Lipoxin A4 activates ALX/FPR2 Receptor to Regulate Conjunctival Goblet Cell Secretion
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Jeffrey A. Bair | C. Serhan | Dayu Li | R. Hodges | D. Dartt | M. Shatos | Marit Lippestad
[1] K. Gronert,et al. Female-Specific Downregulation of Tissue Polymorphonuclear Neutrophils Drives Impaired Regulatory T Cell and Amplified Effector T Cell Responses in Autoimmune Dry Eye Disease , 2015, The Journal of Immunology.
[2] P. Mohler,et al. Role of CaMKII in cardiac arrhythmias. , 2015, Trends in cardiovascular medicine.
[3] I. Judson,et al. Targeting protein kinase C in sarcoma. , 2014, Biochimica et biophysica acta.
[4] D. Dartt,et al. Conjunctival epithelial and goblet cell function in chronic inflammation and ocular allergic inflammation , 2014, Current opinion in allergy and clinical immunology.
[5] Charles N. Serhan,et al. Pro-resolving lipid mediators are leads for resolution physiology , 2014, Nature.
[6] S. Bonini. Allergy and the eye. , 2014, Chemical immunology and allergy.
[7] D. Dartt,et al. Modulation of Conjunctival Goblet Cell Function by Inflammatory Cytokines , 2013, Mediators of inflammation.
[8] M. Perretti,et al. Ligand-specific conformational change of the G-protein–coupled receptor ALX/FPR2 determines proresolving functional responses , 2013, Proceedings of the National Academy of Sciences.
[9] Jonathan E. Moore,et al. Staphylococcus aureus Activates the NLRP3 Inflammasome in Human and Rat Conjunctival Goblet Cells , 2013, PloS one.
[10] Jianwei Jiao,et al. Effect of VIP on intracellular [Ca2+], extracellular regulated kinase 1/2, and secretion in cultured rat conjunctival goblet cells. , 2013, Investigative ophthalmology & visual science.
[11] C. Serhan,et al. Resolvin D1 and Aspirin-Triggered Resolvin D1 Regulate Histamine-stimulated Conjunctival Goblet Cell Secretion , 2013, Mucosal Immunology.
[12] Dayu Li,et al. Effect of histamine on Ca(2+)-dependent signaling pathways in rat conjunctival goblet cells. , 2012, Investigative ophthalmology & visual science.
[13] Jeffrey A. Bair,et al. Signaling pathways used by EGF to stimulate conjunctival goblet cell secretion. , 2012, Experimental eye research.
[14] Dayu Li,et al. Role of histamine and its receptor subtypes in stimulation of conjunctival goblet cell secretion. , 2012, Investigative ophthalmology & visual science.
[15] C. Serhan,et al. Conjunctival Goblet Cell Secretion Stimulated by Leukotrienes Is Reduced by Resolvins D1 and E1 To Promote Resolution of Inflammation , 2011, The Journal of Immunology.
[16] V. Natarajan,et al. Phospholipase D1, but not D2, regulates protein secretion via Rho/ROCK in a Ras/Raf-independent, MEK-dependent manner in rat lacrimal gland. , 2011, Investigative ophthalmology & visual science.
[17] M. Perretti,et al. FPR2/ALX receptor expression and internalization are critical for lipoxin A4 and annexin‐derived peptide‐stimulated phagocytosis , 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[18] F. Hofmann,et al. A complex of CaV1.2/PKC is involved in muscarinic signaling in smooth muscle , 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[19] J. Gu,et al. ERK/p44p42 mitogen-activated protein kinase mediates EGF-stimulated proliferation of conjunctival goblet cells in culture. , 2008, Investigative ophthalmology & visual science.
[20] D. Dartt,et al. Effect of OPC-12759 on EGF receptor activation, p44/p42 MAPK activity, and secretion in conjunctival goblet cells. , 2008, Experimental eye research.
[21] P. Maderna,et al. Lipoxins and Annexin-1: Resolution of Inflammation and Regulation of Phagocytosis of Apoptotic Cells , 2006, TheScientificWorldJournal.
[22] Marc Parmentier,et al. Formyl peptide receptors: a promiscuous subfamily of G protein-coupled receptors controlling immune responses. , 2006, Cytokine & growth factor reviews.
