Quantitative cortical synapse proteomics of a transgenic migraine mouse model with mutated CaV2.1 calcium channels
暂无分享,去创建一个
M. Ferrari | A. Smit | S. Spijker | I. Sidorov | R. Frants | M. Loos | A. M. van den Maagdenberg | O. Mayboroda | A. Deelder | K. Li | L. Broos | O. Klychnikov | M. Ferrari
[1] M. Ferrari,et al. Enhanced Excitatory Transmission at Cortical Synapses as the Basis for Facilitated Spreading Depression in CaV2.1 Knockin Migraine Mice , 2009, Neuron.
[2] K. Kolkova. Biosynthesis of NCAM , 2008, Neurochemical Research.
[3] K. Kolkova,et al. Signaling Pathways Involved in NCAM-Induced Neurite Outgrowth , 2008, Neurochemical Research.
[4] A. V. D. Maagdenberg,et al. The S218L familial hemiplegic migraine mutation promotes deinhibition of Cav2.1 calcium channels during direct G-protein regulation , 2008, Pflügers Archiv - European Journal of Physiology.
[5] V. Berezin,et al. The NCAM‐derived P2 peptide facilitates recovery of cognitive and motor function and ameliorates neuropathology following traumatic brain injury , 2008, The European journal of neuroscience.
[6] Michael J. Albright,et al. Increased thalamocortical synaptic response and decreased layer IV innervation in GAP-43 knockout mice. , 2007, Journal of neurophysiology.
[7] Ka Wan Li,et al. Quantitative proteomics and protein network analysis of hippocampal synapses of CaMKIIalpha mutant mice. , 2007, Journal of proteome research.
[8] M. Ferrari,et al. Migraine: gene mutations and functional consequences , 2007, Current opinion in neurology.
[9] V. Berezin,et al. GAP‐43 regulates NCAM‐180‐mediated neurite outgrowth , 2006, Journal of neurochemistry.
[10] J. Presley,et al. Characterization of the localization and function of NECAP 1 in neurons , 2006, Journal of neurochemistry.
[11] R. Baloh,et al. Mutation in the glutamate transporter EAAT1 causes episodic ataxia, hemiplegia, and seizures , 2005, Neurology.
[12] Mu-ming Poo,et al. Shrinkage of Dendritic Spines Associated with Long-Term Depression of Hippocampal Synapses , 2004, Neuron.
[13] Simon Kaja,et al. A Cacna1a Knockin Migraine Mouse Model with Increased Susceptibility to Cortical Spreading Depression , 2004, Neuron.
[14] Jes Olesen,et al. The International Classification of Headache Disorders: 2nd edition. , 2004, Cephalalgia : an international journal of headache.
[15] M. Gonzalez-Gaitan,et al. Role of Drosophila Rab5 during endosomal trafficking at the synapse and evoked neurotransmitter release , 2003, The Journal of cell biology.
[16] G. Seabrook,et al. The familial hemiplegic migraine mutation R192q reduces G‐protein‐mediated inhibition of p/q‐type (Cav2.1) calcium channels expressed in human embryonic kidney cells , 2003, The Journal of physiology.
[17] E. Vicaut,et al. The clinical spectrum of familial hemiplegic migraine associated with mutations in a neuronal calcium channel. , 2001, The New England journal of medicine.
[18] L Shapiro,et al. Making memories stick: cell-adhesion molecules in synaptic plasticity. , 2000, Trends in cell biology.
[19] F. Walsh,et al. Neurite Outgrowth Stimulated by Neural Cell Adhesion Molecules Requires Growth-Associated Protein-43 (GAP-43) Function and Is Associated with GAP-43 Phosphorylation in Growth Cones , 1998, The Journal of Neuroscience.
[20] T. Südhof,et al. A Tripartite Protein Complex with the Potential to Couple Synaptic Vesicle Exocytosis to Cell Adhesion in Brain , 1998, Cell.
[21] R. Ophoff,et al. Clinical and genetic analysis of a large Dutch family with autosomal dominant vascular retinopathy, migraine and Raynaud's phenomenon. , 1998, Brain : a journal of neurology.
[22] P. Caroni,et al. Intrinsic Neuronal Determinants Locally Regulate Extrasynaptic and Synaptic Growth at the Adult Neuromuscular Junction , 1997, The Journal of cell biology.
[23] Dennis E Bulman,et al. Familial Hemiplegic Migraine and Episodic Ataxia Type-2 Are Caused by Mutations in the Ca2+ Channel Gene CACNL1A4 , 1996, Cell.
[24] J. Kapfhammer,et al. Overexpression of the neural growth-associated protein GAP-43 induces nerve sprouting in the adult nervous system of transgenic mice , 1995, Cell.
[25] W. Regehr,et al. Calcium control of transmitter release at a cerebellar synapse , 1995, Neuron.
[26] R. Baloh,et al. Episodic ataxia associated with EAAT1 mutation C186S affecting glutamate reuptake. , 2009, Archives of neurology.
[27] Peter J. Goadsby,et al. 32 – Migraine as a Cerebral Ionopathy with Abnormal Central Sensory Processing* , 2007 .
[28] M. Mosevitsky. Nerve ending "signal" proteins GAP-43, MARCKS, and BASP1. , 2005, International review of cytology.