Mitofusin 2 ablation increases endoplasmic reticulum–mitochondria coupling
暂无分享,去创建一个
Elisa Greotti | Tullio Pozzan | Paola Pizzo | Riccardo Filadi | Alberto Luini | A. Luini | T. Pozzan | P. Pizzo | R. Filadi | E. Greotti | Gabriele Turacchio | Gabriele Turacchio | Elisa Greotti
[1] J. Vance. Phospholipid synthesis in a membrane fraction associated with mitochondria. , 1990, The Journal of biological chemistry.
[2] J. Vance,et al. A unique mitochondria-associated membrane fraction from rat liver has a high capacity for lipid synthesis and contains pre-Golgi secretory proteins including nascent lipoproteins. , 1994, The Journal of biological chemistry.
[3] M. Madesh,et al. Control of apoptosis by IP(3) and ryanodine receptor driven calcium signals. , 2000, Cell calcium.
[4] A. Santel,et al. Control of mitochondrial morphology by a human mitofusin. , 2001, Journal of cell science.
[5] A. Lombès,et al. Membrane topology and mitochondrial targeting of mitofusins, ubiquitous mammalian homologs of the transmembrane GTPase Fzo. , 2002, Journal of cell science.
[6] D. Schmitt,et al. Subcellular compartmentalization of ceramide metabolism: MAM (mitochondria-associated membrane) and/or mitochondria? , 2004, The Biochemical journal.
[7] J. McCaffery,et al. Structural Basis of Mitochondrial Tethering by Mitofusin Complexes , 2004, Science.
[8] F. Cordelières,et al. A guided tour into subcellular colocalization analysis in light microscopy , 2006, Journal of microscopy.
[9] C. Mannella,et al. Structural and functional features and significance of the physical linkage between ER and mitochondria , 2006, The Journal of cell biology.
[10] Y. Yoon,et al. Mitochondrial clustering induced by overexpression of the mitochondrial fusion protein Mfn2 causes mitochondrial dysfunction and cell death. , 2007, European journal of cell biology.
[11] L. Scorrano,et al. Mitofusin 2 tethers endoplasmic reticulum to mitochondria , 2008, Nature.
[12] M. Duchen,et al. Mitochondria: the hub of cellular Ca2+ signaling. , 2008, Physiology.
[13] G. Hajnóczky,et al. MAM: more than just a housekeeper. , 2009, Trends in cell biology.
[14] Benedikt Westermann,et al. Mitochondrial fusion and fission in cell life and death , 2010, Nature Reviews Molecular Cell Biology.
[15] P. Pandolfi,et al. PML Regulates Apoptosis at Endoplasmic Reticulum by Modulating Calcium Release , 2010, Science.
[16] David N. Mastronarde,et al. ER sliding dynamics and ER–mitochondrial contacts occur on acetylated microtubules , 2010, The Journal of cell biology.
[17] M. Bortolozzi,et al. Ca2+ hot spots on the mitochondrial surface are generated by Ca2+ mobilization from stores, but not by activation of store-operated Ca2+ channels. , 2010, Molecular cell.
[18] György Hajnóczky,et al. Imaging interorganelle contacts and local calcium dynamics at the ER-mitochondrial interface. , 2010, Molecular cell.
[19] T. Pozzan,et al. Mitochondria: the calcium connection. , 2010, Biochimica et biophysica acta.
[20] M. Birnbaum,et al. Essential Regulation of Cell Bioenergetics by Constitutive InsP3 Receptor Ca2+ Transfer to Mitochondria , 2010, Cell.
[21] I. Ambudkar,et al. Faculty Opinions recommendation of A forty-kilodalton protein of the inner membrane is the mitochondrial calcium uniporter. , 2011 .
[22] V. Mootha,et al. Integrative genomics identifies MCU as an essential component of the mitochondrial calcium uniporter , 2011, Nature.
[23] M. Bortolozzi,et al. Presenilin 2 modulates endoplasmic reticulum (ER)–mitochondria interactions and Ca2+ cross-talk , 2011, Proceedings of the National Academy of Sciences.
[24] Matthew West,et al. ER Tubules Mark Sites of Mitochondrial Division , 2011, Science.
[25] J. Vicencio,et al. Increased ER–mitochondrial coupling promotes mitochondrial respiration and bioenergetics during early phases of ER stress , 2011, Journal of Cell Science.
[26] T. Pozzan,et al. Mitochondrial Ca2+ homeostasis: mechanism, role, and tissue specificities , 2012, Pflügers Archiv - European Journal of Physiology.
[27] Michael R. Duchen,et al. Mitochondria, calcium-dependent neuronal death and neurodegenerative disease , 2012, Pflügers Archiv - European Journal of Physiology.
[28] G. Voeltz,et al. Endoplasmic reticulum–mitochondria contacts: function of the junction , 2012, Nature Reviews Molecular Cell Biology.
[29] R. Balaban,et al. Role of mitochondrial Ca2+ in the regulation of cellular energetics. , 2012, Biochemistry.
[30] L. Orci,et al. Mitofusin-2 Independent Juxtaposition of Endoplasmic Reticulum and Mitochondria: An Ultrastructural Study , 2012, PloS one.
[31] H. Higgs,et al. An Actin-Dependent Step in Mitochondrial Fission Mediated by the ER-Associated Formin INF2 , 2013, Science.
[32] L. Scorrano,et al. Silencing of the Charcot–Marie–Tooth disease-associated gene GDAP1 induces abnormal mitochondrial distribution and affects Ca2+ homeostasis by reducing store-operated Ca2+ entry , 2013, Neurobiology of Disease.
[33] Yasushi Hiraoka,et al. Autophagosomes form at ER–mitochondria contact sites , 2013, Nature.
[34] B. Kornmann,et al. The molecular hug between the ER and the mitochondria. , 2013, Current opinion in cell biology.
[35] T. Simmen,et al. Where the endoplasmic reticulum and the mitochondrion tie the knot: the mitochondria-associated membrane (MAM). , 2013, Biochimica et biophysica acta.
[36] Q. Nie,et al. Mitofusin 2 deficiency leads to oxidative stress that contributes to insulin resistance in rat skeletal muscle cells , 2014, Molecular Biology Reports.
[37] G. Hong,et al. Role of Mitofusin-2 in High Mobility Group Box-1 Protein-Mediated Apoptosis of T Cells in Vitro , 2014, Cellular Physiology and Biochemistry.
[38] P. Pontisso,et al. SERPINB3 protects from oxidative damage by chemotherapeutics through inhibition of mitochondrial respiratory complex I , 2013, Oncotarget.
[39] W. Prinz. Bridging the gap: Membrane contact sites in signaling, metabolism, and organelle dynamics , 2014, The Journal of cell biology.
[40] B. Westermann,et al. Making connections: interorganelle contacts orchestrate mitochondrial behavior. , 2014, Trends in cell biology.
[41] C. Dieterich,et al. Mitofusin 2 is required to maintain mitochondrial coenzyme Q levels , 2015, The Journal of cell biology.