Differences in inflammation, MMP activation and collagen damage account for gender difference in murine cardiac rupture following myocardial infarction.
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Lu Fang | Yean Leng Lim | A. Dart | Xiao-Ming Gao | Y. Lim | Xiaojun Du | H. Kiriazis | L. Fang | X. Moore | Helen Kiriazis | Anthony M Dart | Z. Ming | Xiao-Ming Gao | Xiao-Lei Moore | Yidan Su | Ziqiu Ming | Xiao-Jun Du | Yidan Su
[1] D R Boughner,et al. Role of Collagen in Acute Myocardial Infarct Expansion , 1991, Circulation.
[2] A. Takeshita,et al. Targeted deletion of MMP-2 attenuates early LV rupture and late remodeling after experimental myocardial infarction. , 2003, American journal of physiology. Heart and circulatory physiology.
[3] P. Libby,et al. Matrix metalloproteinase inhibition attenuates early left ventricular enlargement after experimental myocardial infarction in mice. , 1999, Circulation.
[4] J. Skurnick,et al. Cardiac Rupture in Acute Myocardial Infarction: A Reassessment , 2002, The American journal of forensic medicine and pathology.
[5] Leslie L. Clark,et al. Specific Temporal Profile of Matrix Metalloproteinase Release Occurs in Patients After Myocardial Infarction: Relation to Left Ventricular Remodeling , 2006, Circulation.
[6] Xiaojun Du,et al. Relaxin Reverses Cardiac and Renal Fibrosis in Spontaneously Hypertensive Rats , 2005, Hypertension.
[7] M. Cavasin,et al. Testosterone enhances early cardiac remodeling after myocardial infarction, causing rupture and degrading cardiac function. , 2006, American journal of physiology. Heart and circulatory physiology.
[8] M. Cavasin,et al. Gender differences in cardiac function during early remodeling after acute myocardial infarction in mice. , 2004, Life sciences.
[9] E. Antman,et al. Fatal cardiac rupture among patients treated with thrombolytic agents and adjunctive thrombin antagonists: observations from the Thrombolysis and Thrombin Inhibition in Myocardial Infarction 9 Study. , 1999, Journal of the American College of Cardiology.
[10] D. Stajer,et al. Neutrophils in human myocardial infarction with rupture of the free wall. , 2005, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.
[11] K. Nakao,et al. Overexpression of Brain Natriuretic Peptide Facilitates Neutrophil Infiltration and Cardiac Matrix Metalloproteinase-9 Expression After Acute Myocardial Infarction , 2004, Circulation.
[12] A. Maggi,et al. Regulation of the lipopolysaccharide signal transduction pathway by 17β-estradiol in macrophage cells , 2004, The Journal of Steroid Biochemistry and Molecular Biology.
[13] P. Libby,et al. Targeted deletion of matrix metalloproteinase-9 attenuates left ventricular enlargement and collagen accumulation after experimental myocardial infarction. , 2000, The Journal of clinical investigation.
[14] A. Dart,et al. Mouse model of post-infarct ventricular rupture: time course, strain- and gender-dependency, tensile strength, and histopathology. , 2005, Cardiovascular research.
[15] R. O'rourke,et al. Comparative Accuracy of Apical Biplane Cross‐sectional Echocardiography and Gated Equilibrium Radionuclide Angiography for Estimating Left Ventricular Size and Performance , 1981, Circulation.
[16] I. Godsland,et al. 17β-Oestradiol enhances release of matrix metalloproteinase-2 from human vascular smooth muscle cells , 1998 .
[17] S. Sankey,et al. Estrogen and testosterone have opposing effects on chronic cardiac remodeling and function in mice with myocardial infarction. , 2003, American journal of physiology. Heart and circulatory physiology.
[18] Merry Lindsey,et al. Matrix-Dependent Mechanism of Neutrophil-Mediated Release and Activation of Matrix Metalloproteinase 9 in Myocardial Ischemia/Reperfusion , 2001, Circulation.
[19] J. Hayakawa,et al. Estrogen and raloxifene inhibit the monocytic chemoattractant protein-1-induced migration of human monocytic cells via nongenomic estrogen receptor &agr; , 2006, Menopause.
[20] E. Antman,et al. Ventricular septal and free wall rupture complicating acute myocardial infarction: experience in the Multicenter Investigation of Limitation of Infarct Size. , 1989, American heart journal.
[21] R. Visse,et al. Matrix Metalloproteinases Regulation and Dysregulation in the Failing Heart , 2002 .
[22] David Handelsman,et al. Dihydrotestosterone promotes vascular cell adhesion molecule-1 expression in male human endothelial cells via a nuclear factor-kappaB-dependent pathway. , 2004, Endocrinology.
[23] Y. Okada,et al. Targeted deletion or pharmacological inhibition of MMP-2 prevents cardiac rupture after myocardial infarction in mice. , 2005, The Journal of clinical investigation.
[24] A. Luttun,et al. Inhibition of plasminogen activators or matrix metalloproteinases prevents cardiac rupture but impairs therapeutic angiogenesis and causes cardiac failure , 1999, Nature Medicine.
[25] R. M. Rubison,et al. A composite view of cardiac rupture in the United States National Registry of Myocardial Infarction. , 1996, Journal of the American College of Cardiology.
[26] M. Mori,et al. Suppression by 17beta-estradiol of monocyte adhesion to vascular endothelial cells is mediated by estrogen receptors. , 2004, Life sciences.
[27] M. Radomski,et al. Matrix metalloproteinase-2 contributes to ischemia-reperfusion injury in the heart. , 2000, Circulation.
[28] G. Jerums,et al. The breakdown of pre‐existing advanced glycation end products is associated with reduced renal fibrosis in experimental diabetes , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[29] A. Dart,et al. Serial echocardiographic assessment of left ventricular dimensions and function after myocardial infarction in mice. , 2000, Cardiovascular research.
[30] W. Roberts,et al. Out-of-hospital sudden death from left ventricular free wall rupture during acute myocardial infarction as the first and only manifestation of atherosclerotic coronary artery disease. , 1994, The American journal of cardiology.
[31] A. Dart,et al. Lower Risk of Postinfarct Rupture in Mouse Heart Overexpressing &bgr;2-Adrenergic Receptors: Importance of Collagen Content , 2002, Journal of cardiovascular pharmacology.
[32] Richard T. Lee,et al. Selective Matrix Metalloproteinase Inhibition Reduces Left Ventricular Remodeling but Does Not Inhibit Angiogenesis After Myocardial Infarction , 2002, Circulation.