Solar flair.

[1]  M. Stampfer Cholera toxin stimulation of human mammary epithelial cells in culture , 1982, In Vitro.

[2]  V. Tsikitis,et al.  Biology of Ductal Carcinoma in Situ Classification Based on Biologic Potential , 2006, American journal of clinical oncology.

[3]  G. Evan,et al.  Reversible kinetic analysis of Myc targets in vivo provides novel insights into Myc-mediated tumorigenesis. , 2006, Cancer research.

[4]  Howard Y. Chang,et al.  Genetic regulators of large-scale transcriptional signatures in cancer , 2006, Nature Genetics.

[5]  Gavin D. Grant,et al.  Common markers of proliferation , 2006, Nature Reviews Cancer.

[6]  P. Hall,et al.  An expression signature for p53 status in human breast cancer predicts mutation status, transcriptional effects, and patient survival. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[7]  J. Campisi,et al.  Caspase‐independent cytochrome c release is a sensitive measure of low‐level apoptosis in cell culture models , 2005, Aging cell.

[8]  I. Poola,et al.  Identification of MMP-1 as a putative breast cancer predictive marker by global gene expression analysis , 2005, Nature Medicine.

[9]  T. Barrette,et al.  Mining for regulatory programs in the cancer transcriptome , 2005, Nature Genetics.

[10]  S. Ghosh,et al.  Regulating Inducible Transcription Through Controlled Localization , 2005, Science's STKE.

[11]  Yudong D. He,et al.  A cell proliferation signature is a marker of extremely poor outcome in a subpopulation of breast cancer patients. , 2005, Cancer research.

[12]  Martha R. Stampfer,et al.  Chromatin Inactivation Precedes De Novo DNA Methylation during the Progressive Epigenetic Silencing of the RASSF1A Promoter , 2005, Molecular and Cellular Biology.

[13]  S. Byers,et al.  Oncogene-induced basement membrane invasiveness in human mammary epithelial cells , 1994, Clinical & Experimental Metastasis.

[14]  Stanley N Cohen,et al.  Disparate effects of telomere attrition on gene expression during replicative senescence of human mammary epithelial cells cultured under different conditions , 2004, Oncogene.

[15]  B. Henderson,et al.  BARD1 regulates BRCA1 apoptotic function by a mechanism involving nuclear retention. , 2004, Experimental cell research.

[16]  K. Chin,et al.  In situ analyses of genome instability in breast cancer , 2004, Nature Genetics.

[17]  Jill P. Mesirov,et al.  GeneCluster 2.0: an advanced toolset for bioarray analysis , 2004, Bioinform..

[18]  S. Hankinson,et al.  Insulin-like growth factors and neoplasia , 2004, Nature Reviews Cancer.

[19]  E. Liu,et al.  BRCA1 Interacts with and Is Required for Paclitaxel-Induced Activation of Mitogen-Activated Protein Kinase Kinase Kinase 3 , 2004, Cancer Research.

[20]  David Botstein,et al.  Different gene expression patterns in invasive lobular and ductal carcinomas of the breast. , 2004, Molecular biology of the cell.

[21]  E. Montgomery,et al.  Telomere Length Abnormalities Occur Early in the Initiation of Epithelial Carcinogenesis , 2004, Clinical Cancer Research.

[22]  T. Tuttle,et al.  Trends in the treatment of ductal carcinoma in situ of the breast. , 2004, Journal of the National Cancer Institute.

[23]  Z. Weng,et al.  Detection of functional DNA motifs via statistical over-representation. , 2004, Nucleic acids research.

[24]  Cynthia A Afshari,et al.  Histologically normal human mammary epithelia with silenced p16(INK4a) overexpress COX-2, promoting a premalignant program. , 2004, Cancer cell.

[25]  N. Bundred,et al.  COX-2 expression is associated with an aggressive phenotype in ductal carcinoma in situ , 2004, British Journal of Cancer.

[26]  P. Yaswen,et al.  Loss of p53 function accelerates acquisition of telomerase activity in indefinite lifespan human mammary epithelial cell lines , 2003, Oncogene.

