Distinct mode of apoptosis induced by genotoxic agent etoposide and serum withdrawal in neuroblastoma cells.

[1]  K. Jellinger,et al.  Alzheimer Disease: DMA Fragmentation Indicates Increased Neuronal Vulnerability, but not Apoptosis , 1998, Journal of neuropathology and experimental neurology.

[2]  B. Pettmann,et al.  Neuronal Cell Death , 1998, Neuron.

[3]  D. Green,et al.  DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1. , 1998, Molecular cell.

[4]  L. Rubin,et al.  Phosphorylation of c-Jun Is Necessary for Apoptosis Induced by Survival Signal Withdrawal in Cerebellar Granule Neurons , 1998, The Journal of Neuroscience.

[5]  S. Nagata,et al.  A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD , 1998, Nature.

[6]  Full length article , 1997, Brain Research.

[7]  M. Malim,et al.  Suppression of tumor necrosis factor-induced cell death by inhibitor of apoptosis c-IAP2 is under NF-kappaB control. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[8]  M. Mattson,et al.  Activation of NF‐κB protects hippocampal neurons against oxidative stress‐induced apoptosis: Evidence for induction of manganese superoxide dismutase and suppression of peroxynitrite production and protein tyrosine nitration , 1997, Journal of neuroscience research.

[9]  M. Dragunow,et al.  Etoposide-induced PC12 cell death: apoptotic morphology without oligonucleosomal DNA fragmentation or dependency upon de novo protein synthesis. , 1997, Brain research. Molecular brain research.

[10]  T. Herdegen,et al.  The c-Jun transcription factor – bipotential mediator of neuronal death, survival and regeneration , 1997, Trends in Neurosciences.

[11]  A. Levine p53, the Cellular Gatekeeper for Growth and Division , 1997, Cell.

[12]  P. Walker,et al.  Identification of a novel 97 kDa endonuclease capable of internucleosomal DNA cleavage. , 1997, Biochemistry.

[13]  A. Novelli,et al.  Inhibition of protein phosphatases induces IGF‐1‐blocked neurotrophin‐insensitive neuronal apoptosis , 1996, FEBS letters.

[14]  A. Salminen,et al.  Changes associated with aging and replicative senescence in the regulation of transcription factor nuclear factor-kappa B. , 1996, The Biochemical journal.

[15]  I. Fewer,et al.  Strong c-Jun immunoreactivity is associated with apoptotic cell death in human tumors of the central nervous system , 1996, Neuroscience Letters.

[16]  S. Orrenius,et al.  CPP32/Apopain Is a Key Interleukin 1 Converting Enzyme-like Protease Involved in Fas-mediated Apoptosis (*) , 1996, The Journal of Biological Chemistry.

[17]  S. Orrenius,et al.  Constitutive nuclear NF kappa B/rel DNA-binding activity of rat thymocytes is increased by stimuli that promote apoptosis, but not inhibited by pyrrolidine dithiocarbamate. , 1995, The Biochemical journal.

[18]  L. Ghibelli,et al.  Non-oxidative loss of glutathione in apoptosis via GSH extrusion. , 1995, Biochemical and biophysical research communications.

[19]  S. Lipton,et al.  Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function , 1995, Neuron.

[20]  T. Cotter,et al.  Inhibition of apoptosis by antioxidants in the human HL-60 leukemia cell line. , 1995, Biochemical pharmacology.

[21]  V. Kagan,et al.  Antioxidant paradoxes of phenolic compounds: peroxyl radical scavenger and lipid antioxidant, etoposide (VP-16), inhibits sarcoplasmic reticulum Ca(2+)-ATPase via thiol oxidation by its phenoxyl radical. , 1995, Archives of biochemistry and biophysics.

[22]  Patrick R. Griffin,et al.  Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis , 1995, Nature.

[23]  M. Dragunow,et al.  The role of inducible transcription factors in apoptotic nerve cell death , 1995, Brain Research Reviews.

