Peripheral Demyelination and Neuropathic Pain Behavior in Periaxin-Deficient Mice

[1]  C. Woolf,et al.  Transcriptional and posttranslational plasticity and the generation of inflammatory pain. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[2]  T D Bird,et al.  Neuropathic pain in Charcot-Marie-Tooth disease. , 1998, Archives of physical medicine and rehabilitation.

[3]  B. Ghetti,et al.  Neurological Illness in Transgenic Mice Expressing a Prion Protein with an Insertional Mutation , 1998, Neuron.

[4]  R. Abagyan,et al.  Novel Anchorage of GluR2/3 to the Postsynaptic Density by the AMPA Receptor–Binding Protein ABP , 1998, Neuron.

[5]  Virginia M. Y. Lee,et al.  Myelin-Associated Glycoprotein Is a Myelin Signal that Modulates the Caliber of Myelinated Axons , 1998, The Journal of Neuroscience.

[6]  D. Sherman,et al.  Two PDZ Domain Proteins Encoded by the Murine Periaxin Gene Are the Result of Alternative Intron Retention and Are Differentially Targeted in Schwann Cells* , 1998, The Journal of Biological Chemistry.

[7]  M. Koltzenburg.  Painful neuropathies. , 1998, Current opinion in neurology.

[8]  D. Riethmacher,et al.  Severe neuropathies in mice with targeted mutations in the ErbB3 receptor , 1997, Nature.

[9]  M. Sheng,et al.  Ion channel targeting in neurons , 1997, BioEssays : news and reviews in molecular, cellular and developmental biology.

[10]  R. Martini.  Animal models for inherited peripheral neuropathies , 1997, Journal of anatomy.

[11]  C. Stucky,et al.  Receptive properties of mouse sensory neurons innervating hairy skin. , 1997, Journal of neurophysiology.

[12]  D. Lynch,et al.  INHERITED PERIPHERAL NEUROPATHIES , 1997 .

[13]  Emiko Suzuki,et al.  A multivalent PDZ-domain protein assembles signalling complexes in a G-protein-coupled cascade , 1997, Nature.

[14]  K. Willecke,et al.  Structural Abnormalities and Deficient Maintenance of Peripheral Nerve Myelin in Mice Lacking the Gap Junction Protein Connexin 32 , 1997, The Journal of Neuroscience.

[15]  P. Seeburg,et al.  Interaction of ion channels and receptors with PDZ domain proteins , 1997, Current Opinion in Neurobiology.

[16]  M. Sheng,et al.  Disulfide-Linked Head-to-Head Multimerization in the Mechanism of Ion Channel Clustering by PSD-95 , 1997, Neuron.

[17]  Richard L. Huganir,et al.  GRIP: a synaptic PDZ domain-containing protein that interacts with AMPA receptors , 1997, Nature.

[18]  A. Malmberg,et al.  Efficacy of spinal NMDA receptor antagonism in formalin hyperalgesia and nerve injury evoked allodynia in the rat. , 1997, The Journal of pharmacology and experimental therapeutics.

[19]  M. Bunge,et al.  Actin Plays a Role in Both Changes in Cell Shape and Gene- Expression Associated with Schwann Cell Myelination , 1997, The Journal of Neuroscience.

[20]  D. Borsook.  Molecular neurobiology of pain , 1997 .

[21]  N. Tapon,et al.  Rho, Rac and Cdc42 GTPases regulate the organization of the actin cytoskeleton. , 1997, Current opinion in cell biology.

[22]  J. Levine,et al.  C-fiber mechanical stimulus–response functions are different in inflammatory versus neuropathic hyperalgesia in the rat , 1996, Neuroscience.

[23]  Eli Eliav,et al.  Abnormal discharge originates at the site of nerve injury in experimental constriction neuropathy (CCI) in the rat , 1996, Pain.

[24]  R. Mirsky,et al.  Schwann cell development, differentiation and myelination , 1996, Current Opinion in Neurobiology.

[25]  K. Jessen.  Glial cell development , 1996 .

[26]  D. Sherman,et al.  Periaxin expression in myelinating Schwann cells: modulation by axon-glial interactions and polarized localization during development. , 1995, Development.

[27]  A. Aguzzi,et al.  Hypermyelination and demyelinating peripheral neuropathy in Pmp22-deficient mice , 1995, Nature Genetics.

