Transforming growth factor β activates Rac1 and Cdc42Hs GTPases and the JNK pathway in skeletal muscle cells
暂无分享,去创建一个
C. Gauthier-Rouvière | S. Charrasse | F. Comunale | Mayya Meriane | Sophie Charrasse | Franck Comunale | Cécile Gauthier‐Rouvière | M. Meriane | Mayya Meriane
[1] H. Heller,et al. Rac1 Inhibits Myogenic Differentiation by Preventing the Complete Withdrawal of Myoblasts from the Cell Cycle* , 2001, The Journal of Biological Chemistry.
[2] F. McCormick,et al. An essential role for Rac in Ras transformation , 1995, Nature.
[3] P. Roux,et al. The small GTPases Cdc42Hs, Rac1 and RhoG delineate Raf-independent pathways that cooperate to transform NIH3T3 cells , 1997, Current Biology.
[4] R. Davis,et al. Evidence for a Role of Rho-like GTPases and Stress-activated Protein Kinase/c-Jun N-terminal Kinase (SAPK/JNK) in Transforming Growth Factor β-mediated Signaling* , 1997, The Journal of Biological Chemistry.
[5] Y. Hiroi,et al. The Rho Family G Proteins Play a Critical Role in Muscle Differentiation , 1998, Molecular and Cellular Biology.
[6] R. Schwartz,et al. RhoA Signaling via Serum Response Factor Plays an Obligatory Role in Myogenic Differentiation* , 1998, The Journal of Biological Chemistry.
[7] A. Lassar,et al. Regulatory mechanisms that coordinate skeletal muscle differentiation and cell cycle withdrawal. , 1994, Current opinion in cell biology.
[8] K. Skorecki,et al. Extracellular signal-regulated kinase and the small GTP-binding protein, Rac, contribute to the effects of transforming growth factor-beta1 on gene expression. , 1996, The Journal of biological chemistry.
[9] S. Ferrari,et al. Inhibition of myogenesis by transforming growth factor beta is density-dependent and related to the translocation of transcription factor MEF2 to the cytoplasm. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[10] R. Derynck,et al. Regulation of Smad signalling by protein associations and signalling crosstalk. , 1999, Trends in cell biology.
[11] S. Schmid,et al. Regulation of receptor-mediated endocytosis by Rho and Rac , 1996, Nature.
[12] M. Primig,et al. Critical activities of Rac1 and Cdc42Hs in skeletal myogenesis: antagonistic effects of JNK and p38 pathways. , 2000, Molecular biology of the cell.
[13] A. Ashworth,et al. An essential role for Rho, Rac, and Cdc42 GTPases in cell cycle progression through G1 , 1995, Science.
[14] M. Primig,et al. RhoA GTPase and serum response factor control selectively the expression of MyoD without affecting Myf5 in mouse myoblasts. , 1998, Molecular biology of the cell.
[15] F. McCormick,et al. A role for Rho in Ras transformation. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[16] Naoya Yamamoto,et al. Smad1 and smad5 act downstream of intracellular signalings of BMP-2 that inhibits myogenic differentiation and induces osteoblast differentiation in C2C12 myoblasts. , 1997, Biochemical and biophysical research communications.
[17] B. Black,et al. Transcriptional control of muscle development by myocyte enhancer factor-2 (MEF2) proteins. , 1998, Annual review of cell and developmental biology.
[18] N. Tapon,et al. Rho, Rac and Cdc42 GTPases regulate the organization of the actin cytoskeleton. , 1997, Current opinion in cell biology.
[19] A. Blangy,et al. Biogenesis of N-cadherin-dependent cell-cell contacts in living fibroblasts is a microtubule-dependent kinesin-driven mechanism. , 2002, Molecular biology of the cell.
[20] A. Hall,et al. Rho GTPases and the actin cytoskeleton. , 1998, Science.
[21] C. Der,et al. Activation of Rac1, RhoA, and mitogen-activated protein kinases is required for Ras transformation , 1995, Molecular and cellular biology.
[22] G. Falcone,et al. Distinct effects of Rac1 on differentiation of primary avian myoblasts. , 1999, Molecular biology of the cell.
[23] M. Aepfelbacher,et al. Spreading of differentiating human monocytes is associated with a major increase in membrane-bound CDC42. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[24] T. Jessell,et al. A role for rhoB in the delamination of neural crest cells from the dorsal neural tube. , 1998, Development.
