Clinicopathological behaviour of multiple oral dysplastic lesions compared with that of single lesions.

[1]  O. Hamadah,et al.  Factors affecting carbon dioxide laser treatment for oral precancer: A patient cohort study , 2009, Lasers in surgery and medicine.

[2]  G. Dakubo,et al.  Clinical implications and utility of field cancerization , 2007, Cancer Cell International.

[3]  P. Thomson,et al.  Cancerisation within the oral cavity: the use of 'field mapping biopsies' in clinical management. , 2007, Oral oncology.

[4]  R. Greer.  Pathology of malignant and premalignant oral epithelial lesions. , 2006, Otolaryngologic clinics of North America.

[5]  J. Soames,et al.  Epithelial cell proliferative activity and oral cancer progression , 2002, Cell proliferation.

[6]  P. Thomson,et al.  Interventional laser surgery: an effective surgical and diagnostic tool in oral precancer management. , 2002, International journal of oral and maxillofacial surgery.

[7]  G. Lodi,et al.  Interventions for treating oral leukoplakia. , 2006, The Cochrane database of systematic reviews.

[8]  A. Hirai,et al.  High malignant transformation rate of widespread multiple oral leukoplakias. , 2008, Oral diseases.

[9]  H Lumerman,et al.  Oral epithelial dysplasia and the development of invasive squamous cell carcinoma. , 1995, Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics.

[10]  K. Chew,et al.  Which oral white lesions will become malignant? An image cytometric study. , 1990, Oral surgery, oral medicine, and oral pathology.

[11]  M. Gorsky,et al.  Oral leukoplakia and malignant transformation. A follow‐up study of 257 patients , 1984, Cancer.

[12]  D. Slaughter,et al.  “Field cancerization” in oral stratified squamous epithelium. Clinical implications of multicentric origin , 1953, Cancer.