Novel variant isoform of G‐CSF receptor involved in induction of proliferation of FDCP‐2 cells: Relevance to the pathogenesis of myelodysplastic syndrome
暂无分享,去创建一个
Yasuo Ikeda | Hideaki Nakajima | Yoshitaka Miyakawa | Hiromichi Matsushita | M. Kizaki | Y. Miyakawa | Y. Ikeda | H. Nakajima | H. Matsushita | Norihiro Awaya | H. Uchida | Hideo Uchida | Kentaro Kinjo | Masahiro Kizaki | K. Kinjo | N. Awaya
[1] S. Nagata,et al. Growth and differentiation signals mediated by different regions in the cytoplasmic domain of granulocyte colony-stimulating factor receptor , 1993, Cell.
[2] A. Veerman,et al. Identification of a nonsense mutation in the granulocyte-colony-stimulating factor receptor in severe congenital neutropenia. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[3] G. Stark,et al. Jak1 plays an essential role for receptor phosphorylation and Stat activation in response to granulocyte colony-stimulating factor. , 1997, Blood.
[4] G. Wagemaker,et al. Impaired response of myelodysplastic marrow progenitors to stimulation with recombinant haemopoietic growth factors. , 1991, Leukemia.
[5] A. Williams,et al. The myelodysplastic syndrome: analysis of laboratory characteristics in relation to the FAB classification , 1985, British journal of haematology.
[6] L. Hoefsloot,et al. A point mutation in the granulocyte colony-stimulating factor receptor (G-CSF-R) gene in a case of acute myeloid leukemia results in the overexpression of a novel G-CSF-R isoform. , 1995, Blood.
[7] Robert,et al. Rapid Activation of the STAT 3 Transcription Factor by Granulocyte Colony-Stimulating Factor , 2002 .
[8] L. Gillespie,et al. Cloning of a Fibroblast Growth Factor Receptor 1 Splice Variant from Xenopus Embryos That Lacks a Protein Kinase C Site Important for the Regulation of Receptor Activity (*) , 1995, The Journal of Biological Chemistry.
[9] P. Lamb,et al. Rapid activation of the STAT3 transcription factor by granulocyte colony-stimulating factor , 1994 .
[10] E. Scolnick,et al. Growth of factor-dependent hemopoietic precursor cell lines , 1980, The Journal of experimental medicine.
[11] J. Adamson,et al. Pancytopenia as a clonal disorder of a multipotent hematopoietic stem cell. , 1984, The Journal of clinical investigation.
[12] K. Welte,et al. Mutations in the gene for the granulocyte colony-stimulating-factor receptor in patients with acute myeloid leukemia preceded by severe congenital neutropenia. , 1995, The New England journal of medicine.
[13] M. Ballmaier,et al. Implications of Mutations in Hematopoietic Growth Factor Receptor Genes in Congenital Cytopenias , 2001, Annals of the New York Academy of Sciences.
[14] J. Palmblad,et al. Mutations in the granulocyte colony-stimulating factor receptor gene in patients with severe congenital neutropenia , 1997, Leukemia.
[15] P. Lamb,et al. Multiple signaling pathways induced by granulocyte colony-stimulating factor involving activation of JAKs, STAT5, and/or STAT3 are required for regulation of three distinct classes of immediate early genes. , 1996, Blood.
[16] G. Demetri,et al. Granulocyte colony-stimulating factor and its receptor. , 1991, Blood.
[17] S. Nagata,et al. Functional domains of the granulocyte colony‐stimulating factor receptor. , 1991, The EMBO journal.
[18] Koeffler,et al. Human preleukemia. , 1980, Annals of internal medicine.
[19] D. Linch,et al. Analysis of granulocyte colony stimulating factor receptor isoforms, polymorphisms and mutations in normal haemopoietic cells and acute myeloid leukaemia blasts , 1996, British journal of haematology.
[20] S. Knuutila,et al. Erythroid and granulocyte-macrophage colony formation in myelodysplastic syndromes. , 2009, Scandinavian journal of haematology.
[21] A. Yoshikawa,et al. Distinct signal transduction through the tyrosine‐containing domains of the granulocyte colony‐stimulating factor receptor. , 1995, The EMBO journal.
