Mechanisms underlying mutually exclusive expression of virulence genes by malaria parasites

[1]  M. Duraisingh,et al.  Heterochromatin‐mediated control of virulence gene expression , 2006, Molecular microbiology.

[2]  E. Berdougo,et al.  Strict Pairing of var Promoters and Introns Is Required for var Gene Silencing in the Malaria Parasite Plasmodium falciparum* , 2006, Journal of Biological Chemistry.

[3]  M. Frank,et al.  Mutually Exclusive Expression of Virulence Genes by Malaria Parasites Is Regulated Independently of Antigen Production , 2006, PLoS pathogens.

[4]  A. Cowman,et al.  A var gene promoter controls allelic exclusion of virulence genes in Plasmodium falciparum malaria , 2006, Nature.

[5]  S. Ralph,et al.  Antigenic variation in Plasmodium falciparum is associated with movement of var loci between subnuclear locations. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[6]  Manoj T. Duraisingh,et al.  Heterochromatin Silencing and Locus Repositioning Linked to Regulation of Virulence Genes in Plasmodium falciparum , 2005, Cell.

[7]  A. Corcoran Immunoglobulin locus silencing and allelic exclusion. , 2005, Seminars in immunology.

[8]  T. Wellems,et al.  A Silenced Plasmodium falciparum var Promoter Can Be Activated In Vivo through Spontaneous Deletion of a Silencing Element in the Intron , 2005, Eukaryotic Cell.

[9]  Hitoshi Sakano,et al.  One neuron-one receptor rule in the mouse olfactory system. , 2004, Trends in genetics : TIG.

[10]  D. Reinberg,et al.  Epigenetic Dynamics of Imprinted X Inactivation During Early Mouse Development , 2004, Science.

[11]  T. Wellems,et al.  Plasmodium falciparum var Genes Are Regulated by Two Regions with Separate Promoters, One Upstream of the Coding Region and a Second within the Intron* , 2003, Journal of Biological Chemistry.

[12]  Keith Gull,et al.  A pol I transcriptional body associated with VSG mono-allelic expression in Trypanosoma brucei , 2001, Nature.

[13]  T. Wellems,et al.  Malaria: Cooperative silencing elements in var genes , 2001, Nature.

[14]  Jeannie T. Lee,et al.  A functional role for Tsix transcription in blocking Xist RNA accumulation but not in X-chromosome choice , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[15]  T. Wellems,et al.  Transformation of malaria parasites by the spontaneous uptake and expression of DNA from human erythrocytes. , 2001, Nucleic acids research.

[16]  Thomas E. Wellems,et al.  Frequent ectopic recombination of virulence factor genes in telomeric chromosome clusters of P. falciparum , 2000, Nature.

[17]  P. Borst,et al.  Subnuclear localization of the active variant surface glycoprotein gene expression site in Trypanosoma brucei. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[18]  A. Scherf,et al.  Antigenic variation in malaria: in situ switching, relaxed and mutually exclusive transcription of var genes during intra‐erythrocytic development in Plasmodium falciparum , 1998, The EMBO journal.

[19]  W. Trager,et al.  Human malaria parasites in continuous culture. , 1976, Science.

[20]  J. Thompson In situ detection of RNA in blood- and mosquito-stage malaria parasites. , 2002, Methods in molecular medicine.

[21]  S. Kyes,et al.  Antigenic variation at the infected red cell surface in malaria. , 2001, Annual review of microbiology.

[22]  R. A. Drewell,et al.  Epigenetic reprogramming of the genome--from the germ line to the embryo and back again. , 2001, The International journal of developmental biology.

[23]  N. Bartelt,et al.  Nanostructures: Self-assembled domain patterns , 2001, Nature.