[27]  L. Murphy,et al.  Psoriasin (S100A7) expression is associated with poor outcome in estrogen receptor-negative invasive breast cancer. , 2003, Clinical cancer research : an official journal of the American Association for Cancer Research.

[28]  P. Yaswen,et al.  Human epithelial cell immortalization as a step in carcinogenesis. , 2003, Cancer letters.

[29]  M. West,et al.  Gene expression predictors of breast cancer outcomes , 2003, The Lancet.

[30]  A. Seth,et al.  Gene expression profiling of ductal carcinomas in situ and invasive breast tumors. , 2003, Anticancer research.

[31]  Manel Esteller,et al.  Methylation of p16(INK4a) promoters occurs in vivo in histologically normal human mammary epithelia. , 2003, Cancer research.

[32]  Kornelia Polyak,et al.  Molecular markers in ductal carcinoma in situ of the breast. , 2003, Molecular cancer research : MCR.

[33]  E. Wingender,et al.  MATCH: A tool for searching transcription factor binding sites in DNA sequences. , 2003, Nucleic acids research.

[34]  K. Chew,et al.  Methylation of p 16 INK 4 a Promoters Occurs in Vivo in Histologically Normal Human Mammary Epithelia 1 , 2003 .

[35]  D. Slonim From patterns to pathways: gene expression data analysis comes of age , 2002, Nature Genetics.

[36]  Jaejoon Won,et al.  Sp1 and Sp3 Recruit Histone Deacetylase to Repress Transcription of Human Telomerase Reverse Transcriptase (hTERT) Promoter in Normal Human Somatic Cells* , 2002, The Journal of Biological Chemistry.

[37]  John D. Watson,et al.  Identifying Genes Regulated in a Myc-dependent Manner* , 2002, The Journal of Biological Chemistry.

[38]  Jayanta Debnath,et al.  The Role of Apoptosis in Creating and Maintaining Luminal Space within Normal and Oncogene-Expressing Mammary Acini , 2002, Cell.

[39]  D. Haber,et al.  BRCA1 Regulates the Interferon γ-mediated Apoptotic Response* , 2002, The Journal of Biological Chemistry.

[40]  H. Avraham,et al.  A Novel Tricomplex of BRCA1, Nmi, and c-Myc Inhibits c-Myc-induced Human Telomerase Reverse Transcriptase Gene (hTERT) Promoter Activity in Breast Cancer* , 2002, The Journal of Biological Chemistry.

[41]  M. King,et al.  BRCA1 transcriptionally regulates genes involved in breast tumorigenesis , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[42]  T. Volkert,et al.  E2F integrates cell cycle progression with DNA repair, replication, and G(2)/M checkpoints. , 2002, Genes & development.

[43]  D. Haber,et al.  BRCA1 regulates the interferon gamma-mediated apoptotic response. , 2002, The Journal of biological chemistry.

[44]  Alexander E. Kel,et al.  MATCHTM: a tool for searching transcription factor binding sites in DNA sequences , 2003, Nucleic Acids Res..

[45]  D. Slonim,et al.  Evaluation of normalization procedures for oligonucleotide array data based on spiked cRNA controls , 2001, Genome Biology.

[46]  R. Tibshirani,et al.  Gene expression patterns of breast carcinomas distinguish tumor subclasses with clinical implications , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[47]  R. Schreiber,et al.  Overexpression of IRF9 confers resistance to antimicrotubule agents in breast cancer cells. , 2001, Cancer research.

[48]  M. Byrne,et al.  Preparation of mRNA for Expression Monitoring , 2001, Current protocols in neuroscience.

[49]  R. Donato,et al.  S100: a multigenic family of calcium-modulated proteins of the EF-hand type with intracellular and extracellular functional roles. , 2001, The international journal of biochemistry & cell biology.

[50]  P. Yaswen,et al.  Expression of the telomerase catalytic subunit, hTERT, induces resistance to transforming growth factor β growth inhibition in p16INK4A(−) human mammary epithelial cells , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[51]  J W Gray,et al.  The ZNF217 gene amplified in breast cancers promotes immortalization of human mammary epithelial cells. , 2001, Cancer research.

[52]  F. Christians,et al.  E2Fs regulate the expression of genes involved in differentiation, development, proliferation, and apoptosis. , 2001, Genes & development.

