Lipopolysaccharide Mediates Endothelial Apoptosis by a FADD-dependent Pathway*
暂无分享,去创建一个
A. Karsan | P. Chaudhary | J. Harlan | L Hood | F. Wong | A Karsan | J M Harlan | F Wong | P M Chaudhary | K B Choi | Leroy Hood | K. Choi | Fred W. S. Wong
[1] A. Karsan,et al. Fibroblast growth factor-2 inhibits endothelial cell apoptosis by Bcl-2-dependent and independent mechanisms. , 1997, The American journal of pathology.
[2] M. Ueffing,et al. Critical involvement of transmembrane tumor necrosis factor-alpha in endothelial programmed cell death mediated by ionizing radiation and bacterial endotoxin. , 1995, Blood.
[3] S. Nagata,et al. The Fas death factor , 1995, Science.
[4] R. Thieringer,et al. CD14 enhances cellular responses to endotoxin without imparting ligand-specific recognition. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[5] C. Raetz,et al. Biochemistry of endotoxins. , 1990, Annual review of biochemistry.
[6] Matthias Mann,et al. FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex , 1996, Cell.
[7] S. Wright,et al. Mice Genetically Hyporesponsive to Lipopolysaccharide (LPS) Exhibit a Defect in Endocytic Uptake of LPS and Ceramide , 1997, The Journal of experimental medicine.
[8] M. Cybulsky,et al. Acute inflammation and microthrombosis induced by endotoxin, interleukin-1, and tumor necrosis factor and their implication in gram-negative infection. , 1988, Laboratory investigation; a journal of technical methods and pathology.
[9] D. Goeddel,et al. The TNF receptor 1-associated protein TRADD signals cell death and NF-κB activation , 1995, Cell.
[10] Miller Ad,et al. Improved Retroviral Vectors for Gene Transfer and Expression , 1989 .
[11] V. Dixit,et al. Fas-associated Death Domain Protein Interleukin-1β-converting Enzyme 2 (FLICE2), an ICE/Ced-3 Homologue, Is Proximally Involved in CD95- and p55-mediated Death Signaling* , 1997, The Journal of Biological Chemistry.
[12] D. Wallach,et al. Cell death induction by TNF: a matter of self control. , 1997, Trends in biochemical sciences.
[13] R. Swerlick,et al. HMEC-1: establishment of an immortalized human microvascular endothelial cell line. , 1992, The Journal of investigative dermatology.
[14] D. Goeddel,et al. FADD: essential for embryo development and signaling from some, but not all, inducers of apoptosis. , 1998, Science.
[15] N. Reiner,et al. Bacterial lipopolysaccharide induces the association and coordinate activation of p53/56lyn and phosphatidylinositol 3-kinase in human monocytes. , 1996, Journal of immunology.
[16] R. Cotran,et al. The role of endothelial cells in inflammation. , 1990, Transplantation.
[17] D. Golenbock,et al. CD11c/CD18, a transmembrane signaling receptor for lipopolysaccharide , 1995, The Journal of experimental medicine.
[18] N. Davidson,et al. Specific proteolytic cleavage of poly(ADP-ribose) polymerase: an early marker of chemotherapy-induced apoptosis. , 1993, Cancer research.
[19] David Wallach,et al. Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death , 1996, Cell.
[20] R. Ulevitch,et al. Lipopolysaccharide activation of human endothelial and epithelial cells is mediated by lipopolysaccharide-binding protein and soluble CD14. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[21] B. Aggarwal,et al. Early events in TNF signaling: a story of associations and dissociations , 1997, Journal of leukocyte biology.
[22] Vishva M. Dixit,et al. RAIDD is a new 'death' adaptor molecule , 1997, Nature.
[23] T. Pohlman,et al. Human endothelial cell response to lipopolysaccharide, interleukin-1, and tumor necrosis factor is regulated by protein synthesis. , 1989, Cellular immunology.
[24] G. Evan,et al. A License to Kill , 1996, Cell.
[25] J. Minna,et al. Evaluation of a tetrazolium-based semiautomated colorimetric assay: assessment of radiosensitivity. , 1987, Cancer research.
[26] N. Lalwani,et al. Fas ligation triggers apoptosis in macrophages but not endothelial cells , 1994, European journal of immunology.
[27] M. Arnaout,et al. Outside-in signaling by lipopolysaccharide through a tailless integrin. , 1997, Journal of immunology.
[28] M. Read,et al. Cell-free pool of CD14 mediates activation of transcription factor NF-kappa B by lipopolysaccharide in human endothelial cells. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[29] R. Ulevitch,et al. Recognition of endotoxin by cells leading to transmembrane signaling. , 1994, Current opinion in immunology.
[30] S. Goyert,et al. Endotoxin-mediated endothelial cell injury and activation: role of soluble CD14 , 1993 .
[31] A. Karsan,et al. Endothelial Cell Death Induced by Tumor Necrosis Factor-α Is Inhibited by the Bcl-2 Family Member, A1* , 1996, The Journal of Biological Chemistry.
[32] D. Durden,et al. Lipopolysaccharide stimulates the tyrosine phosphorylation of mitogen-activated protein kinases p44, p42, and p41 in vascular endothelial cells in a soluble CD14-dependent manner. Role of protein tyrosine phosphorylation in lipopolysaccharide-induced stimulation of endothelial cells. , 1995, Journal of immunology.
[33] J. Randolph,et al. Bacterial lipopolysaccharide has structural similarity to ceramide and stimulates ceramide-activated protein kinase in myeloid cells. , 1994, The Journal of biological chemistry.
[34] B. Meyrick,et al. In vitro effects of endotoxin on bovine and sheep lung microvascular and pulmonary artery endothelial cells , 1989, Journal of cellular physiology.
[35] A. Karsan,et al. Tumor necrosis factor and endothelial cell death. , 1998, Trends in cardiovascular medicine.
[36] D. Goeddel,et al. A death-domain-containing receptor that mediates apoptosis , 1996, Nature.
[37] S. Murota,et al. Age related decline in cytokine induced nitric oxide synthase activation and apoptosis in cultured endothelial cells: minimal involvement of nitric oxide in the apoptosis , 1995, Mechanisms of Ageing and Development.
[38] A. Chinnaiyan,et al. Signal Transduction by DR3, a Death Domain-Containing Receptor Related to TNFR-1 and CD95 , 1996, Science.
[39] A. Chinnaiyan,et al. FADD/MORT1 Is a Common Mediator of CD95 (Fas/APO-1) and Tumor Necrosis Factor Receptor-induced Apoptosis (*) , 1996, The Journal of Biological Chemistry.
[40] K. Bauer,et al. Activation of apoptosis by Apo-2 ligand is independent of FADD but blocked by CrmA , 1996, Current Biology.