Negative Epistasis between Sickle and Foetal Haemoglobin Suggests a Reduction in Protection against Malaria
暂无分享,去创建一个
R. Snow | N. Geller | J. Makani | B. Mmbando | N. Jeffries | Josephine Mgaya | S. Cox | S. N. Mtatiro | D. Soka | S. Rwezaula | E. Meda | E. Msaki | J. Mgaya
[1] Jane-Jane Chen. Stress-enhanced translation of γ-globin mRNA. , 2014, Blood.
[2] D. Kwiatkowski,et al. Epistasis between the haptoglobin common variant and α+thalassemia influences risk of severe malaria in Kenyan children. , 2014, Blood.
[3] S. Hay,et al. Global Burden of Sickle Cell Anaemia in Children under Five, 2010–2050: Modelling Based on Demographics, Excess Mortality, and Interventions , 2013, PLoS medicine.
[4] Sunetra Gupta,et al. NEGATIVE EPISTASIS BETWEEN α+ THALASSAEMIA AND SICKLE CELL TRAIT CAN EXPLAIN INTERPOPULATION VARIATION IN SOUTH ASIA , 2011, Evolution; international journal of organic evolution.
[5] Tobago. Population and Housing Census. , 2011 .
[6] J. Keefer,et al. A Role for Fetal Hemoglobin and Maternal Immune IgG in Infant Resistance to Plasmodium falciparum Malaria , 2011, PloS one.
[7] F. Kirkham,et al. Mortality in Sickle Cell Anemia in Africa: A Prospective Cohort Study in Tanzania , 2011, PloS one.
[8] J. Haldane,et al. THE RATE OF MUTATION OF HUMAN GENES , 2010 .
[9] Stephen W. Hartley,et al. Fetal hemoglobin in sickle cell anemia: genome-wide association studies suggest a regulatory region in the 5' olfactory receptor gene cluster. , 2010, Blood.
[10] K. Marsh,et al. Malaria in patients with sickle cell anemia: burden, risk factors, and outcome at the outpatient clinic and during hospitalization. , 2010, Blood.
[11] O. Pybus,et al. Epistatic interactions between genetic disorders of hemoglobin can explain why the sickle-cell gene is uncommon in the Mediterranean , 2009, Proceedings of the National Academy of Sciences.
[12] Mario Recker,et al. Negative epistasis between the malaria-protective effects of α+-thalassemia and the sickle cell trait , 2005, Nature Genetics.
[13] Dianne J Terlouw,et al. Protective effects of the sickle cell gene against malaria morbidity and mortality , 2002, The Lancet.
[14] G. Stamatoyannopoulos,et al. Transgenic mice expressing human fetal globin are protected from malaria by a novel mechanism. , 1998, Blood.
[15] J. Craig,et al. Genetics of Hb F/F cell variance in adults and heterocellular hereditary persistence of fetal hemoglobin. , 1998, Hemoglobin.
[16] O. Platt,et al. Mortality in sickle cell disease. Life expectancy and risk factors for early death. , 1994, The New England journal of medicine.
[17] M. Friedman. Oxidant damage mediates variant red cell resistance to malaria , 1979, Nature.
[18] R. Wilson,et al. Effects of foetal haemoglobin on susceptibility of red cells to Plasmodium falciparum , 1977, Nature.
[19] A. Allison,et al. Protection Afforded by Sickle-cell Trait Against Subtertian Malarial Infection , 1954, British medical journal.
[20] G. Stamatoyannopoulos,et al. Fetal hemoglobin in acute and chronic states of erythroid expansion. , 1993, Blood.
[21] L. Luzzatto,et al. Fetal haemoglobin and malaria. , 1976, Lancet.