[53]  Thea D. Tlsty,et al.  Normal human mammary epithelial cells spontaneously escape senescence and acquire genomic changes , 2001, Nature.

[54]  P. J. Welch,et al.  Identification of Id4 as a regulator of BRCA1 expression by using a ribozyme-library-based inverse genomics approach. , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[55]  Y. Cong,et al.  Histone Deacetylation Is Involved in the Transcriptional Repression of hTERT in Normal Human Cells* , 2000, The Journal of Biological Chemistry.

[56]  A. Iavarone,et al.  Id2 is a retinoblastoma protein target and mediates signalling by Myc oncoproteins , 2000, Nature.

[57]  T. Rohan,et al.  Role of the insulin-like growth factor family in cancer development and progression. , 2000, Journal of the National Cancer Institute.

[58]  Christian A. Rees,et al.  Molecular portraits of human breast tumours , 2000, Nature.

[59]  C. Deng,et al.  Roles of BRCA1 and its interacting proteins , 2000, BioEssays : news and reviews in molecular, cellular and developmental biology.

[60]  E. Lander,et al.  Expression analysis with oligonucleotide microarrays reveals that MYC regulates genes involved in growth, cell cycle, signaling, and adhesion. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[61]  M. Yutsudo,et al.  Sp1 cooperates with c-Myc to activate transcription of the human telomerase reverse transcriptase gene (hTERT). , 2000, Nucleic acids research.

[62]  J. Mesirov,et al.  Molecular classification of cancer: class discovery and class prediction by gene expression monitoring. , 1999, Science.

[63]  P. Yaswen,et al.  Viral oncogenes accelerate conversion to immortality of cultured conditionally immortal human mammary epithelial cells , 1999, Oncogene.

[64]  B. Williams,et al.  Identification of genes differentially regulated by interferon α, β, or γ using oligonucleotide arrays , 1998 .

[65]  D. Botstein,et al.  Cluster analysis and display of genome-wide expression patterns. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[66]  A. Brenner,et al.  Increased p16 expression with first senescence arrest in human mammary epithelial cells and extended growth capacity with p16 inactivation , 1998, Oncogene.

[67]  J. Herman,et al.  Alterations in DNA methylation: a fundamental aspect of neoplasia. , 1998, Advances in cancer research.

[68]  B. Williams,et al.  Identification of genes differentially regulated by interferon alpha, beta, or gamma using oligonucleotide arrays. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[69]  P. Yaswen,et al.  Gradual phenotypic conversion associated with immortalization of cultured human mammary epithelial cells. , 1997, Molecular biology of the cell.

[70]  F. Miller,et al.  Transforming and oncogenic potential of activated c-Ha-ras in three immortalized human breast epithelial cell lines. , 1997, Anticancer research.

[71]  R. Weinberg,et al.  hEST2, the Putative Human Telomerase Catalytic Subunit Gene, Is Up-Regulated in Tumor Cells and during Immortalization , 1997, Cell.

[72]  J. Geradts,et al.  High frequency of aberrant p16(INK4A) expression in human breast cancer. , 1996, The American journal of pathology.

[73]  M. Stampfer,et al.  Insulin receptor overexpression in 184B5 human mammary epithelial cells induces a ligand‐dependent transformed phenotype , 1995, Journal of cellular biochemistry.

[74]  H. Soule,et al.  Characterization of epithelial phenotypes in mortal and immortal human breast cells , 1992, International journal of cancer.

[75]  M. Kraus,et al.  Oncogenic potential of erbB-2 in human mammary epithelial cells. , 1991, Oncogene.

[76]  F. McCormick,et al.  Transformation of human mammary epithelial cells by oncogenic retroviruses. , 1988, Cancer research.

[77]  M. Stampfer,et al.  Induction of transformation and continuous cell lines from normal human mammary epithelial cells after exposure to benzo[a]pyrene. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[78]  S. L. Hammond,et al.  Serum-free growth of human mammary epithelial cells: rapid clonal growth in defined medium and extended serial passage with pituitary extract. , 1984, Proceedings of the National Academy of Sciences of the United States of America.