Genetic analysis of atherosclerosis identifies a major susceptibility locus in the major histocompatibility complex of mice.

[1]  Tom R. Gaunt,et al.  Plasma urate concentration and risk of coronary heart disease: a Mendelian randomisation analysis , 2016, The lancet. Diabetes & endocrinology.

[2]  Weibin Shi,et al.  Mapping and Congenic Dissection of Genetic Loci Contributing to Hyperglycemia and Dyslipidemia in Mice , 2016, PloS one.

[3]  A. Manichaikul,et al.  Genetic linkage of hyperglycemia and dyslipidemia in an intercross between BALB/cJ and SM/J Apoe-deficient mouse strains , 2015, BMC Genetics.

[4]  Sebastian M. Armasu,et al.  A comprehensive 1000 Genomes-based genome-wide association meta-analysis of coronary artery disease , 2015, Nature Genetics.

[5]  Zhenqi Liu,et al.  Variation in Type 2 Diabetes-Related Phenotypes among Apolipoprotein E-Deficient Mouse Strains , 2015, PloS one.

[6]  Tom R. Gaunt,et al.  Mendelian randomization of blood lipids for coronary heart disease , 2014, European heart journal.

[7]  A. Lusis,et al.  Genetics of common forms of heart disease: a long and winding road. , 2013, Circulation research.

[8]  A. Manichaikul,et al.  Atherosclerosis Susceptibility Loci Identified in an Extremely Atherosclerosis‐Resistant Mouse Strain , 2013, Journal of the American Heart Association.

[9]  Weibin Shi,et al.  New quantitative trait loci for carotid atherosclerosis identified in an intercross derived from apolipoprotein E-deficient mouse strains , 2013, Physiological genomics.

[10]  J. Danesh,et al.  Large-scale association analysis identifies new risk loci for coronary artery disease , 2012, Nature Genetics.

[11]  A. Khera,et al.  Association Between Family History and Coronary Heart Disease Death Across Long-Term Follow-Up in Men: The Cooper Center Longitudinal Study , 2012, Circulation.

[12]  Weibin Shi,et al.  Genetic Analysis of Atherosclerosis and Glucose Homeostasis in an Intercross Between C57BL/6 and BALB/cJ Apolipoprotein E–Deficient Mice , 2012, Circulation. Cardiovascular genetics.

[13]  A. Manichaikul,et al.  Characterization of Ath29, a major mouse atherosclerosis susceptibility locus, and identification of Rcn2 as a novel regulator of cytokine expression. , 2011, American journal of physiology. Heart and circulatory physiology.

[14]  S. Sündermann,et al.  Cosegregation of Aortic Root Atherosclerosis and Intermediate Lipid Phenotypes on Chromosomes 2 and 8 in an Intercross of C57BL/6 and BALBc/ByJ Low-Density Lipoprotein Receptor−/− Mice , 2011, Arteriosclerosis, thrombosis, and vascular biology.

[15]  Mark I. McCarthy,et al.  A genome-wide association study in Europeans and South Asians identifies five new loci for coronary artery disease , 2011, Nature Genetics.

[16]  S. Bradley,et al.  Balance of meprin A and B in mice affects the progression of experimental inflammatory bowel disease. , 2011, American journal of physiology. Gastrointestinal and liver physiology.

[17]  Weibin Shi,et al.  Genes Within the MHC Region Have a Dramatic Influence on Radiation-Enhanced Atherosclerosis in Mice , 2010, Circulation. Cardiovascular genetics.

[18]  H. Tomita,et al.  Aortic Arch Curvature and Atherosclerosis Have Overlapping Quantitative Trait Loci in a Cross Between 129S6/SvEvTac and C57BL/6J Apolipoprotein E-Null Mice , 2010, Circulation research.

[19]  Mark D. Huffman,et al.  Heart Disease and Stroke Statistics—2015 Update: A Report From the American Heart Association , 2009, Circulation.

[20]  S. Dobrin,et al.  Quantitative Trait Locus Analysis of Carotid Atherosclerosis in an Intercross Between C57BL/6 and C3H Apolipoprotein E–Deficient Mice , 2008, Stroke.

[21]  Eric E Schadt,et al.  Identification of Pathways for Atherosclerosis in Mice: Integration of Quantitative Trait Locus Analysis and Global Gene Expression Data , 2007, Circulation research.

[22]  J. Weber,et al.  Quantitative Trait Locus Analysis of Atherosclerosis in an Intercross Between C57BL/6 and C3H Mice Carrying the Mutant Apolipoprotein E Gene , 2006, Genetics.

[23]  Jeffrey M. Bhasin,et al.  Atherosclerosis Susceptibility Loci Identified From a Strain Intercross of Apolipoprotein E–Deficient Mice via a High-Density Genome Scan , 2005, Arteriosclerosis, thrombosis, and vascular biology.

[24]  Per Eriksson,et al.  Positional identification of TNFSF4, encoding OX40 ligand, as a gene that influences atherosclerosis susceptibility , 2005, Nature Genetics.

[25]  Ralph B D'Agostino,et al.  Risk of complications of pregnancy in women with type 1 diabetes: nationwide prospective study in the Netherlands , 2004, BMJ : British Medical Journal.

[26]  Robert W. Williams,et al.  The nature and identification of quantitative trait loci: a community's view , 2003, Nature Reviews Genetics.

[27]  Eric S. Lander,et al.  The mosaic structure of variation in the laboratory mouse genome , 2002, Nature.

[28]  G. Duyk,et al.  A phenotype-sensitizing Apoe-deficient genetic background reveals novel atherosclerosis predisposition loci in the mouse. , 2002, Genetics.

[29]  A. Tall,et al.  Localization of atherosclerosis susceptibility loci to chromosomes 4 and 6 using the Ldlr knockout mouse model , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[30]  D K Burns,et al.  Massive xanthomatosis and atherosclerosis in cholesterol-fed low density lipoprotein receptor-negative mice. , 1994, The Journal of clinical investigation.

[31]  L. Berkman,et al.  Genetic susceptibility to death from coronary heart disease in a study of twins. , 1994, The New England journal of medicine.

[32]  Carol J. Bult,et al.  The mouse as a model for human biology: a resource guide for complex trait analysis , 2007, Nature Reviews Genetics.

[33]  R. Ross,et al.  ApoE-deficient mice develop lesions of all phases of atherosclerosis throughout the arterial tree. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.