[23] Takao Shimizu,et al. The Lipoxin Receptor ALX: Potent Ligand-Specific and Stereoselective Actions in Vivo , 2006, Pharmacological Reviews.
[24] K. Gronert. Lipoxins in the eye and their role in wound healing. , 2005, Prostaglandins, leukotrienes, and essential fatty acids.
[25] C. Serhan,et al. Anti-inflammatory circuitry: lipoxin, aspirin-triggered lipoxins and their receptor ALX. , 2005, Prostaglandins, leukotrienes, and essential fatty acids.
[26] H. Kuramoto,et al. Regulatory expression of lipoxin A4 receptor in physiologically estrus cycle and pathologically endometriosis. , 2005, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[27] N. Maheshwari,et al. A Role for the Mouse 12/15-Lipoxygenase Pathway in Promoting Epithelial Wound Healing and Host Defense* , 2005, Journal of Biological Chemistry.
[28] S. Ryu,et al. Inhibition of Muscarinic Receptor-linked Phospholipase D Activation by Association with Tubulin* , 2005, Journal of Biological Chemistry.
[29] M. Cobb,et al. Regulation of ERK1 and ERK2 by Glucose and Peptide Hormones in Pancreatic β Cells* , 2003, Journal of Biological Chemistry.
[30] R. Hodges,et al. Isolation and characterization of cultured human conjunctival goblet cells. , 2003, Investigative ophthalmology & visual science.
[31] D. Zoukhri,et al. Activation of mitogen-activated protein kinase by cholinergic agonists and EGF in human compared with rat cultured conjunctival goblet cells. , 2003, Investigative ophthalmology & visual science.
[32] T. Takano,et al. A novel rat lipoxin A4 receptor that is conserved in structure and function , 2003, British journal of pharmacology.
[33] D. Zoukhri,et al. Cholinergic agonists transactivate EGFR and stimulate MAPK to induce goblet cell secretion. , 2003, American journal of physiology. Cell physiology.
[34] B. H. Shah,et al. Calcium-independent activation of extracellularly regulated kinases 1 and 2 by angiotensin II in hepatic C9 cells: roles of protein kinase Cdelta, Src/proline-rich tyrosine kinase 2, and epidermal growth receptor trans-activation. , 2002, Molecular pharmacology.
[35] R. Hodges,et al. Isolation, characterization, and propagation of rat conjunctival goblet cells in vitro. , 2001, Investigative ophthalmology & visual science.
[36] C. Serhan,et al. Activation of Lipoxin a4 Receptors by Aspirin-Triggered Lipoxins and Select Peptides Evokes Ligand-Specific Responses in Inflammation , 2000, The Journal of experimental medicine.
[37] C. Clish,et al. Local and systemic delivery of a stable aspirin-triggered lipoxin prevents neutrophil recruitment in vivo. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[38] T. Takano,et al. Aspirin-triggered 15-Epi-Lipoxin A4 (LXA4) and LXA4 Stable Analogues Are Potent Inhibitors of Acute Inflammation: Evidence for Anti-inflammatory Receptors , 1997, The Journal of experimental medicine.
[39] T. Takano,et al. Lipoxin A4 Stable Analogs Are Potent Mimetics That Stimulate Human Monocytes and THP-1 Cells via a G-protein-linked Lipoxin A4 Receptor* , 1997, The Journal of Biological Chemistry.
[40] C. Serhan,et al. Activation of human monocytes and the acute monocytic leukemia cell line (THP-1) by lipoxins involves unique signaling pathways for lipoxin A4 versus lipoxin B4: evidence for differential Ca2+ mobilization. , 1996, Journal of immunology.
[41] D. Zoukhri,et al. Cholinergic activation of phospholipase D in lacrimal gland acini is independent of protein kinase C and calcium. , 1995, The American journal of physiology.
[42] C. Serhan,et al. Induction of functional lipoxin A4 receptors in HL-60 cells. , 1993, Blood.
[43] F. Luscinskas,et al. Lipoxin A4 and lipoxin B4 stimulate the release but not the oxygenation of arachidonic acid in human neutrophils: Dissociation between lipid remodeling and adhesion , 1990, Journal of cellular physiology.