[22] M. Pfeilstöcker,et al. Myelodysplastic syndromes, from French-American-British to World Health Organization: comparison of classifications on 431 unselected patients from a single institution. , 2001, Blood.
[23] I. Touw,et al. Rarity of dominant-negative mutations of the G-CSF receptor in patients with blast crisis of chronic myeloid leukemia or de novo acute leukemia , 1997, Leukemia.
[24] F. Dong,et al. Distinct cytoplasmic regions of the human granulocyte colony-stimulating factor receptor involved in induction of proliferation and maturation. , 1993, Molecular and cellular biology.
[25] P. Greenberg. The smoldering myeloid leukemic states: clinical and biologic features. , 1983, Blood.
[26] L. Shih,et al. Diagnostic and prognostic values of in vitro culture growth patterns of marrow granulocyte-macrophage progenitors in patients with myelodysplastic syndrome. , 1991, Leukemia.
[27] P. Greenberg,et al. Impact of marrow cytogenetics and morphology on in vitro hematopoiesis in the myelodysplastic syndromes: comparison between recombinant human granulocyte colony-stimulating factor (CSF) and granulocyte-monocyte CSF. , 1990, Blood.
[28] A R Dunn,et al. Mice lacking granulocyte colony-stimulating factor have chronic neutropenia, granulocyte and macrophage progenitor cell deficiency, and impaired neutrophil mobilization. , 1994, Blood.
[29] S. Nagata,et al. Three different mRNAs encoding human granulocyte colony-stimulating factor receptor. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[30] Y. Matsuzawa,et al. Constitutive activation of c-kit in FMA3 murine mastocytoma cells caused by deletion of seven amino acids at the juxtamembrane domain. , 1996, Blood.
[31] G. Stark,et al. Jak 1 Plays an Essential Role for Receptor Phosphorylation and Stat Activation in Response to Granulocyte Colony-Stimulating Factor , 1997 .
[32] D. Tweardy,et al. Increased expression of the differentiation-defective granulocyte colony-stimulating factor receptor mRNA isoform in acute myelogenous leukemia , 1998, Leukemia.
[33] P. Sonneveld,et al. The effects of interleukin-3, GM-CSF, and G-CSF on the growth kinetics of colony-forming cells in myelodysplastic syndromes. , 1990, Leukemia.
[34] M. Yamaguchi,et al. Proliferation and differentiation of myelodysplastic CD34+ cells in serum‐free medium: response to individual colony‐stimulating factors , 1993, British journal of haematology.
[35] P. Greenberg,et al. Expression of EVI1 in myelodysplastic syndromes and other hematologic malignancies without 3q26 translocations. , 1994, Blood.
[36] S. Knuutila,et al. Colony formation by megakaryocyte progenitors in myelodysplatic syndromes , 1989, European journal of haematology.
[37] A. Oates,et al. Tyrosine kinase JAK1 is associated with the granulocyte-colony-stimulating factor receptor and both become tyrosine-phosphorylated after receptor activation. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[38] M. Green,et al. Biochemical mechanisms of constitutive and regulated pre-mRNA splicing. , 1991, Annual review of cell biology.
[39] P. Greenberg,et al. Expression of EVIl in Myelodysplastic Syndromes and Other Hematologic Malignancies Without 3 q 26 Translocations , 2002 .
[40] R. Ohno,et al. Retrovirus-mediated gene transfer of granulocyte colony-stimulating factor receptor (G-CSFR) cDNA into MDS cells and induction of their differentiation by G-CSF. , 2000, Cytokines, cellular & molecular therapy.
[41] Y. Matsuzawa,et al. Neoplastic transformation of normal hematopoietic cells by constitutively activating mutations of c-kit receptor tyrosine kinase. , 1996, Blood.
[42] K. Welte,et al. Clinical relevance of point mutations in the cytoplasmic domain of the granulocyte colony-stimulating factor receptor gene in patients with severe congenital neutropenia. , 1997, Blood.
[43] S. Nagata,et al. Purification and characterization of the receptor for murine granulocyte colony-stimulating factor. , 1990, The Journal of biological chemistry.
[44] P. Fialkow,et al. Evidence for a multistep pathogenesis of a myelodysplastic syndrome. , 1984, Blood.