Metabolic endotoxemia and saturated fat contribute to circulating NGAL concentrations in subjects with insulin resistance
暂无分享,去创建一个
G Nolfe | M. Izquierdo | J. Moreno-Navarrete | W. Ricart | J. Fernandez-Real | F. Ortega | J. Fernández-Real | F. Tinahones | W. Ricart | E. García‐Fuentes | J. Vendrell | G. Mingrone | E. Gorostiaga | C. Martínez | M. Manco | G. Nolfe | M Izquierdo | J. Ibáñez | W Ricart | E Gorostiaga | J M Moreno-Navarrete | M Manco | J Ibáñez | E García-Fuentes | F Ortega | J Vendrell | C Martínez | G Mingrone | F Tinahones | J M Fernández-Real | Mikel Izquierdo | Melania Manco | J. Vendrell | G. Mingrone | F. J. Tinahones | Francisco J. Ortega | Giuseppe Nolfe | F. Tinahones
[1] A. Madan,et al. TNFα release by the nonfat cells of human adipose tissue , 2004, International Journal of Obesity.
[2] Laura Herrero,et al. Obesity, inflammation, and insulin resistance. , 2007, Gastroenterology.
[3] J. Fernández-Real,et al. Massive Weight Loss Decreases Corticosteroid-Binding Globulin Levels and Increases Free Cortisol in Healthy Obese Patients , 2007, Diabetes Care.
[4] S. Ohlson,et al. Interactions between neutrophil gelatinase-associated lipocalin and natural lipophilic ligands. , 1999, Biochimica et biophysica acta.
[5] Nancy R Cook,et al. C-Reactive Protein, the Metabolic Syndrome, and Risk of Incident Cardiovascular Events: An 8-Year Follow-Up of 14 719 Initially Healthy American Women , 2003, Circulation.
[6] J. Ferrières,et al. Metabolic Endotoxemia Initiates Obesity and Insulin Resistance , 2007, Diabetes.
[7] AntonioCeriello,et al. Is Oxidative Stress the Pathogenic Mechanism Underlying Insulin Resistance, Diabetes, and Cardiovascular Disease? The Common Soil Hypothesis Revisited , 2004 .
[8] C. Apovian. A high-fat meal induces low-grade endotoxemia: evidence of a novel mechanism of postprandial inflammation , 2008 .
[9] J. Cowland,et al. IL-1β-Specific Up-Regulation of Neutrophil Gelatinase-Associated Lipocalin Is Controlled by IκB-ζ1 , 2006, Journal of Immunology.
[10] Shizuo Akira,et al. Lipocalin 2 mediates an innate immune response to bacterial infection by sequestrating iron , 2004, Nature.
[11] E. Kraegen,et al. Lipocalin-2 is an inflammatory marker closely associated with obesity, insulin resistance, and hyperglycemia in humans. , 2007, Clinical chemistry.
[12] A. Aljada,et al. Elevation of free fatty acids induces inflammation and impairs vascular reactivity in healthy subjects. , 2003, Diabetes.
[13] S. Simpson. Of Mice . . . , 2004, Science.
[14] A. Van de Voorde,et al. Identification by microsequencing of lipopolysaccharide-induced proteins secreted by mouse macrophages. , 1993, Journal of immunology.
[15] R. Bibiloni,et al. Changes in Gut Microbiota Control Metabolic Endotoxemia-Induced Inflammation in High-Fat Diet–Induced Obesity and Diabetes in Mice , 2008, Diabetes.
[16] G. Hotamisligil,et al. Inflammation and metabolic disorders , 2006, Nature.
[17] J. Fernández-Real,et al. Insulin resistance and chronic cardiovascular inflammatory syndrome. , 2003, Endocrine reviews.
[18] E. Ferrannini,et al. Leptin pulsatility in formerly obese women , 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[19] N. Houstis,et al. The Adipokine Lipocalin 2 Is Regulated by Obesity and Promotes Insulin Resistance , 2007, Diabetes.
[20] A. Gabrielsen,et al. Induction of Neutrophil Gelatinase-Associated Lipocalin in Vascular Injury via Activation of Nuclear Factor-κB , 2006 .
[21] H. Wallenburg,et al. Tumor necrosis factor-alpha in whole blood cultures of preeclamptic patients and healthy pregnant and nonpregnant women. , 2004, Hypertension in pregnancy.
