 2005, Nano letters.

[13]  S. Alavian,et al.  Apoptosis in liver diseases--detection and therapeutic applications. , 2005, Medical science monitor : international medical journal of experimental and clinical research.

[14]  A. Wendel,et al.  Tumor necrosis factor: how to make a killer molecule tumor-specific? , 2005, Current cancer drug targets.

[15]  G. Andrievsky,et al.  Is the C60 Fullerene Molecule Toxic?! , 2005 .

[16]  K. Ausman,et al.  C60 in water: nanocrystal formation and microbial response. , 2005, Environmental science & technology.

[17]  C. Thompson,et al.  Death by design: apoptosis, necrosis and autophagy. , 2004, Current opinion in cell biology.

[18]  Xian‐Wen Wei,et al.  Determination of proteins with fullerol by a resonance light scattering technique. , 2004, Analytical biochemistry.

[19]  B. Liu,et al.  A Novel Human Phosphatidylethanolamine-binding Protein Resists Tumor Necrosis Factor α-induced Apoptosis by Inhibiting Mitogen-activated Protein Kinase Pathway Activation and Phosphatidylethanolamine Externalization* , 2004, Journal of Biological Chemistry.

[20]  J. West,et al.  The Differential Cytotoxicity of Water-Soluble Fullerenes , 2004 .

[21]  J. Au,et al.  Apoptosis: A New Pharmacodynamic Endpoint , 1997, Pharmaceutical Research.

[22]  H. Shepard,et al.  Resistance of tumor cells to tumor necrosis factor , 1988, Journal of Clinical Immunology.

[23]  J. Remacle,et al.  Caspase activation precedes PTP opening in TNF-alpha-induced apoptosis in L929 cells. , 2004, Mitochondrion.

[24]  M. Prato,et al.  Fullerene derivatives: an attractive tool for biological applications. , 2003, European journal of medicinal chemistry.

[25]  Z. Marković,et al.  Temperature measurement of carbon arc plasma in helium , 2003 .

[26]  Z. Xiaodong,et al.  Preparation of (99m)Tc-C(60)(OH)(x) and its biodistribution studies. , 2002, Nuclear medicine and biology.

[27]  D. Ferrari,et al.  Activation and caspase-mediated inhibition of PARP: a molecular switch between fibroblast necrosis and apoptosis in death receptor signaling. , 2002, Molecular biology of the cell.

[28]  S. Kong,et al.  Tumour Necrosis Factor Induced an Early Release of Superoxide and a Late Mitochondrial Membrane Depolarization in L929 Cells , 2001, Neurosignals.

[29]  K. O’Malley,et al.  Fullerene-based antioxidants and neurodegenerative disorders. , 2001, Parkinsonism & related disorders.

[30]  P. Vandenabeele,et al.  Apoptotic and necrotic cell death induced by death domain receptors , 2001, Cellular and Molecular Life Sciences CMLS.

[31]  J. Wu,et al.  Polyhydroxylated C60, fullerenols, as glutamate receptor antagonists and neuroprotective agents , 2000, Journal of neuroscience research.

[32]  C. Franceschi,et al.  C60 carboxyfullerene exerts a protective activity against oxidative stress-induced apoptosis in human peripheral blood mononuclear cells. , 2000, Biochemical and biophysical research communications.

[33]  C. Franceschi,et al.  Carboxyfullerenes protect human keratinocytes from ultraviolet-B-induced apoptosis. , 2000, The Journal of investigative dermatology.

[34]  H. Inagawa,et al.  A new method of antitumor therapy with a high dose of TNF perfusion for unresectable liver tumors. , 2000, Anticancer research.

[35]  T. Linke,et al.  Overexpression of Acid Ceramidase Protects from Tumor Necrosis Factor–Induced Cell Death , 2000, The Journal of experimental medicine.

[36]  M. Luster,et al.  The Role of Tumor Necrosis Factor α in Chemical‐Induced Hepatotoxicity , 2000, Annals of the New York Academy of Sciences.

