β3AR-Dependent Brain-Derived Neurotrophic Factor (BDNF) Generation Limits Chronic Postischemic Heart Failure
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L. Finos | W. Koch | D. Liccardo | E. Gao | N. Ferrara | N. Paolocci | A. Megighian | G. Rengo | L. Marcucci | K. Kammers | A. Cannavo | Andrea Elia | Jacopo Agrimi | N. Feng | Gizem Keceli | G. G. Altobelli | Seungho Jun | Federica Marzano
[1] V. Fuster,et al. Beta-3 adrenergic receptor overexpression reverses aortic stenosis–induced heart failure and restores balanced mitochondrial dynamics , 2022, Basic Research in Cardiology.
[2] T. Matsukawa,et al. Cardioprotective effects of enteral vs. parenteral lactoferrin administration on myocardial ischemia-reperfusion injury in a rat model of stunned myocardium , 2022, BMC Pharmacology and Toxicology.
[3] D. Kass,et al. Myocardial brain-derived neurotrophic factor regulates cardiac bioenergetics through the transcription factor Yin Yang 1. , 2022, Cardiovascular research.
[4] B. O’Rourke,et al. Hydropersulfides (RSSH) Outperform Post-Conditioning and Other Reactive Sulfur Species in Limiting Ischemia–Reperfusion Injury in the Isolated Mouse Heart , 2022, Antioxidants.
[5] M. Wojciechowska,et al. Insight into the Role of the PI3K/Akt Pathway in Ischemic Injury and Post-Infarct Left Ventricular Remodeling in Normal and Diabetic Heart , 2022, Cells.
[6] D. Kass,et al. MTORC1-Regulated Metabolism Controlled by TSC2 Limits Cardiac Reperfusion Injury. , 2021, Circulation research.
[7] A. Zamani,et al. Acute treatment with TrkB agonist LM22A-4 confers neuroprotection and preserves myelin integrity in a mouse model of pediatric traumatic brain injury , 2020, Experimental Neurology.
[8] J. Balligand,et al. The Beta3 Adrenergic Receptor in Healthy and Pathological Cardiovascular Tissues , 2020, Cells.
[9] H. Calkins,et al. Psychosocial Stress Hastens Disease Progression and Sudden Death in Mice with Arrhythmogenic Cardiomyopathy , 2020, Journal of clinical medicine.
[10] Pei-Feng Li,et al. Brain-derived neurotrophic factor mimetic, 7,8-dihydroxyflavone, protects against myocardial ischemia by rebalancing optic atrophy 1 processing. , 2019, Free radical biology & medicine.
[11] H. Völzke,et al. Brain-derived neurotrophic factor is related with adverse cardiac remodeling and high NTproBNP , 2019, Scientific Reports.
[12] Junhua Xiao,et al. TrkB Agonist LM22A-4 Increases Oligodendroglial Populations During Myelin Repair in the Corpus Callosum , 2019, Front. Mol. Neurosci..
[13] M. Bellingham,et al. The Role of Altered BDNF/TrkB Signaling in Amyotrophic Lateral Sclerosis , 2019, Front. Cell. Neurosci..
[14] B. Hempstead,et al. BDNF Actions in the Cardiovascular System: Roles in Development, Adulthood and Response to Injury , 2019, Front. Physiol..
[15] A. Lochner,et al. Cardioprotective Effects of Beta3-Adrenergic Receptor (β3-AR) Pre-, Per-, and Post-treatment in Ischemia–Reperfusion , 2019, Cardiovascular Drugs and Therapy.
[16] Dong I. Lee,et al. Nitroxyl (HNO) targets phospholamban cysteines 41 and 46 to enhance cardiac function , 2019, The Journal of general physiology.
[17] Stephen T. C. Wong,et al. Sustained Adrenergic Signaling Promotes Intratumoral Innervation through BDNF Induction. , 2018, Cancer research.
[18] Jeremy J. Walsh,et al. Exercise and circulating BDNF: Mechanisms of release and implications for the design of exercise interventions. , 2018, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.
[19] D. Leosco,et al. β1-Blockade Prevents Post-Ischemic Myocardial Decompensation Via β3AR-Dependent Protective Sphingosine-1 Phosphate Signaling. , 2017, Journal of the American College of Cardiology.
[20] M. Mattson,et al. Brain metabolism in health, aging, and neurodegeneration , 2017, The EMBO journal.
[21] S. Johansson,et al. Mortality and morbidity trends after the first year in survivors of acute myocardial infarction: a systematic review , 2017, BMC Cardiovascular Disorders.
[22] J. Balligand. Cardiac salvage by tweaking with beta-3-adrenergic receptors. , 2016, Cardiovascular research.
[23] L. Tessarollo,et al. BDNF modulates heart contraction force and long-term homeostasis through truncated TrkB.T1 receptor activation , 2015, The Journal of cell biology.
[24] Wenbo Wang,et al. Protective effects of LM22A-4 on injured spinal cord nerves. , 2015, International journal of clinical and experimental pathology.
[25] Z. Du,et al. Brain-Derived Neurotrophic Factor Regulates TRPC3/6 Channels and Protects Against Myocardial Infarction in Rodents , 2015, International journal of biological sciences.
[26] K. Ye,et al. Activation of muscular TrkB by its small molecular agonist 7,8-dihydroxyflavone sex-dependently regulates energy metabolism in diet-induced obese mice. , 2015, Chemistry & biology.
[27] D. Kass,et al. Constitutive BDNF/TrkB signaling is required for normal cardiac contraction and relaxation , 2015, Autonomic Neuroscience.
