Multiplex mapping of chromatin accessibility and DNA methylation within targeted single molecules identifies epigenetic heterogeneity in neural stem cells and glioblastoma
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Alberto Riva | Russell P. Darst | A. Riva | M. Kladde | Brent A Reynolds | B. Reynolds | Nancy H Nabilsi | Loic P Deleyrolle | Russell P Darst | Michael P Kladde | L. Deleyrolle | N. Nabilsi
[1] Peter A. Jones,et al. OCT4 establishes and maintains nucleosome-depleted regions that provide additional layers of epigenetic regulation of its target genes , 2011, Proceedings of the National Academy of Sciences.
[2] Carolina E. Pardo,et al. Simultaneous Single‐Molecule Mapping of Protein‐DNA Interactions and DNA Methylation by MAPit , 2011, Current protocols in molecular biology.
[3] Gangning Liang,et al. Genome-wide mapping of nucleosome positioning and DNA methylation within individual DNA molecules , 2012, Genome research.
[4] Cizhong Jiang,et al. Nucleosome positioning and gene regulation: advances through genomics , 2009, Nature Reviews Genetics.
[5] G. Reifenberger,et al. Genetic alteration and expression of the phosphoinositol‐3‐kinase/Akt pathway genes PIK3CA and PIKE in human glioblastomas , 2005, Neuropathology and applied neurobiology.
[6] A. Verma. MGMT Gene Silencing and Benefit From Temozolomide in Glioblastoma , 2006 .
[7] A. Feinberg,et al. Increased methylation variation in epigenetic domains across cancer types , 2011, Nature Genetics.
[8] M. Frommer,et al. CpG islands in vertebrate genomes. , 1987, Journal of molecular biology.
[9] H. Mehdorn,et al. The expression of mismatch repair proteins MLH1, MSH2 and MSH6 correlates with the Ki67 proliferation index and survival in patients with recurrent glioblastoma , 2010, Neurological research.
[10] A. Feinberg,et al. Stochastic epigenetic variation as a driving force of development, evolutionary adaptation, and disease , 2010, Proceedings of the National Academy of Sciences.
[11] Carolina E. Pardo,et al. WIF1 is a frequent target for epigenetic silencing in squamous cell carcinoma of the cervix. , 2011, Carcinogenesis.
[12] T. Tsuruo,et al. Promotion of glioma cell survival by acyl-CoA synthetase 5 under extracellular acidosis conditions , 2009, Oncogene.
[13] R. Mitra,et al. Bisulfite Patch PCR enables multiplexed sequencing of promoter methylation across cancer samples. , 2010, Genome research.
[14] J. Visvader,et al. Cancer stem cells in solid tumours: accumulating evidence and unresolved questions , 2008, Nature Reviews Cancer.
[15] Peter A. Jones,et al. A decade of exploring the cancer epigenome — biological and translational implications , 2011, Nature Reviews Cancer.
[16] Darryl Shibata,et al. Tumour susceptibility and spontaneous mutation in mice deficient in Mlh1, Pms1 and Pms2 DMA mismatch repair , 1998, Nature Genetics.
[17] H. Augustin,et al. Transcriptional profiling of human glioblastoma vessels indicates a key role of VEGF‐A and TGFβ2 in vascular abnormalization , 2012, The Journal of pathology.
[18] J. Herman,et al. Gene silencing in cancer in association with promoter hypermethylation. , 2003, The New England journal of medicine.
[19] A. Vescovi,et al. Evidence for label-retaining tumour-initiating cells in human glioblastoma. , 2011, Brain : a journal of neurology.
[20] A. Monks,et al. Characterization of MLH1 and MSH2 DNA mismatch repair proteins in cell lines of the NCI anticancer drug screen , 2000, Cancer Chemotherapy and Pharmacology.
[21] Carolina E. Pardo,et al. DNA methyltransferase probing of chromatin structure within populations and on single molecules. , 2009, Methods in molecular biology.
[22] H. Yoshioka,et al. Ubiquitous Expression of the α1(XIX) Collagen Gene (Col19a1) during Mouse Embryogenesis Becomes Restricted to a Few Tissues in the Adult Organism* , 1997, The Journal of Biological Chemistry.
