Beyond BOTOX: advantages and limitations of individual botulinum neurotoxins
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[1] E. Callaway. A molecular and genetic arsenal for systems neuroscience , 2005, Trends in Neurosciences.
[2] L. Maffei,et al. Antiepileptic Effects of Botulinum Neurotoxin E , 2005, The Journal of Neuroscience.
[3] D. Dinsdale,et al. Botulinum neurotoxin C initiates two different programs for neurite degeneration and neuronal apoptosis , 2005, The Journal of cell biology.
[4] T. Bliss,et al. Long-term potentiation and cognitive drug discovery. , 2005, Current opinion in investigational drugs.
[5] Benjamin L Walter,et al. Surgical treatment for Parkinson's disease , 2004, The Lancet Neurology.
[6] G. Ahnert-Hilger,et al. Botulinum neurotoxin type D enables cytosolic delivery of enzymatically active cargo proteins to neurones via unfolded translocation intermediates , 2004, Journal of neurochemistry.
[7] H. Lüders,et al. Frontal lobe epilepsy. , 2004, Epileptic disorders : international epilepsy journal with videotape.
[8] B. Davletov,et al. A molecular basis underlying differences in the toxicity of botulinum serotypes A and E , 2004, EMBO reports.
[9] H. Zeringue,et al. Post-transcriptional gene silencing in neurons , 2004, Current Opinion in Neurobiology.
[10] J. Sobel,et al. Foodborne Botulism in the United States, 1990–2000 , 2004, Emerging infectious diseases.
[11] H. Bigalke,et al. Synaptotagmins I and II Act as Nerve Cell Receptors for Botulinum Neurotoxin G* , 2004, Journal of Biological Chemistry.
[12] T. Südhof. The synaptic vesicle cycle , 2004 .
[13] J. Jankovic,et al. Botulinum toxin in clinical practice , 2004, Journal of Neurology, Neurosurgery & Psychiatry.
[14] M. Cherington. Botulism: update and review. , 2004, Seminars in neurology.
[15] C. Montecucco,et al. Central injection of botulinum neurotoxins: behavioural effects in mice , 2004, Behavioural pharmacology.
[16] A. B. Scott. Development of botulinum toxin therapy. , 2004, Dermatologic clinics.
[17] Helen Ho,et al. Plasma membrane localization signals in the light chain of botulinum neurotoxin. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[18] Roberto Eleopra,et al. Different types of botulinum toxin in humans , 2004, Movement disorders : official journal of the Movement Disorder Society.
[19] Frank J Erbguth,et al. Historical notes on botulism, Clostridium botulinum, botulinum toxin, and the idea of the therapeutic use of the toxin , 2004, Movement disorders : official journal of the Movement Disorder Society.
[20] C. Frassoni,et al. SNAP-25 Modulation of Calcium Dynamics Underlies Differences in GABAergic and Glutamatergic Responsiveness to Depolarization , 2004, Neuron.
[21] L. Simpson,et al. Identification of the major steps in botulinum toxin action. , 2004, Annual review of pharmacology and toxicology.
[22] Colin Rickman,et al. High Affinity Interaction of Syntaxin and SNAP-25 on the Plasma Membrane Is Abolished by Botulinum Toxin E* , 2004, Journal of Biological Chemistry.
[23] A. Dickenson,et al. Retargeted clostridial endopeptidases: Inhibition of nociceptive neurotransmitter release in vitro, and antinociceptive activity in in vivo models of pain , 2002, Movement disorders : official journal of the Movement Disorder Society.
[24] H. Bellen,et al. Synaptotagmin I, a Ca2+ sensor for neurotransmitter release , 2003, Trends in Neurosciences.
[25] O. Dolly. Synaptic Transmission: Inhibition of Neurotransmitter Release by Botulinum Toxins , 2003, Headache.
[26] B. Davletov,et al. Getting muscles moving again after botulinum toxin: novel therapeutic challenges. , 2003, Trends in molecular medicine.