[28] Jelle J Goeman,et al. Rotation‐based multiple testing in the multivariate linear model , 2014, Biometrics.
[29] V. Fuster,et al. β3 adrenergic receptor selective stimulation during ischemia/reperfusion improves cardiac function in translational models through inhibition of mPTP opening in cardiomyocytes , 2014, Basic Research in Cardiology.
[30] M. Srivatsan,et al. Neuroprotective effects of physical activity on the brain: a closer look at trophic factor signaling , 2014, Front. Cell. Neurosci..
[31] Xin Lu,et al. Nebivolol Protects against Myocardial Infarction Injury via Stimulation of Beta 3-Adrenergic Receptors and Nitric Oxide Signaling , 2014, PloS one.
[32] Jelle J. Goeman,et al. Multiple hypothesis testing in genomics , 2014, Statistics in medicine.
[33] M. Mattson,et al. Evidence that BDNF regulates heart rate by a mechanism involving increased brainstem parasympathetic neuron excitability , 2014, Journal of neurochemistry.
[34] D. Leosco,et al. Vascular Endothelial Growth Factor Blockade Prevents the Beneficial Effects of &bgr;-Blocker Therapy on Cardiac Function, Angiogenesis, and Remodeling in Heart Failure , 2013, Circulation. Heart failure.
[35] S. McClintock,et al. Small molecules activating TrkB receptor for treating a variety of CNS disorders. , 2013, CNS & neurological disorders drug targets.
[36] Huirong Liu,et al. Autoantibodies against the β3-Adrenoceptor Protect from Cardiac Dysfunction in a Rat Model of Pressure Overload , 2013, PloS one.
[37] J. Hensler,et al. Reduced BDNF attenuates inflammation and angiogenesis to improve survival and cardiac function following myocardial infarction in mice , 2013, American journal of physiology. Heart and circulatory physiology.
[38] I. Komuro,et al. Brain-Derived Neurotrophic Factor Protects Against Cardiac Dysfunction After Myocardial Infarction via a Central Nervous System–Mediated Pathway , 2012, Arteriosclerosis, Thrombosis and Vascular Biology.
[39] D. Rognan,et al. Identification of a low-molecular weight TrkB antagonist with anxiolytic and antidepressant activity in mice. , 2011, The Journal of clinical investigation.
[40] M. Cerqueira,et al. Myocardial iodine-123 meta-iodobenzylguanidine imaging and cardiac events in heart failure. Results of the prospective ADMIRE-HF (AdreView Myocardial Imaging for Risk Evaluation in Heart Failure) study. , 2010, Journal of the American College of Cardiology.
[41] M. Bilgen,et al. Small molecule BDNF mimetics activate TrkB signaling and prevent neuronal degeneration in rodents. , 2010, Journal of Clinical Investigation.
[42] S. Cohen-Cory,et al. Brain‐derived neurotrophic factor and the development of structural neuronal connectivity , 2010, Developmental neurobiology.
[43] L. Minichiello. TrkB signalling pathways in LTP and learning , 2009, Nature Reviews Neuroscience.
[44] K. Fukuda,et al. Cardiac Innervation and Sudden Cardiac Death , 2009, Current cardiology reviews.
[45] Henriette Pilegaard,et al. Evidence for a release of brain‐derived neurotrophic factor from the brain during exercise , 2009, Experimental physiology.
[46] M. Febbraio,et al. Brain-derived neurotrophic factor is produced by skeletal muscle cells in response to contraction and enhances fat oxidation via activation of AMP-activated protein kinase , 2009, Diabetologia.
[47] Y. Kakinuma,et al. Chronic intermittent fasting improves the survival following large myocardial ischemia by activation of BDNF/VEGF/PI3K signaling pathway. , 2009, Journal of molecular and cellular cardiology.
[48] Brian Olshansky,et al. Parasympathetic nervous system and heart failure: pathophysiology and potential implications for therapy. , 2008, Circulation.
[49] C. Pike,et al. Norepinephrine induces BDNF and activates the PI-3K and MAPK cascades in embryonic hippocampal neurons. , 2007, Cellular signalling.
[50] I. Shiojima,et al. Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure. , 2005, The Journal of clinical investigation.
[51] Michael V. Cohen,et al. Xanthine oxidase contributes to preconditioning's preservation of left ventricular developed pressure in isolated rat heart: developed pressure may not be an appropriate end-point for studies of preconditioning , 2002, Basic Research in Cardiology.
[52] S. Meri,et al. Nerve growth factor and brain‐derived neurotrophic factor mRNAs are regulated in distinct cell populations of rat heart after ischaemia and reperfusion , 2001, The Journal of pathology.
[53] K. Desai,et al. Targeted Disruption of the β2 Adrenergic Receptor Gene* , 1999, The Journal of Biological Chemistry.
[54] G. Barsh,et al. Targeted disruption of the mouse beta1-adrenergic receptor gene: developmental and cardiovascular effects. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[55] D P Zipes,et al. Influence of Myocardial Ischemia and Infarction on Autonomic Innervation of Heart , 1990, Circulation.
[56] D C Harrison,et al. Decreased catecholamine sensitivity and beta-adrenergic-receptor density in failing human hearts. , 1982, The New England journal of medicine.
[57] E. Braunwald,et al. CATECHOLAMINE EXCRETION AND CARDIAC STORES OF NOREPINEPHRINE IN CONGESTIVE HEART FAILURE. , 1965, The American journal of medicine.