Complement C3 Drives Autophagy-Dependent Restriction of Cyto-invasive Bacteria.
暂无分享,去创建一个
D. Philpott | S. Girardin | I. Tattoli | Matthew T. Sorbara | J. Rohde | D. Isenman | R. van Dalen | M. Abdel-Nour | Jessica Tsalikis | Imogen Sirluck-Schroeder | Elisabeth G. Foerster | Joseph Mangiapane | E. G. Foerster | E. Foerster | John Rohde
[1] M. Krönke,et al. The β2 Integrin Mac-1 Induces Protective LC3-Associated Phagocytosis of Listeria monocytogenes. , 2018, Cell host & microbe.
[2] M. Huber-Lang,et al. Regulation of epithelial cell expressed C3 in the intestine - Relevance for the pathophysiology of inflammatory bowel disease? , 2017, Molecular immunology.
[3] G. Tsokos,et al. Intracellular Activation of Complement 3 Is Responsible for Intestinal Tissue Damage during Mesenteric Ischemia , 2017, The Journal of Immunology.
[4] B. Kwon,et al. Extracellular stimulation of VSIG4/complement receptor Ig suppresses intracellular bacterial infection by inducing autophagy , 2016, Autophagy.
[5] C. Streutker,et al. NLRX1 Acts as an Epithelial-Intrinsic Tumor Suppressor through the Modulation of TNF-Mediated Proliferation. , 2016, Cell reports.
[6] R. Einspanier,et al. Mycobacterium bovis BCG Interferes with miR-3619-5p Control of Cathepsin S in the Process of Autophagy , 2016, Front. Cell. Infect. Microbiol..
[7] J. Atkinson,et al. Complement regulator CD46: genetic variants and disease associations , 2015, Human Genomics.
[8] V. Frémeaux-Bacchi,et al. Complement System Part I – Molecular Mechanisms of Activation and Regulation , 2015, Front. Immunol..
[9] Lubka T. Roumenina,et al. Complement System Part II: Role in Immunity , 2015, Front. Immunol..
[10] S. Meri,et al. The outer membrane protease PgtE of Salmonella enterica interferes with the alternative complement pathway by cleaving factors B and H , 2015, Front. Microbiol..
[11] S. Girardin,et al. Emerging themes in bacterial autophagy. , 2015, Current opinion in microbiology.
[12] L. James,et al. Intracellular sensing of complement C3 activates cell autonomous immunity , 2014, Science.
[13] A. Stadnyk,et al. The Complement System in Inflammatory Bowel Disease , 2014, Inflammatory bowel diseases.
[14] Jacob D. Jaffe,et al. Atg16L1 T300A variant decreases selective autophagy resulting in altered cytokine signaling and decreased antibacterial defense , 2014, Proceedings of the National Academy of Sciences.
[15] J. Brumell,et al. Bacteria–autophagy interplay: a battle for survival , 2014, Nature Reviews Microbiology.
[16] R. Xavier,et al. Atg16l1 is required for autophagy in intestinal epithelial cells and protection of mice from Salmonella infection. , 2013, Gastroenterology.
[17] D. Philpott,et al. Listeria phospholipases subvert host autophagic defenses by stalling pre‐autophagosomal structures , 2013, The EMBO journal.
[18] D. Philpott,et al. The protein ATG16L1 suppresses inflammatory cytokines induced by the intracellular sensors Nod1 and Nod2 in an autophagy-independent manner. , 2013, Immunity.
[19] Le Cong,et al. Multiplex Genome Engineering Using CRISPR/Cas Systems , 2013, Science.
[20] M. Overholtzer,et al. Edinburgh Research Explorer Interaction between FIP200 and ATG16L1 distinguishes ULK1 complex-dependent and -independent autophagy , 2022 .
[21] Sangeeta Khare,et al. Guidelines for the use and interpretation of assays formonitoring autophagy (3rd edition) , 2016 .
[22] E. Wiemer,et al. Recruitment of the Major Vault Protein by InlK: A Listeria monocytogenes Strategy to Avoid Autophagy , 2011, PLoS pathogens.
[23] K. Takeuchi,et al. The increased mucosal mRNA expressions of complement C3 and interleukin‐17 in inflammatory bowel disease , 2010, Clinical and experimental immunology.
[24] R. Xavier,et al. Crohn's disease‐associated adherent‐invasive E. coli are selectively favoured by impaired autophagy to replicate intracellularly , 2010, Cellular microbiology.
[25] M. Vidal,et al. Autophagy induction by the pathogen receptor CD46. , 2009, Cell host & microbe.
[26] T. Noda,et al. The Atg16L complex specifies the site of LC3 lipidation for membrane biogenesis in autophagy. , 2008, Molecular biology of the cell.
[27] A. Gruber,et al. Extending the Host Range of Listeria monocytogenes by Rational Protein Design , 2007, Cell.
[28] Veronica Canadien,et al. Listeria monocytogenes Evades Killing by Autophagy During Colonization of Host Cells , 2007, Autophagy.
[29] S. Meri,et al. The surface protease PgtE of Salmonella enterica affects complement activity by proteolytically cleaving C3b, C4b and C5 , 2007, FEBS letters.
[30] Thomas Lengauer,et al. A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1 , 2007, Nature Genetics.
[31] C. Stathopoulos,et al. Omptin proteins: an expanding family of outer membrane proteases in Gram-negative Enterobacteriaceae (Review) , 2007, Molecular membrane biology.
[32] Masaaki Komatsu,et al. Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice , 2005, The Journal of cell biology.
[33] Laurent Beaugerie,et al. High prevalence of adherent-invasive Escherichia coli associated with ileal mucosa in Crohn's disease. , 2004, Gastroenterology.
[34] Jean-Frederic Colombel,et al. Adherent Invasive Escherichia coli Strains from Patients with Crohn's Disease Survive and Replicate within Macrophages without Inducing Host Cell Death , 2001, Infection and Immunity.
[35] M. Goldberg,et al. Disruption of IcsP, the major Shigella protease that cleaves IcsA, accelerates actin‐based motility , 1997, Molecular microbiology.