[27] J. Dolly,et al. Dynamics of motor nerve terminal remodeling unveiled using SNARE-cleaving botulinum toxins: the extent and duration are dictated by the sites of SNAP-25 truncation , 2003, Molecular and Cellular Neuroscience.
[28] Leonard A. Smith,et al. Evaluation of the Therapeutic Usefulness of Botulinum Neurotoxin B, C1, E, and F Compared with the Long Lasting Type A , 2003, The Journal of Biological Chemistry.
[29] M. Montal,et al. Translocation of botulinum neurotoxin light chain protease through the heavy chain channel , 2003, Nature Structural Biology.
[30] A. Dickenson,et al. Inhibition of Release of Neurotransmitters from Rat Dorsal Root Ganglia by a Novel Conjugate of a Clostridium botulinumToxin A Endopeptidase Fragment and Erythrina cristagalliLectin* , 2002, The Journal of Biological Chemistry.
[31] C. Schengrund,et al. Botulinum Neurotoxin A Activity Is Dependent upon the Presence of Specific Gangliosides in Neuroblastoma Cells Expressing Synaptotagmin I* , 2002, The Journal of Biological Chemistry.
[32] C. Lewis. Botox cosmetic: a look at looking good. , 2002, FDA consumer.
[33] Eric A. Johnson,et al. Development of a delivery vehicle for intracellular transport of botulinum neurotoxin antagonists 1 , 2002, FEBS letters.
[34] H. Keshishian,et al. Molecular genetic approaches to the targeted suppression of neuronal activity , 2001, Current Biology.
[35] K. Aoki,et al. Botulinum toxin type A and other botulinum toxin serotypes: a comparative review of biochemical and pharmacological actions , 2001, European journal of neurology.
[36] E. Neale,et al. The Role of the Synaptic Protein SNAP-25 in the Potency of Botulinum Neurotoxin Type A* , 2001, The Journal of Biological Chemistry.
[37] M. Adler,et al. Persistence of botulinum neurotoxin A demonstrated by sequential administration of serotypes A and E in rat EDL muscle. , 2001, Toxicon : official journal of the International Society on Toxinology.
[38] E. Matveeva,et al. N-ethylmaleimide sensitive factor (NSF) structure and function. , 2001, International review of cytology.
[39] J. Cabaniols,et al. Targeting of SNAP-25 to Membranes Is Mediated by Its Association with the Target SNARE Syntaxin* , 2000, The Journal of Biological Chemistry.
[40] G. Schiavo,et al. Neurotoxins affecting neuroexocytosis. , 2000, Physiological reviews.
[41] M. Veit. Palmitoylation of the 25-kDa synaptosomal protein (SNAP-25) in vitro occurs in the absence of an enzyme, but is stimulated by binding to syntaxin. , 1999, The Biochemical journal.
[42] S. J. Martin,et al. Synaptic plasticity and memory: an evaluation of the hypothesis. , 2000, Annual review of neuroscience.
[43] G. Oyler,et al. Persistence of botulinum neurotoxin action in cultured spinal cord cells 1,2 , 1999, FEBS letters.
[44] E. Johnson,et al. Clostridial toxins as therapeutic agents: benefits of nature's most toxic proteins. , 1999, Annual review of microbiology.
[45] Reinhard Jahn,et al. Crystal structure of a SNARE complex involved in synaptic exocytosis at 2.4 Å resolution , 1998, Nature.
[46] A. B. Maksymowych,et al. Binding and Transcytosis of Botulinum Neurotoxin by Polarized Human Colon Carcinoma Cells* , 1998, The Journal of Biological Chemistry.
[47] L. Williamson,et al. Syntaxin and 25‐kDa synaptosomal‐associated protein: Differential effects of botulinum neurotoxins C1 and A on neuronal survival , 1998, Journal of neuroscience research.
