Killer immunoglobulin-like receptors can predict TKI treatment-free remission in chronic myeloid leukemia patients.
暂无分享,去创建一个
C. Carcassi | G. Caocci | G. la Nasa | R. Littera | S. Galimberti | B. Martino | C. Baratè | M. Trawinska | E. Abruzzese | M. Greco | C. Labate | O. Mulas | C. Gambacorti Passerini | S. Lai | Paola Ragatzu | G. La Nasa
[1] Martin C. Müller,et al. Early Disease Relapse after Tyrosine Kinase Inhibitor Treatment Discontinuation in CML Is Related Both to Low Number and Impaired Function of NK-Cells , 2014 .
[2] C. Gambacorti-Passerini,et al. The Risk of Relapse in CML Patients Who Discontinued imatinib Can Be Predicted Based on Patients Age and the Results of dPCR Analysis , 2014 .
[3] J. Cayuela,et al. Loss of major molecular response as a trigger for restarting tyrosine kinase inhibitor therapy in patients with chronic-phase chronic myelogenous leukemia who have stopped imatinib after durable undetectable disease. , 2014, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[4] S. Mustjoki,et al. Immunology and Immunotherapy of Chronic Myeloid Leukemia , 2014, Current Hematologic Malignancy Reports.
[5] U. Olsson‐Strömberg,et al. Disease Relapse After Tyrosine Kinase Inhibitor Treatment Discontinuation in Chronic Myeloid Leukaemia is Related to Both Low Number and Impaired Function of NK Cells , 2014 .
[6] T. Yoshimoto,et al. Sustained upregulation of effector natural killer cells in chronic myeloid leukemia after discontinuation of imatinib , 2013, Cancer science.
[7] L. Costa. Defects in DBA: more than meets the eye , 2013 .
[8] J. Melo,et al. Safety and efficacy of imatinib cessation for CML patients with stable undetectable minimal residual disease: results from the TWISTER study. , 2013, Blood.
[9] C. Carcassi,et al. Homozygosity for killer immunoglobin-like receptor haplotype A predicts complete molecular response to treatment with tyrosine kinase inhibitors in chronic myeloid leukemia patients. , 2013, Experimental hematology.
[10] P. Parham,et al. Variable NK cell receptors and their MHC class I ligands in immunity, reproduction and human evolution , 2013, Nature Reviews Immunology.
[11] D. Greco,et al. Killer-cell immunoglobulin-like receptor gene profile predicts good molecular response to dasatinib therapy in chronic myeloid leukemia. , 2012, Experimental hematology.
[12] D. Marin,et al. KIR2DS1 genotype predicts for complete cytogenetic response and survival in newly diagnosed chronic myeloid leukemia patients treated with imatinib , 2012, Leukemia.
[13] Philippe Rousselot,et al. Discontinuation of imatinib in patients with chronic myeloid leukaemia who have maintained complete molecular remission for at least 2 years: the prospective, multicentre Stop Imatinib (STIM) trial. , 2010, The Lancet. Oncology.
[14] K. Campbell,et al. 自然杀伤细胞与癌症——杀伤细胞免疫球蛋白样受体(KIR)的调控 , 2010, Zhongguo fei ai za zhi = Chinese journal of lung cancer.
[15] P. Gourraud,et al. Killer immunoglobulin-like receptors (KIR2DL2 and/or KIR2DS2) in presence of their ligand (HLA-C1 group) protect against chronic myeloid leukaemia. , 2009, Tissue antigens.
[16] J. Goldman,et al. Why do chronic myelogenous leukemia stem cells survive allogeneic stem cell transplantation or imatinib: does it really matter? , 2006, Leukemia & lymphoma.
[17] K. Hsu,et al. The killer cell immunoglobulin‐like receptor (KIR) genomic region: gene‐order, haplotypes and allelic polymorphism , 2002, Immunological reviews.