[22] J. B. Kim,et al. Activation of Toll-like receptor 4 is associated with insulin resistance in adipocytes. , 2006, Biochemical and biophysical research communications.
[23] S. Haffner,et al. Chronic subclinical inflammation as part of the insulin resistance syndrome: the Insulin Resistance Atherosclerosis Study (IRAS). , 2000, Circulation.
[24] R. Gomis,et al. Evidence of expression of endotoxin receptors CD14, toll‐like receptors TLR4 and TLR2 and associated molecule MD‐2 and of sensitivity to endotoxin (LPS) in islet beta cells , 2003, Clinical and experimental immunology.
[25] T. Wadden,et al. Tumor Necrosis Factor-α in Sera of Obese Patients: Fall with Weight Loss , 1998 .
[26] M. A. Crook,et al. Is Type II diabetes mellitus a disease of the innate immune system? , 1998, Diabetologia.
[27] W. Ricart,et al. Effect of massive weight loss on inflammatory adipocytokines and the innate immune system in morbidly obese women. , 2007, The Journal of clinical endocrinology and metabolism.
[28] J. Cook,et al. Abnormal Connexin Expression Underlies Delayed Wound Healing in Diabetic Skin , 2007, Diabetes.
[29] Tongzheng Liu,et al. Sensitivity of mice to lipopolysaccharide is increased by a high saturated fat and cholesterol diet , 2007, Journal of Inflammation.
[30] T. Wadden,et al. Tumor necrosis factor-alpha in sera of obese patients: fall with weight loss. , 1998, The Journal of clinical endocrinology and metabolism.
[31] T. Hartung,et al. Whole blood cytokine response as a measure of immunotoxicity. , 2001, Toxicology in vitro : an international journal published in association with BIBRA.
[32] M. Desai,et al. Obesity is associated with macrophage accumulation in adipose tissue. , 2003, The Journal of clinical investigation.
[33] V. Giusti,et al. Adipose tissue is a major source of interleukin-1 receptor antagonist: upregulation in obesity and inflammation. , 2003, Diabetes.
[34] D. Bernlohr,et al. The role of lipocalin 2 in the regulation of inflammation in adipocytes and macrophages. , 2008, Molecular endocrinology.
[35] A. Aljada,et al. Both lipid and protein intakes stimulate increased generation of reactive oxygen species by polymorphonuclear leukocytes and mononuclear cells. , 2002, The American journal of clinical nutrition.
[36] J. Fernández-Real,et al. Burden of infection and insulin resistance in healthy middle-aged men. , 2006, Diabetes care.
[37] E. Bonora,et al. Homeostasis model assessment closely mirrors the glucose clamp technique in the assessment of insulin sensitivity: studies in subjects with various degrees of glucose tolerance and insulin sensitivity. , 2000, Diabetes care.
[38] J. Fernández-Real,et al. Serum Ferritin as a Component of the Insulin Resistance Syndrome , 1998, Diabetes Care.
[39] H. Wallenburg,et al. Tumor Necrosis Factor‐α in Whole Blood Cultures of Preeclamptic Patients and Healthy Pregnant and Nonpregnant Women , 2004 .
[40] R. Turner,et al. Homeostasis model assessment: insulin resistance and β-cell function from fasting plasma glucose and insulin concentrations in man , 1985, Diabetologia.
[41] D. Hwang,et al. The modulation of inflammatory gene expression by lipids: mediation through Toll-like receptors. , 2006, Molecules and cells.
[42] B. Pedersen,et al. Effect of short-term intralipid infusion on the immune response during low-dose endotoxemia in humans. , 2008, American journal of physiology. Endocrinology and metabolism.
[43] R. Armen,et al. Ligand preference inferred from the structure of neutrophil gelatinase associated lipocalin. , 2000, Biochemistry.
[44] Herbert Tilg,et al. Adipocytokines: mediators linking adipose tissue, inflammation and immunity , 2006, Nature Reviews Immunology.
[45] H. Sengeløv,et al. Isolation and primary structure of NGAL, a novel protein associated with human neutrophil gelatinase. , 1993, The Journal of biological chemistry.
[46] N. Barzilai,et al. Hyperglycemia-induced Production of Acute Phase Reactants in Adipose Tissue* , 2001, The Journal of Biological Chemistry.
[47] J. Cowland,et al. IL-1beta-specific up-regulation of neutrophil gelatinase-associated lipocalin is controlled by IkappaB-zeta. , 2006, Journal of immunology.