[23] Carolina E. Pardo,et al. Epigenetic diversity of Kaposi’s sarcoma–associated herpesvirus , 2013, Nucleic acids research.
[24] Carolina E. Pardo,et al. DNA methyltransferase accessibility protocol for individual templates by deep sequencing. , 2012, Methods in enzymology.
[25] S. Rafii,et al. Alternative promoters regulate transcription of the gene that encodes stem cell surface protein AC133. , 2004, Blood.
[26] François Fuks,et al. DNA methylation and histone modifications: teaming up to silence genes. , 2005, Current opinion in genetics & development.
[27] M. Kool,et al. Mismatch repair deficiency: a temozolomide resistance factor in medulloblastoma cell lines that is uncommon in primary medulloblastoma tumours , 2012, British Journal of Cancer.
[28] J. V. Van Etten,et al. Cloning, characterization and expression of the gene coding for a cytosine-5-DNA methyltransferase recognizing GpC. , 1998, Nucleic acids research.
[29] Ian M. Carr,et al. MethylViewer: computational analysis and editing for bisulfite sequencing and methyltransferase accessibility protocol for individual templates (MAPit) projects , 2010, Nucleic Acids Res..
[30] Peter A. Jones,et al. Gene Reactivation by 5-Aza-2′-Deoxycytidine–Induced Demethylation Requires SRCAP–Mediated H2A.Z Insertion to Establish Nucleosome Depleted Regions , 2012, PLoS genetics.
[31] Peter A. Jones,et al. Hypomethylation of a LINE-1 Promoter Activates an Alternate Transcript of the MET Oncogene in Bladders with Cancer , 2010, PLoS genetics.
[32] S. Markowitz,et al. Mismatch repair mutations override alkyltransferase in conferring resistance to temozolomide but not to 1,3-bis(2-chloroethyl)nitrosourea. , 1996, Cancer research.
[33] M. Kladde,et al. Gal4p-Mediated Chromatin Remodeling Depends on Binding Site Position in Nucleosomes but Does Not Require DNA Replication , 1998, Molecular and Cellular Biology.
[34] Brent A Reynolds,et al. Isolation, expansion, and differentiation of adult Mammalian neural stem and progenitor cells using the neurosphere assay. , 2009, Methods in molecular biology.
[35] D. Ward,et al. Mutation in the DNA mismatch repair gene homologue hMLH 1 is associated with hereditary non-polyposis colon cancer , 1994, Nature.
[36] Scott A. Hoose,et al. Single-molecule and population probing of chromatin structure using DNA methyltransferases. , 2007, Methods.
[37] M. Kladde,et al. Direct study of DNA‐protein interactions in repressed and active chromatin in living cells. , 1996, The EMBO journal.
[38] M. Esteller. Cancer epigenomics: DNA methylomes and histone-modification maps , 2007, Nature Reviews Genetics.
[39] M. S. Kallos,et al. Bioreactor expansion of human neural precursor cells in serum‐free media retains neurogenic potential , 2009, Biotechnology and bioengineering.
[40] Martin J. van den Bent,et al. Radiotherapy plus concomitant and adjuvant temozolomide for glioblastoma. , 2005, The New England journal of medicine.
[41] Erik Sundström,et al. Cellular composition of long‐term human spinal cord‐ and forebrain‐derived neurosphere cultures , 2006, Journal of neuroscience research.
[42] Austin G Smith,et al. CD133 (Prominin) Negative Human Neural Stem Cells Are Clonogenic and Tripotent , 2009, PloS one.
[43] Daiya Takai,et al. Comprehensive analysis of CpG islands in human chromosomes 21 and 22 , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[44] S. Schreiber,et al. Signaling Network Model of Chromatin , 2002, Cell.
[45] Ben S. Wittner,et al. A Chromatin-Mediated Reversible Drug-Tolerant State in Cancer Cell Subpopulations , 2010, Cell.
[46] G. Reifenberger,et al. Promoter methylation and expression of MGMT and the DNA mismatch repair genes MLH1, MSH2, MSH6 and PMS2 in paired primary and recurrent glioblastomas , 2011, International journal of cancer.