Activation of NF-kappaB-dependent gene expression by Salmonella flagellins FliC and FljB.

[1]  C. Samuel,et al.  Innate interferon response in macrophage and epithelial cells infected with wild-type compared to DNA adenine methylase and flagellin mutant Salmonella enterica serovar Typhimurium. , 2007, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.

[2]  D. M. Heithoff,et al.  Altered Levels of Salmonella DNA Adenine Methylase Are Associated with Defects in Gene Expression, Motility, Flagellar Synthesis, and Bile Resistance in the Pathogenic Strain 14028 but Not in the Laboratory Strain LT2 , 2006, Journal of bacteriology.

[3]  R. Flavell,et al.  Involvement of Toll-like receptor 5 in the recognition of flagellated bacteria , 2006, Proceedings of the National Academy of Sciences.

[4]  C. Coban,et al.  Detection of pathogenic intestinal bacteria by Toll-like receptor 5 on intestinal CD11c+ lamina propria cells , 2006, Nature Immunology.

[5]  J. Bertin,et al.  Cytosolic flagellin requires Ipaf for activation of caspase-1 and interleukin 1β in salmonella-infected macrophages , 2006, Nature Immunology.

[6]  Alan Aderem,et al.  Cytoplasmic flagellin activates caspase-1 and secretion of interleukin 1β via Ipaf , 2006, Nature Immunology.

[7]  N. Subramanian,et al.  Lysophospholipid sensing triggers secretion of flagellin from pathogenic salmonella , 2006, Nature Immunology.

[8]  S. McSorley,et al.  Salmonella flagellin, a microbial target of the innate and adaptive immune system. , 2005, Immunology letters.

[9]  R. Alaniz,et al.  CD4+-T-Cell Responses Generated during Murine Salmonella enterica Serovar Typhimurium Infection Are Directed towards Multiple Epitopes within the Natural Antigen FliC , 2005, Infection and Immunity.

[10]  A. Aderem,et al.  Evasion of Toll-like receptor 5 by flagellated bacteria. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[11]  B. Cookson,et al.  FliC-Specific CD4+ T Cell Responses Are Restricted by Bacterial Regulation of Antigen Expression1 , 2005, The Journal of Immunology.

[12]  M. D. de Veer,et al.  Flagellin acting via TLR5 is the major activator of key signaling pathways leading to NF-κB and proinflammatory gene program activation in intestinal epithelial cells , 2004, BMC Microbiology.

[13]  M. Greenblatt,et al.  A Toll-like Receptor That Prevents Infection by Uropathogenic Bacteria , 2004, Science.

[14]  M. Wick.  Living in the danger zone: innate immunity to Salmonella. , 2004, Current opinion in microbiology.

[15]  C. Josenhans,et al.  Helicobacter pylori flagellins have very low intrinsic activity to stimulate human gastric epithelial cells via TLR5. , 2003, Microbes and infection.

[16]  A. Aderem,et al.  Toll-like receptor 5 recognizes a conserved site on flagellin required for protofilament formation and bacterial motility , 2003, Nature Immunology.

[17]  J. Madara,et al.  Flagellin Is the Major Proinflammatory Determinant of Enteropathogenic Salmonella1 , 2003, The Journal of Immunology.

[18]  K. Namba,et al.  Complete atomic model of the bacterial flagellar filament by electron cryomicroscopy , 2003, Nature.

[19]  S. Mizel,et al.  Identification of a Sequence in Human Toll-like Receptor 5 Required for the Binding of Gram-negative Flagellin* , 2003, Journal of Biological Chemistry.

[20]  K. Hughes,et al.  Flagellar Phase Variation in Salmonella enterica Is Mediated by a Posttranscriptional Control Mechanism , 2003, Journal of bacteriology.

[21]  C. Samuel,et al.  Tissue Selectivity of Interferon-Stimulated Gene Expression in Mice Infected with Dam+ versus Dam−Salmonella enterica Serovar Typhimurium Strains , 2002, Infection and Immunity.

[22]  T. Wallis,et al.  Absence of All Components of the Flagellar Export and Synthesis Machinery Differentially Alters Virulence of Salmonella enterica Serovar Typhimurium in Models of Typhoid Fever, Survival in Macrophages, Tissue Culture Invasiveness, and Calf Enterocolitis , 2001, Infection and Immunity.

[23]  P. Godowski,et al.  Cutting Edge: Bacterial Flagellin Activates Basolaterally Expressed TLR5 to Induce Epithelial Proinflammatory Gene Expression1 , 2001, The Journal of Immunology.

[24]  S. Makino,et al.  Salmonella enteritidis FliC (Flagella Filament Protein) Induces Human β-Defensin-2 mRNA Production by Caco-2 Cells* , 2001, The Journal of Biological Chemistry.

[25]  S. Srinivasula,et al.  Identification of Ipaf, a Human Caspase-1-activating Protein Related to Apaf-1* , 2001, The Journal of Biological Chemistry.

[26]  T. Wallis,et al.  Flagellar Phase Variation of Salmonella enterica Serovar Typhimurium Contributes to Virulence in the Murine Typhoid Infection Model but Does Not InfluenceSalmonella-Induced Enteropathogenesis , 2001, Infection and Immunity.

[27]  S. Akira,et al.  The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5 , 2001, Nature.

[28]  K. Hughes,et al.  Coupling of Flagellar Gene Expression to Flagellar Assembly in Salmonella enterica Serovar Typhimurium andEscherichia coli , 2000, Microbiology and Molecular Biology Reviews.

[29]  Patrick F. McDermott,et al.  High-Affinity Interaction between Gram-Negative Flagellin and a Cell Surface Polypeptide Results in Human Monocyte Activation , 2000, Infection and Immunity.

[30]  C. Szabó,et al.  Bacterial Invasion Is Not Required for Activation of NF-κB in Enterocytes , 1999, Infection and Immunity.

[31]  Emma Allen-Vercoe,et al.  Lack of flagella disadvantages Salmonella enterica serovar Enteritidis during the early stages of infection in the rat. , 2003, Journal of medical microbiology.

[32]  C. Szabó,et al.  Bacterial invasion is not required for activation of NF-kappaB in enterocytes. , 1999, Infection and immunity.