[48] B. Gähwiler,et al. Ca2+ or Sr2+ Partially Rescues Synaptic Transmission in Hippocampal Cultures Treated with Botulinum Toxin A and C, But Not Tetanus Toxin , 1997, The Journal of Neuroscience.
[49] L. Gomella,et al. In vitro characterization of botulinum toxin types A, C and D action on human tissues: combined electrophysiologic, pharmacologic and molecular biologic approaches. , 1997, The Journal of pharmacology and experimental therapeutics.
[50] A. Brünger,et al. A Structural Change Occurs upon Binding of Syntaxin to SNAP-25* , 1997, The Journal of Biological Chemistry.
[51] J. Brown,et al. Clostridial Neurotoxins and Substrate Proteolysis in Intact Neurons , 1996, The Journal of Biological Chemistry.
[52] J. Dolly,et al. Botulinum neurotoxin C1 cleaves both syntaxin and SNAP-25 in intact and permeabilized chromaffin cells: correlation with its blockade of catecholamine release. , 1996, Biochemistry.
[53] Kazuki Sato,et al. The high‐affinity binding of Clostridium botulinum type B neurotoxin to synaptotagmin II associated with gangliosides GT1b/GD1a , 1996, FEBS letters.
[54] R. Jahn,et al. Clostridial neurotoxins: new tools for dissecting exocytosis. , 1994, Trends in cell biology.
[55] H. Wellhöner. Tetanus and botulinum neurotoxins , 1994 .
[56] F. Benfenati,et al. Botulinum neurotoxins serotypes A and E cleave SNAP‐25 at distinct COOH‐terminal peptide bonds , 1993, FEBS letters.
[57] Thomas C. Südhof,et al. Botulinum neurotoxin A selectively cleaves the synaptic protein SNAP-25 , 1993, Nature.
[58] Paul Tempst,et al. SNAP receptors implicated in vesicle targeting and fusion , 1993, Nature.
[59] R. Scheller,et al. Syntaxin: a synaptic protein implicated in docking of synaptic vesicles at presynaptic active zones. , 1992, Science.
[60] J. Polli,et al. Developmental expression of the 25-kDa synaptosomal-associated protein (SNAP-25) in rat brain. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[61] H. Kurazono,et al. The complete sequence of botulinum neurotoxin type A and comparison with other clostridial neurotoxins. , 1990, The Journal of biological chemistry.
[62] L. Tauc,et al. Inhibition of neurotransmitter release by botulinum neurotoxins and tetanus toxin at Aplysia synapses: role of the constituent chains. , 1990, Journal de physiologie.
[63] A. de Paiva,et al. Light chain of botulinum neurotoxin is active in mammalian motor nerve terminals when delivered via liposomes , 1990, FEBS letters.
[64] F E Bloom,et al. The identification of a novel synaptosomal-associated protein, SNAP-25, differentially expressed by neuronal subpopulations , 1989, The Journal of cell biology.
[65] T. Südhof,et al. A synaptic vesicle membrane protein is conserved from mammals to Drosophila , 1989, Neuron.
[66] J. Dolly,et al. Characterization of the Inhibitory Action of Botulinum Neurotoxin Type A on the Release of Several Transmitters from Rat Cerebrocortical Synaptosomes , 1988, Journal of neurochemistry.
[67] R. Scheller,et al. VAMP-1: a synaptic vesicle-associated integral membrane protein. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[68] J. Black,et al. Interaction of 125I-labeled botulinum neurotoxins with nerve terminals. I. Ultrastructural autoradiographic localization and quantitation of distinct membrane acceptors for types A and B on motor nerves , 1986, The Journal of cell biology.
[69] G. Bergey,et al. Botulinum a neurotoxin inhibits non-cholinergic synaptic transmission in mouse spinal cord neurons in culture , 1985, Brain Research.
[70] R. Dirnhofer,et al. Isolation of Clostridium botulinum type G and identification of type G botulinal toxin in humans: report of five sudden unexpected deaths. , 1981, The Journal of infectious diseases.