A monoclonal antibody against the CD18 leukocyte adhesion molecule prevents indomethacin-induced gastric damage in the rabbit.

[1]  J. Wallace,et al.  Gastric ulceration induced by nonsteroidal anti-inflammatory drugs is a neutrophil-dependent process. , 1990, The American journal of physiology.

[2]  J. Wallace,et al.  The mucoid cap over superficial gastric damage in the rat: A high-pH microenvironment dissipated by nonsteroidal antiinflammatory drugs and endothelin , 1990 .

[3]  P. Kvietys,et al.  Ethanol-induced injury to the rat gastric mucosa. Role of neutrophils and xanthine oxidase-derived radicals. , 1990, Gastroenterology.

[4]  J. Wallace,et al.  Lipid mediators of inflammation in gastric ulcer. , 1990, The American journal of physiology.

[5]  P. Kubes,et al.  Platelet-activating factor-induced microvascular dysfunction: role of adherent leukocytes. , 1990, The American journal of physiology.

[6]  M. Grisham,et al.  Leukocyte adherence to venular endothelium during ischemia-reperfusion. , 1989, The American journal of physiology.

[7]  E. Tuomanen,et al.  Reduction of inflammation, tissue damage, and mortality in bacterial meningitis in rabbits treated with monoclonal antibodies against adhesion-promoting receptors of leukocytes , 1989, The Journal of experimental medicine.

[8]  E. Chi,et al.  A monoclonal antibody to the adherence-promoting leukocyte glycoprotein, CD18, reduces organ injury and improves survival from hemorrhagic shock and resuscitation in rabbits. , 1988, The Journal of clinical investigation.

[9]  G. Zimmerman,et al.  Neutrophil adherence to human endothelium in vitro occurs by CDw18 (Mo1, MAC-1/LFA-1/GP 150,95) glycoprotein-dependent and -independent mechanisms. , 1988, The Journal of clinical investigation.

[10]  P. Guth,et al.  Effect of aspirin plus hydrochloric acid on the gastric mucosal microcirculation. , 1987, Gastroenterology.

[11]  M. Grisham,et al.  Role of neutrophils in hemorrhagic shock-induced gastric mucosal injury in the rat. , 1987, Gastroenterology.

[12]  J. Koo,et al.  Focal Gastric Mucosal Blood Flow in Aspirin‐induced Ulceration , 1987, Annals of surgery.

[13]  H. Ochs,et al.  An endothelial cell surface factor(s) induced in vitro by lipopolysaccharide, interleukin 1, and tumor necrosis factor-alpha increases neutrophil adherence by a CDw18-dependent mechanism. , 1986, Journal of immunology.

[14]  M. Arnaout,et al.  Monoclonal Antibodies That Identify Mo1 and LFA-1, Two Human Leukocyte Membrane Glycoproteins: A Review , 1986 .

[15]  T. Springer The LFA-1, Mac-1 glycoprotein family and its deficiency in an inherited disease. , 1985, Federation proceedings.

[16]  J. Fantone Mechanisms of Chemotactic Factor Stimulation of Polymorphonuclear Leukocytes: Modulation by Prostaglandins , 1985 .

[17]  S. Ashley,et al.  Focal gastric mucosal blood flow at the site of aspirin-induced ulceration. , 1985, American journal of surgery.

[18]  R. Steinman,et al.  Specific antimononuclear phagocyte monoclonal antibodies. Application to the purification of dendritic cells and the tissue localization of macrophages , 1983, The Journal of experimental medicine.

[19]  D. Priebat,et al.  Measurement of cutaneous inflammation: estimation of neutrophil content with an enzyme marker. , 1982, The Journal of investigative dermatology.

[20]  B. Whittle Temporal Relationship Between Cyclooxygenase Inhibition, as Measured by Prostacyclin Biosynthesis, and the Gastrointestinal Damage Induced by Indomethacin in the Rat , 1981 .