Glycated collagen cross-linking alters cardiac mechanics in volume-overload hypertrophy.
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Andrew D McCulloch | A. McCulloch | J. Omens | Keith L Herrmann | Jeffrey H Omens | Keith L. Herrmann | K. Herrmann
[1] R. Dunn. Regional blood flow and metabolite levels in the left ventricular free wall and septum during aortic insufficiency: implications for the development of asymmetric septal hypertrophy. , 1986, Journal of the American College of Cardiology.
[2] M. Huijberts,et al. Breakers of advanced glycation end products restore large artery properties in experimental diabetes. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[3] J. Covell,et al. Collagen characterization in volume-overload- and pressure-overload-induced cardiac hypertrophy in minipigs. , 1993, The American journal of physiology.
[4] F. Plum. Handbook of Physiology. , 1960 .
[5] A. Cerami,et al. An advanced glycation endproduct cross-link breaker can reverse age-related increases in myocardial stiffness. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[6] G. Bianchi,et al. Effect of propionyl-L-carnitine on the mechanics of right and left papillary muscles from volume-overloaded rat hearts. , 1996, Journal of cardiovascular pharmacology.
[7] N. Simionescu,et al. The Cardiovascular System , 1983 .
[8] J W Covell,et al. Dependence of ventricular distensibility on filling of the opposite ventricle. , 1967, The American journal of physiology.
[9] C. Elmets,et al. Relation between complications of type I diabetes mellitus and collagen-linked fluorescence. , 1986, The New England journal of medicine.
[10] S. Skinner. Rapid method for the purification of the elastin cross-links, desmosine and isodesmosine. , 1982, Journal of chromatography.
[11] K. Tracey,et al. Advanced glycosylation products quench nitric oxide and mediate defective endothelium-dependent vasodilatation in experimental diabetes. , 1991, The Journal of clinical investigation.
[12] T. Regan,et al. Effects of Metformin on Collagen Glycation and Diastolic Dysfunction in Diabetic Myocardium , 1998, Journal of cardiovascular pharmacology and therapeutics.
[13] G. Norton,et al. Aminoguanidine prevents the decreased myocardial compliance produced by streptozotocin-induced diabetes mellitus in rats. , 1996, Circulation.
[14] 難波 隆志. Regulation of fibrillar collagen gene expression and protein accumulation in volume-overloaded cardiac hypertrophy , 2002 .
[15] M Duriez,et al. Aminoguanidine prevents age-related arterial stiffening and cardiac hypertrophy. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[16] Y. Obeng,et al. Nuclear‐magnetic‐resonance characterization of doped SiO2 films used in integrated circuits , 1995 .
[17] W. Sibbald,et al. Right ventricular function in acute disease states: Pathophysiologic considerations , 1983, Critical care medicine.
[18] F. Spinale,et al. Cellular and extracellular remodeling with the development and recovery from tachycardia-induced cardiomyopathy: changes in fibrillar collagen, myocyte adhesion capacity and proteoglycans. , 1996, Journal of molecular and cellular cardiology.
[19] H. Oxlund,et al. Inhibition of cross-links in collagen is associated with reduced stiffness of the aorta in young rats. , 1998, Atherosclerosis.
[20] J. F. Woessner,et al. The determination of hydroxyproline in tissue and protein samples containing small proportions of this imino acid. , 1961, Archives of biochemistry and biophysics.
[21] G. Norton,et al. Exercise-induced cardiac hypertrophy is associated with an increased myocardial compliance. , 1995, Journal of applied physiology.
[22] W Grossman,et al. Cardiac hypertrophy: useful adaptation or pathologic process? , 1980, The American journal of medicine.
[23] J. Covell,et al. Increase in Cross‐Linking of Type I and Type III Collagens Associated With Volume‐Overload Hypertrophy , 1988, Circulation research.
[24] A. Cerami,et al. Protein glycation, diabetes, and aging. , 2001, Recent progress in hormone research.
[25] D. Farrar,et al. Pacing‐Induced Dilated Cardiomyopathy Increases Left‐to‐RightVentricular Systolic Interaction , 1993, Circulation.
[26] M. Zile,et al. Inhibition of collagen cross-linking: effects on fibrillar collagen and ventricular diastolic function. , 1995, The American journal of physiology.
[27] R. Bucala,et al. Prevention of cardiovascular and renal pathology of aging by the advanced glycation inhibitor aminoguanidine. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[28] D. Gibson,et al. Relation of regional echo amplitude to left ventricular function and the electrocardiogram in left ventricular hypertrophy. , 1984, British heart journal.
[29] A. Bailey,et al. Non-enzymic glycation of fibrous collagen: reaction products of glucose and ribose. , 1995, The Biochemical journal.
[30] G. Norton,et al. Myocardial stiffness is attributed to alterations in cross-linked collagen rather than total collagen or phenotypes in spontaneously hypertensive rats. , 1997, Circulation.
[31] J. Omens,et al. Mechanical regulation of myocardial growth during volume-overload hypertrophy in the rat. , 1997, The American journal of physiology.
[32] T. Regan,et al. Effects of glucose intolerance on myocardial function and collagen-linked glycation. , 1999, Diabetes.
[33] B. Nilsson. Biological effects of aminoguanidine: An update , 1999, Inflammation Research.
[34] A. McCulloch,et al. Contribution of collagen matrix to passive left ventricular mechanics in isolated rat hearts. , 1994, The American journal of physiology.
[35] A. Cerami,et al. Aminoguanidine prevents diabetes-induced arterial wall protein cross-linking. , 1986, Science.
[36] T. Koob,et al. Quantitation of hydroxypyridinium crosslinks in collagen by high-performance liquid chromatography. , 1984, Analytical biochemistry.