[25] C. Tournigand,et al. Differential roles of JNK and Smad2 signaling pathways in the inhibition of c-Myc-induced cell death by TGF-β , 2000, Oncogene.
[26] J. D. Brown,et al. MEKK-1, a Component of the Stress (Stress-activated Protein Kinase/c-Jun N-terminal Kinase) Pathway, Can Selectively Activate Smad2-mediated Transcriptional Activation in Endothelial Cells* , 1999, The Journal of Biological Chemistry.
[27] S. Hauschka,et al. Synergistic interactions between bFGF and a TGF-beta family member may mediate myogenic signals from the neural tube. , 1997, Development.
[28] H. Arnold,et al. Muscle differentiation: more complexity to the network of myogenic regulators. , 1998, Current opinion in genetics & development.
[29] John G. Collard,et al. Rac Downregulates Rho Activity: Reciprocal Balance between Both Gtpases Determines Cellular Morphology and Migratory Behavior , 1999 .
[30] C. Heldin,et al. Transforming growth factor-beta-induced mobilization of actin cytoskeleton requires signaling by small GTPases Cdc42 and RhoA. , 2002, Molecular biology of the cell.
[31] Victor K. Lin,et al. Myogenin, a factor regulating myogenesis, has a domain homologous to MyoD , 1989, Cell.
[32] A. Blangy,et al. N-cadherin–dependent cell–cell contact regulates Rho GTPases and β-catenin localization in mouse C2C12 myoblasts , 2002, The Journal of cell biology.
[33] A. McMahon,et al. Combinatorial signaling by Sonic hedgehog and Wnt family members induces myogenic bHLH gene expression in the somite. , 1995, Genes & development.
[34] G. Spizz,et al. Regulation of myogenic differentiation by type beta transforming growth factor , 1986, The Journal of cell biology.
[35] P. Auvinen,et al. Endosome dynamics regulated by a Rho protein , 1996, Nature.
[36] P. Cohen,et al. Stress-activated Protein Kinase-2/p38 and a Rapamycin-sensitive Pathway Are Required for C2C12 Myogenesis* , 1999, The Journal of Biological Chemistry.
[37] J. Massagué. TGF-beta signal transduction. , 1998, Annual review of biochemistry.
[38] J. Davoust,et al. Cytokinesis arrest and redistribution of actin-cytoskeleton regulatory components in cells expressing the Rho GTPase CDC42Hs. , 1996, Journal of cell science.
[39] C. Nobes,et al. Rho, Rac, and Cdc42 GTPases regulate the assembly of multimolecular focal complexes associated with actin stress fibers, lamellipodia, and filopodia , 1995, Cell.
[40] P. Roux,et al. RhoG GTPase controls a pathway that independently activates Rac1 and Cdc42Hs. , 1998, Molecular biology of the cell.
[41] H. Moses,et al. Transforming growth factor-beta1 mediates epithelial to mesenchymal transdifferentiation through a RhoA-dependent mechanism. , 2001, Molecular biology of the cell.
[42] A. Hall,et al. The Small GTPases Rho and Rac Are Required for the Establishment of Cadherin-dependent Cell–Cell Contacts , 1997, The Journal of cell biology.
[43] G. Cossu,et al. How is myogenesis initiated in the embryo? , 1996, Trends in genetics : TIG.
[44] K. Skorecki,et al. Extracellular Signal-regulated Kinase and the Small GTP-binding Protein, Rac, Contribute to the Effects of Transforming Growth Factor-β1 on Gene Expression* , 1996, The Journal of Biological Chemistry.
[45] O. Pourquié,et al. Lateral and Axial Signals Involved in Avian Somite Patterning: A Role for BMP4 , 1996, Cell.
[46] L. Lim,et al. Rho family GTPases and neuronal growth cone remodelling: relationship between increased complexity induced by Cdc42Hs, Rac1, and acetylcholine and collapse induced by RhoA and lysophosphatidic acid , 1997, Molecular and cellular biology.
[47] C. Nobes,et al. Rho, rac and cdc42 GTPases: regulators of actin structures, cell adhesion and motility. , 1995, Biochemical Society transactions.
[48] E. Olson,et al. Defining the regulatory networks for muscle development. , 1996, Current opinion in genetics & development.
[49] J. Massagué,et al. Type beta transforming growth factor is an inhibitor of myogenic differentiation. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[50] P. Roux,et al. Participation of small GTPases Rac1 and Cdc42Hs in myoblast transformation , 2002, Oncogene.