[37]  D. Katschinski,et al.  Pivotal Role of Reactive Oxygen Species as Intracellular Mediators of Hyperthermia-induced Apoptosis* , 2000, The Journal of Biological Chemistry.

[38]  S. Okada,et al.  Laser flash photolysis study on photophysical and photochemical properties of C60 fine particles , 2000 .

[39]  M. Wilson,et al.  Modes of L929 cell death induced by TNF-alpha and other cytotoxic agents. , 1999, Cytokine.

[40]  R. Paus,et al.  Death deflected: IL‐15 inhibits TNF‐α‐mediated apoptosis in fibroblasts by TRAF2 recruitment to the IL‐15Rα chain , 1999, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[41]  E. Androphy,et al.  Human Papillomavirus Type 16 E6-enhanced Susceptibility of L929 Cells to Tumor Necrosis Factor α Correlates with Increased Accumulation of Reactive Oxygen Species* , 1999, The Journal of Biological Chemistry.

[42]  C. Franceschi,et al.  C3‐Fullero‐tris‐methanodicarboxylic acid protects epithelial cells from radiation‐induced anoikia by influencing cell adhesion ability , 1999, FEBS letters.

[43]  K. O’Malley,et al.  Distinct Mechanisms Underlie Neurotoxin-Mediated Cell Death in Cultured Dopaminergic Neurons , 1999, The Journal of Neuroscience.

[44]  E. Androphy,et al.  Human papillomavirus type 16 E6-enhanced susceptibility of L929 cells to tumor necrosis factor alpha correlates with increased accumulation of reactive oxygen species. , 1999, The Journal of biological chemistry.

[45]  J. Lemasters.  V. Necrapoptosis and the mitochondrial permeability transition: shared pathways to necrosis and apoptosis. , 1999, The American journal of physiology.

[46]  W. Fiers,et al.  Redox regulation of TNF signaling , 1999, BioFactors.

[47]  W. Fiers,et al.  Inhibition of Caspases Increases the Sensitivity of L929 Cells to Necrosis Mediated by Tumor Necrosis Factor , 1998, The Journal of experimental medicine.

[48]  Shen,et al.  Carboxyfullerenes as neuroprotective agents. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[49]  D. McConkey,et al.  Ras signaling in tumor necrosis factor‐induced apoptosis. , 1996, The EMBO journal.

[50]  D. Choi,et al.  Buckminsterfullerenol Free Radical Scavengers Reduce Excitotoxic and Apoptotic Death of Cultured Cortical Neurons , 1996, Neurobiology of Disease.

[51]  W. Fiers,et al.  Two tumour necrosis factor receptors: structure and function. , 1995, Trends in cell biology.

[52]  A. Lichtenstein,et al.  Atypical apoptotic cell death induced in L929 targets by exposure to tumor necrosis factor. , 1995, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.

[53]  M. Leist,et al.  Murine hepatocyte apoptosis induced in vitro and in vivo by TNF-alpha requires transcriptional arrest. , 1994, Journal of immunology.

[54]  W. Fiers,et al.  Molecular mechanisms of tumor necrosis factor‐induced cytotoxicity , 1994, FEBS letters.

[55]  T. Hennet,et al.  Tumour necrosis factor-α induces superoxide anion generation in mitochondria of L929 cells , 1993 .

[56]  P. Vassalli,et al.  The pathophysiology of tumor necrosis factors. , 1992, Annual review of immunology.

[57]  D. Kufe,et al.  Recombinant human tumor necrosis factor administered as a 24-hour intravenous infusion. A phase I and pharmacologic study. , 1988, Journal of the National Cancer Institute.

[58]  D. A. Flick,et al.  Comparison of in vitro cell cytotoxic assays for tumor necrosis factor. , 1984, Journal of immunological methods.

[59]  T. Chou,et al.  Quantitative analysis of dose-effect relationships: the combined effects of multiple drugs or enzyme inhibitors. , 1984, Advances in enzyme regulation.

[60]  H. Kowarzyk.  Structure and Function. , 1910, Nature.