Myocardial contrast echocardiography versus dobutamine echocardiography for predicting functional recovery after acute myocardial infarction treated with primary coronary angioplasty.

[1]  S. Kaul,et al.  There may be more to myocardial viability than meets the eye. , 1995, Circulation.

[2]  E. Zajac,et al.  Ability of the no-reflow phenomenon during an acute myocardial infarction to predict left ventricular dysfunction at one-month follow-up. , 1995, The American journal of cardiology.

[3]  V. Dilsizian,et al.  Relation between thallium uptake and contractile response to dobutamine. Implications regarding myocardial viability in patients with chronic coronary artery disease and left ventricular dysfunction. , 1995, Circulation.

[4]  M. Hori,et al.  Temporal changes in myocardial perfusion patterns in patients with reperfused anterior wall myocardial infarction. Their relation to myocardial viability. , 1995, Circulation.

[5]  S. Kaul,et al.  Identification of viable myocardium with contrast echocardiography in patients with poor left ventricular systolic function caused by recent or remote myocardial infarction. , 1995, The American journal of cardiology.

[6]  M. Hori,et al.  Myocardial salvage: its assessment and prediction by the analysis of serial myocardial contrast echocardiograms in patients with acute myocardial infarction. , 1994, American heart journal.

[7]  M. Hori,et al.  Dobutamine stress echocardiography predicts reversible dysfunction and quantitates the extent of irreversibly damaged myocardium after reperfusion of anterior myocardial infarction. , 1994, Journal of the American College of Cardiology.

[8]  S. Kaul,et al.  Dobutamine Echocardiography for Determining the Extent of Myocardial Salvage After Reperfusion: An Experimental Evaluation , 1994, Circulation.

[9]  L. Tavazzi,et al.  Spontaneous Delayed Recovery of Perfusion and Contraction After the First 5 Weeks After Anterior Infarction: Evidence for the Presence of Hibernating Myocardium in the Infarcted Area , 1994, Circulation.

[10]  U Sechtem,et al.  Assessment of viable myocardium by dobutamine transesophageal echocardiography and comparison with fluorine-18 fluorodeoxyglucose positron emission tomography. , 1994, Journal of the American College of Cardiology.

[11]  C. Autore,et al.  Influence of residual perfusion within the infarct zone on the natural history of left ventricular dysfunction after acute myocardial infarction: a myocardial contrast echocardiographic study. , 1994, Journal of the American College of Cardiology.

[12]  S. Kaul,et al.  Microvascular integrity indicates myocellular viability in patients with recent myocardial infarction. New insights using myocardial contrast echocardiography. , 1994, Circulation.

[13]  S. Kaul,et al.  Characterization of Spatial Patterns of Flow Within the Reperfused Myocardium by Myocardial Contrast Echocardiography Implications in Determining Extent of Myocardial Salvage , 1993, Circulation.

[14]  Gusto Angiographic Investigators The effects of tissue plasminogen activator, streptokinase, or both on coronary-artery patency, ventricular function, and survival after acute myocardial infarction. , 1993, The New England journal of medicine.

[15]  H Feigenbaum,et al.  Low‐Dose Dobutamine Echocardiography Detects Reversible Dysfunction After Thrombolytic Therapy of Acute Myocardial Infarction , 1993, Circulation.

[16]  A. Kitabatake,et al.  Time Course of Functional Improvement in Stunned Myocardium in Risk Area in Patients With Reperfused Anterior Infarction , 1993, Circulation.

[17]  R O Bonow,et al.  Current Diagnostic Techniques of Assessing Myocardial Viabilit in Patients With Hibernating and Stunned Myocardium , 1993, Circulation.

[18]  S. Kaul,et al.  Functional Significance of Collateral Blood Flow in Patients With Recent Acute Myocardial Infarction: A Study Using Myocardial Contrast Echocardiography , 1992, Circulation.

[19]  A. Kitabatake,et al.  Lack of Myocardial Perfusion Immediately After Successful Thrombolysis: A Predictor of Poor Recovery of Left Ventricular Function in Anterior Myocardial Infarction , 1992, Circulation.

[20]  M. Gheorghiade,et al.  Low-dose dobutamine in patients with acute myocardial infarction identifies viable but not contractile myocardium and predicts the magnitude of improvement in wall motion abnormalities in response to coronary revascularization. , 1991, American heart journal.

[21]  E. Picano,et al.  Stress echocardiography and the human factor: the importance of being expert. , 1990, Journal of the American College of Cardiology.

[22]  P. Rigo,et al.  Identification of viable myocardium by echocardiography during dobutamine infusion in patients with myocardial infarction after thrombolytic therapy: comparison with positron emission tomography. , 1990, Journal of the American College of Cardiology.

[23]  N. Reichek,et al.  Recommendations for quantitation of the left ventricle by two-dimensional echocardiography. American Society of Echocardiography Committee on Standards, Subcommittee on Quantitation of Two-Dimensional Echocardiograms. , 1989, Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography.

[24]  R. Virmani,et al.  Reduction of reperfusion injury in the canine preparation by intracoronary adenosine: importance of the endothelium and the no-reflow phenomenon. , 1987, Circulation.

[25]  H. White,et al.  Effect of intravenous streptokinase on left ventricular function and early survival after acute myocardial infarction. , 1987, The New England journal of medicine.

[26]  M. Verani,et al.  Preservation of cardiac function by coronary thrombolysis during acute myocardial infarction: fact or myth? , 1987, Journal of the American College of Cardiology.

[27]  A. Weyman,et al.  Effects of selectively altering collateral driving pressure on regional perfusion and function in occluded coronary bed in the dog. , 1987, Circulation research.

[28]  S. Vatner,et al.  Effects of coronary artery reperfusion on regional myocardial blood flow and function in conscious baboons. , 1985, Circulation.

[29]  H. S. Mueller,et al.  The Thrombolysis in Myocardial Infarction (TIMI) trial. Phase I findings. , 1985, The New England journal of medicine.

[30]  E. Braunwald,et al.  Ultrastructural Evidence of Microvascular Damage and Myocardial Cell Injury After Coronary Artery Occlusion: Which Comes First? , 1980, Circulation.

[31]  C. Bloor,et al.  Regional redistribution of myocardial blood flow after coronary occlusion and reperfusion in the conscious dog. , 1978, The American journal of cardiology.

[32]  J. Greenfield,et al.  Local effects of acute cellular injury on regional myocardial blood flow. , 1976, The Journal of clinical investigation.

[33]  R. Helfant,et al.  Reversible Asynergy , 1976, Circulation.