[28]  M. Schachner,et al.  Protein zero (P0)–deficient mice show myelin degeneration in peripheral nerves characteristic of inherited human neuropathies , 1995, Nature Genetics.

[29]  P. Seeburg,et al.  Domain interaction between NMDA receptor subunits and the postsynaptic density protein PSD-95. , 1995, Science.

[30]  U. Suter,et al.  Biology and genetics of hereditary motor and sensory neuropathies. , 1995, Annual review of neuroscience.

[31]  B. Pentland,et al.  Pain in the Guillain-Barré syndrome: a clinical review , 1994, Pain.

[32]  M. Koltzenburg,et al.  Nociceptor modulated central sensitization causes mechanical hyperalgesia in acute chemogenic and chronic neuropathic pain. , 1994, Brain : a journal of neurology.

[33]  J. Nichols,et al.  Dicistronic targeting constructs: reporters and modifiers of mammalian gene expression. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[34]  Peter J. Brophy,et al.  Periaxin, a novel protein of myelinating schwann cells with a possible role in axonal ensheathment , 1994, Neuron.

[35]  Richard P. Bunge,et al.  Expanding roles for the Schwann cell: ensheathment, myelination, trophism and regeneration , 1993, Current Opinion in Neurobiology.

[36]  D. Price,et al.  Intrathecal treatment with dextrorphan or ketamine potently reduces pain-related behaviors in a rat model of peripheral mononeuropathy , 1993, Brain Research.

[37]  M. Schachner,et al.  Mouse P 0 gene disruption leads to hypomyelination, abnormal expression of recognition molecules, and degeneration of myelin and axons , 1992, Cell.

[38]  H Bostock,et al.  Ectopic activity in demyelinated spinal root axons of the rat. , 1992, The Journal of physiology.

[39]  J. Vance,et al.  Hereditary motor and sensory neuropathies. , 1991, Journal of medical genetics.

[40]  R. Frederickson,et al.  5' flanking and first intron sequences of the human beta-actin gene required for efficient promoter activity. , 1989, Nucleic acids research.

[41]  P. Wall,et al.  Textbook of pain , 1989 .

[42]  M. Chao,et al.  Axons regulate Schwann cell expression of the major myelin and NGF receptor genes. , 1988, Development.

[43]  R. Dubner,et al.  A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia , 1987, Pain.

[44]  A. Dickenson,et al.  Evidence for a role of the NMDA receptor in the frequency dependent potentiation of deep rat dorsal horn nociceptive neurones following c fibre stimulation. , 1987, Neuropharmacology.

[45]  J. Lehmann,et al.  CPP, a selective N-methyl-D-aspartate (NMDA)-type receptor antagonist: characterization in vitro and in vivo. , 1987, The Journal of pharmacology and experimental therapeutics.

[46]  A. Windebank,et al.  Differentiation-specific regulation of Schwann cell expression of the major myelin glycoprotein. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[47]  B. Shahani,et al.  Pain in Guillain-Barré syndrome. , 1984, Archives of neurology.

[48]  J. Martial,et al.  A method for isolation of intact, translationally active ribonucleic acid. , 1983, DNA.

[49]  William H. Calvin,et al.  Can neuralgias arise from minor demyelination? Spontaneous firing, mechanosensitivity, and afterdischarge from conducting axons , 1982, Experimental Neurology.

[50]  M. Rasminsky.  Hyperexcitability of pathologically myelinated axons and positive symptoms in multiple sclerosis. , 1981, Advances in neurology.

[51]  W. Mcdonald,et al.  Spontaneous and mechanically evoked activity due to central demyelinating lesion , 1980, Nature.

[52]  Tilson Ha,et al.  A method for the routine assessment of fore- and hindlimb grip strength of rats and mice. , 1979 .

[53]  H. Tilson,et al.  A method for the routine assessment of fore- and hindlimb grip strength of rats and mice. , 1979, Neurobehavioral toxicology.

[54]  A. Aguayo,et al.  Myelination of mouse axons by Schwann cells transplanted from normal and abnormal human nerves , 1977, Nature.

[55]  P K Thomas,et al.  Peripheral nerve structure and function in experimental diabetes. , 1974, Journal of the neurological sciences.

[56]  A. Iggo,et al.  The electrophysiological identification of single nerve fibres, with particular reference to the slowest‐conducting vagal afferent fibres in the cat , 1958, The Journal of physiology.