Influence of renal function on the hemodynamic and clinical responses to long-term captopril therapy in severe chronic heart failure.
暂无分享,去创建一个
[1] M. Packer,et al. Hemodynamic and clinical significance of the pulmonary vascular response to long-term captopril therapy in patients with severe chronic heart failure. , 1985, Journal of the American College of Cardiology.
[2] P. Poole‐Wilson,et al. Clinical importance of the renin-angiotensin system in chronic heart failure: double blind comparison of captopril and prazosin. , 1985, British medical journal.
[3] B. W. East,et al. Captopril in heart failure. A double blind controlled trial. , 1984, British heart journal.
[4] M. Packer,et al. Efficacy of captopril in low-renin congestive heart failure: importance of sustained reactive hyperreninemia in distinguishing responders from nonresponders. , 1984, The American journal of cardiology.
[5] P. Hall,et al. Inaccuracy of estimated creatinine clearance for prediction of iothalamate glomerular filtration rate. , 1984, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[6] N. Hollenberg,et al. Renal response to captopril in severe heart failure: role of furosemide in natriuresis and reversal of hyponatremia. , 1984, Annals of internal medicine.
[7] M. Packer,et al. Prostaglandins in severe congestive heart failure. Relation to activation of the renin--angiotensin system and hyponatremia. , 1984, The New England journal of medicine.
[8] M. Packer. Vasodilator and inotropic therapy for severe chronic heart failure: passion and skepticism. , 1983, Journal of the American College of Cardiology.
[9] C. Ronco,et al. Continuous Arteriovenous Hemofiltration in the Critically Ill Patient: Clinical Use and Operational Characteristics , 1983 .
[10] B. Massie,et al. Controlled Trial of Captopril in Chronic Heart Failure: A Rest and Exercise Hemodynamic Study , 1983, Circulation.
[11] N. Madias,et al. Captopril-induced functional renal insufficiency in patients with bilateral renal-artery stenoses or renal-artery stenosis in a solitary kidney. , 1983, The New England journal of medicine.
[12] J. Kassirer,et al. How to 'interpret' azotemia in cardiac failure , 1983 .
[13] P. Cannon,et al. The effect of captopril on renal, coronary, and systemic hemodynamics in patients with severe congestive heart failure. , 1982, American heart journal.
[14] J. Bauer,et al. Clinical appraisal of creatinine clearance as a measurement of glomerular filtration rate. , 1982, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[15] R. Tarazi,et al. Diuretic potency of combined hydrochlorothiazide and furosemide therapy in patients with azotemia. , 1982, The American journal of medicine.
[16] G. Williams,et al. Angiotensin-converting enzyme inhibition and prostaglandins. , 1982, The American journal of cardiology.
[17] J. Cohn,et al. Effect of captopril on renal function in patients with congestive heart failure. , 1981, British heart journal.
[18] J. Halperin,et al. Acute Regional Circulatory and Renal Hemodynamic Effects of Converting‐enzyme Inhibition in Patients with Congestive Heart Failure , 1981, Circulation.
[19] K. Iseki,et al. Blood Concentration and Urinary Excretion of Captopril (SQ 14,225) in Patients with Chronic Renal Failure , 1981, Hypertension.
[20] N. Reichek,et al. Effect of diuresis on the performance of the failing left ventricle in man. , 1981, The American journal of medicine.
[21] J. Cohn,et al. Acute and Long‐term Response to an Oral Converting‐enzyme Inhibitor, Captopril, in Congestive Heart Failure , 1980, Circulation.
[22] N. Hollenberg,et al. Converting Enzyme Inhibition and the Kidney , 1980, Hypertension.
[23] N. Hollenberg,et al. Sustained effectiveness of converting-enzyme inhibition in patients with severe congestive heart failure. , 1980, The New England journal of medicine.
[24] B. Brundage,et al. Immediate and Sustained Hemodynamic and Clinical Improvement in Chronic Heart Failure by an Oral Angiotensin‐converting Enzyme Inhibitor , 1980, Circulation.
[25] R. Gorlin,et al. Hemodynamic evaluation of hydralazine dosage in refractory heart failure , 1980, Clinical pharmacology and therapeutics.
[26] Y. Imai,et al. Role of prostaglandin in the antihypertensive mechanism of captopril in low renin hypertension. , 1980, Clinical science.
[27] J. Dirks. Mechanisms of action and clinical uses of diuretics. , 1979, Hospital practice.
[28] J. Laragh,et al. Reactive hyperreninemia in renovascular hypertension after angiotensin blockage with saralasin or converting enzyme inhibitor. , 1979, Annals of internal medicine.
[29] J. H. Stein,et al. Diuretic-induced changes in renal blood flow and prostaglandin E excretion in the dog. , 1979, The American journal of physiology.
[30] H. Thurston,et al. Converting Enzyme Inhibitor and Saralasin Infusion in Rats: Evidence for an Additional Vasodepressor Property of Converting Enzyme Inhibitor , 1978, Circulation research.
[31] T. Lohmeier,et al. Control of glomerular filtration rate by renin-angiotensin system. , 1977, The American journal of physiology.
[32] D. Morgan,et al. Plasma creatinine and urea: creatinine ratio in patients with raised plasma urea. , 1977, British medical journal.
[33] E. Anggard,et al. Reduction by indomethacin of furosemide effects in the rabbit. , 1976, European journal of pharmacology.
[34] Cockcroft Dw,et al. Prediction of Creatinine Clearance from Serum Creatinine , 1976 .
[35] R. G. Muth. Diuretic properties of furosemide in renal disease. , 1968, Annals of internal medicine.
[36] F. Reubi. CLINICAL USE OF FUROSEMIDE , 1966, Annals of the New York Academy of Sciences.
[37] F. Reubi,et al. Effects of Reduced Glomerular Filtration Rate on Responsiveness to Chlorothiazide and Mercurial Diuretics , 1961, Circulation.
[38] J. Thayer. Renal abnormalities in congestive heart failure. , 1960, Stanford medical bulletin.
[39] W. Hollander,et al. The relationship of cardiovascular and renal hemodynamic function to sodium excretion in patients with severe heart disease but without edema. , 1956, The Journal of clinical investigation.
[40] W. Hollander,et al. The effects of exercise on cardiovascular and renal function in cardiac patients with and without heart failure. , 1955, The Journal of clinical investigation.
[41] A. Merrill,et al. EDEMA AND DECREASED RENAL BLOOD FLOW IN PATIENTS WITH CHRONIC CONGESTIVE HEART FAILURE: EVIDENCE OF "FORWARD FAILURE" AS THE PRIMARY CAUSE OF EDEMA. , 1946, The Journal of clinical investigation.