Construction of plasmids expressing Sars-CoV encoding proteins and their effects on transcription of hfgl2 prothrombinase

[1]  M. D. Chang,et al.  Accelerated induction of apoptosis in insect cells by baculovirus‐expressed SARS‐CoV membrane protein , 2006, FEBS Letters.

[2]  Jianguo Wu,et al.  Nucleocapsid protein of SARS-CoV activates the expression of cyclooxygenase-2 by binding directly to regulatory elements for nuclear factor-kappa B and CCAAT/enhancer binding protein , 2006, The International Journal of Biochemistry & Cell Biology.

[3]  P. Palese,et al.  7a Protein of Severe Acute Respiratory Syndrome Coronavirus Inhibits Cellular Protein Synthesis and Activates p38 Mitogen-Activated Protein Kinase , 2006, Journal of Virology.

[4]  Q. Ning,et al.  Fibrinogen-like protein 2 fibroleukin expression and its correlation with disease progression in murine hepatitis virus type 3-induced fulminant hepatitis and in patients with severe viral hepatitis B. , 2005, World journal of gastroenterology.

[5]  Y. Lau,et al.  Chemokine up-regulation in SARS-coronavirus–infected, monocyte-derived human dendritic cells , 2005, Blood.

[6]  F. Pei,et al.  [Lung pathology and pathogenesis of severe acute respiratory syndrome: a report of six full autopsies]. , 2005, Zhonghua bing li xue za zhi = Chinese journal of pathology.

[7]  K. Timani,et al.  Activation of NF‐κB by the Full‐length Nucleocapsid Protein of the SARS Coronavirus , 2005, Acta biochimica et biophysica Sinica.

[8]  Q. Ning,et al.  Role of Fibrinogen-Like Protein 2 Prothrombinase/Fibroleukin in Experimental and Human Allograft Rejection1 , 2005, The Journal of Immunology.

[9]  Shibo Jiang,et al.  Mapping of Antigenic Sites on the Nucleocapsid Protein of the Severe Acute Respiratory Syndrome Coronavirus , 2004, Journal of Clinical Microbiology.

[10]  V. Chow,et al.  The SARS coronavirus nucleocapsid protein induces actin reorganization and apoptosis in COS-1 cells in the absence of growth factors. , 2004, The Biochemical journal.

[11]  N. Bastien,et al.  Activation of AP-1 signal transduction pathway by SARS coronavirus nucleocapsid protein , 2003, Biochemical and Biophysical Research Communications.

[12]  M. Robertson.  Fgl2: link between hepatitis B and SARS? , 2003, Drug Discovery Today.

[13]  Q. Ning,et al.  The Fgl2/fibroleukin prothrombinase contributes to immunologically mediated thrombosis in experimental and human viral hepatitis. , 2003, The Journal of clinical investigation.

[14]  Christian Drosten,et al.  Characterization of a Novel Coronavirus Associated with Severe Acute Respiratory Syndrome , 2003, Science.

[15]  Obi L. Griffith,et al.  The Genome Sequence of the SARS-Associated Coronavirus , 2003, Science.

[16]  Christian Drosten,et al.  Identification of a novel coronavirus in patients with severe acute respiratory syndrome. , 2003, The New England journal of medicine.

[17]  Q. Ning,et al.  Induction of Prothrombinase fgl2 by the Nucleocapsid Protein of Virulent Mouse Hepatitis Virus Is Dependent on Host Hepatic Nuclear Factor-4α* , 2003, The Journal of Biological Chemistry.

[18]  Huanming Yang,et al.  The M Protein of SARS-CoV: Basic Structural and Immunological Properties , 2003, Genomics, Proteomics & Bioinformatics.

[19]  G. Brooks,et al.  Interaction of the Coronavirus Nucleoprotein with Nucleolar Antigens and the Host Cell , 2002, Journal of Virology.

[20]  P. Marsden,et al.  Molecular and Functional Analysis of the Human Prothrombinase Gene (HFGL2) and Its Role in Viral Hepatitis , 2000, The American Journal of Pathology.

[21]  M. Lai,et al.  The Nucleocapsid Protein of Murine Hepatitis Virus Type 3 Induces Transcription of the Novel fgl2 Prothrombinase Gene* , 1999, The Journal of Biological Chemistry.

[22]  C. Rüegg,et al.  Characterization of human fibroleukin, a fibrinogen-like protein secreted by T lymphocytes. , 1998, Journal of immunology.

[23]  Q. Ning,et al.  Expression of the fgl2 and its protein product (prothrombinase) in tissues during murine hepatitis virus strain-3 (MHV-3) infection. , 1998, Advances in experimental medicine and biology.