[24]  Muneesh Tewari,et al.  Yama/CPP32β, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase , 1995, Cell.

[25]  C. Thompson,et al.  Apoptosis in the pathogenesis and treatment of disease , 1995, Science.

[26]  A. Wyllie,et al.  The genetic regulation of apoptosis. , 1995, Current opinion in genetics & development.

[27]  S. Estus,et al.  Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis , 1994, The Journal of cell biology.

[28]  T. Murphy,et al.  Macromolecular synthesis inhibitors prevent oxidative stress-induced apoptosis in embryonic cortical neurons by shunting cysteine from protein synthesis to glutathione , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[29]  A. Tolkovsky,et al.  Apoptosis is induced in post-mitotic rat sympathetic neurons by arabinosides and topoisomerase II inhibitors in the presence of NGF. , 1994, Journal of cell science.

[30]  S. Grant,et al.  Induction of apoptotic DNA fragmentation and cell death in HL-60 human promyelocytic leukemia cells by pharmacological inhibitors of protein kinase C. , 1994, Cancer research.

[31]  M. Fishman,et al.  Prevention of vertebrate neuronal death by the crmA gene. , 1994, Science.

[32]  Shai Shaham,et al.  The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzyme , 1993, Cell.

[33]  N. Davidson,et al.  Specific proteolytic cleavage of poly(ADP-ribose) polymerase: an early marker of chemotherapy-induced apoptosis. , 1993, Cancer research.

[34]  J. Hickman,et al.  Apoptotic death in epithelial cells: cleavage of DNA to 300 and/or 50 kb fragments prior to or in the absence of internucleosomal fragmentation. , 1993, The EMBO journal.

[35]  A. Eastman Apoptosis: a product of programmed and unprogrammed cell death. , 1993, Toxicology and applied pharmacology.

[36]  Richard J Smeyne,et al.  Continuous c-fos expression precedes programmed cell death in vivo , 1993, Nature.

[37]  X. M. Sun,et al.  Dexamethasone-induced apoptosis involves cleavage of DNA to large fragments prior to internucleosomal fragmentation. , 1993, The Journal of biological chemistry.

[38]  X. M. Sun,et al.  Key morphological features of apoptosis may occur in the absence of internucleosomal DNA fragmentation. , 1992, The Biochemical journal.

[39]  I. Weissman,et al.  Bcl-2 prevents death of factor-deprived cells but fails to prevent apoptosis in targets of cell mediated killing. , 1992, International immunology.

[40]  J. Decaprio,et al.  Treatment of myeloid leukemic cells with the phosphatase inhibitor okadaic acid induces cell cycle arrest at either G1/S or G2/M depending on dose , 1992, Journal of cellular physiology.

[41]  S. Korsmeyer,et al.  bcl-2 inhibits multiple forms of apoptosis but not negative selection in thymocytes , 1991, Cell.

[42]  F. Pohl,et al.  A systematic study of field inversion gel electrophoresis. , 1989, Nucleic acids research.

[43]  R. Roeder,et al.  Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei. , 1983, Nucleic acids research.

[44]  A. Wyllie,et al.  Apoptosis: A Basic Biological Phenomenon with Wide-ranging Implications in Tissue Kinetics , 1972, British Journal of Cancer.

[45]  T. Chase,et al.  Nuclear Factor-κB Contributes to Excitotoxin-Induced Apoptosis in Rat Striatum , 1998 .

[46]  E. White,et al.  Life, death, and the pursuit of apoptosis. , 1996, Genes & development.

[47]  H. Horvitz,et al.  Mechanisms and functions of cell death. , 1991, Annual review of cell biology.

[48]  R. Oppenheim Cell death during development of the nervous system. , 1991, Annual review of neuroscience.

[49]  L. Liu,et al.  DNA topoisomerase poisons as antitumor drugs. , 1989, Annual review of biochemistry.

[50]  A. Wyllie,et al.  Cell death: the significance of apoptosis. , 1980, International review of cytology.