(cid:1) 1 Integrin Gene Excision in the Adult Murine Cardiac Myocyte Causes Defective Mechanical and Signaling Responses
暂无分享,去创建一个
Yusu Gu | N. Dalton | K. Peterson | S. Israeli | T. Yajima | R. Ross | Ying Wu | Michael S. Huang | A. M. Manso | Ruixia Li | P. Liao | Uyen Nguyen
[1] Karen S. Frese,et al. PINCH Proteins Regulate Cardiac Contractility by Modulating Integrin-Linked Kinase-Protein Kinase B Signaling , 2011, Molecular and Cellular Biology.
[2] M. Penn,et al. Significance of Thymosin β4 and Implication of PINCH-1-ILK-α-Parvin (PIP) Complex in Human Dilated Cardiomyopathy , 2011, PloS one.
[3] D. Kuppuswamy,et al. Integrins Are the Necessary Links to Hypertrophic Growth in Cardiomyocytes , 2011, Journal of signal transduction.
[4] S. Meloche,et al. Extracellular Signal-Regulated Kinases 1 and 2 Regulate the Balance Between Eccentric and Concentric Cardiac Growth , 2011, Circulation research.
[5] J. Molkentin,et al. Molecular pathways underlying cardiac remodeling during pathophysiological stimulation. , 2010, Circulation.
[6] Yibin Wang,et al. Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale. , 2010, Physiological reviews.
[7] D. Srivastava,et al. Cardiac fibroblasts regulate myocardial proliferation through beta1 integrin signaling. , 2009, Developmental cell.
[8] Joel C. Miller,et al. Cardiac-Myocyte-Specific Excision of the Vinculin Gene Disrupts Cellular Junctions, Causing Sudden Death or Dilated Cardiomyopathy , 2007, Molecular and Cellular Biology.
[9] N. Schork,et al. Laminin-&agr;4 and Integrin-Linked Kinase Mutations Cause Human Cardiomyopathy Via Simultaneous Defects in Cardiomyocytes and Endothelial Cells , 2007 .
[10] S. Houser,et al. Loss of β1D-integrin function in human ischemic cardiomyopathy , 2007, Basic Research in Cardiology.
[11] I. Shiojima,et al. Regulation of cardiac growth and coronary angiogenesis by the Akt/PKB signaling pathway. , 2006, Genes & development.
[12] A. Yoshimura,et al. Innate Defense Mechanism Against Virus Infection Within the Cardiac Myocyte Requiring gp130-STAT3 Signaling , 2006, Circulation.
[13] J. Coles,et al. Integrin-Linked Kinase Expression Is Elevated in Human Cardiac Hypertrophy and Induces Hypertrophy in Transgenic Mice , 2006, Circulation.
[14] Anthony J. Muslin,et al. Akt2 Regulates Cardiac Metabolism and Cardiomyocyte Survival* , 2006, Journal of Biological Chemistry.
[15] M. Rojas,et al. Myocyte-Restricted Focal Adhesion Kinase Deletion Attenuates Pressure Overload–Induced Hypertrophy , 2006, Circulation research.
[16] S. Nattel,et al. Targeted ablation of ILK from the murine heart results in dilated cardiomyopathy and spontaneous heart failure. , 2006, Genes & development.
[17] Anthony J. Muslin,et al. Akt1 Is Required for Physiological Cardiac Growth , 2006, Circulation.
[18] Masahiko Hoshijima,et al. Mechanical stress-strain sensors embedded in cardiac cytoskeleton: Z disk, titin, and associated structures. , 2006, American journal of physiology. Heart and circulatory physiology.
[19] S. Verma,et al. Altered Expression of Disintegrin Metalloproteinases and Their Inhibitor in Human Dilated Cardiomyopathy , 2006, Circulation.
[20] J. Guan,et al. Inactivation of focal adhesion kinase in cardiomyocytes promotes eccentric cardiac hypertrophy and fibrosis in mice. , 2005, The Journal of clinical investigation.
[21] J. Miyazaki,et al. Mitogen-Activated Protein Kinase Plays a Critical Role in Cardiomyocyte Survival but Not in Cardiac Hypertrophic Growth in Response to Pressure Overload , 2004 .
[22] R. Ross. Molecular and mechanical synergy: cross-talk between integrins and growth factor receptors. , 2004, Cardiovascular research.
[23] M. Latronico,et al. Regulation of Cell Size and Contractile Function by AKT in Cardiomyocytes , 2004, Annals of the New York Academy of Sciences.
[24] W. Pyle,et al. At the crossroads of myocardial signaling: the role of Z-discs in intracellular signaling and cardiac function. , 2004, Circulation research.
[25] J. Molkentin,et al. Redefining the roles of p38 and JNK signaling in cardiac hypertrophy: dichotomy between cultured myocytes and animal models. , 2003, Journal of molecular and cellular cardiology.
[26] E. Engvall,et al. The new frontier in muscular dystrophy research: booster genes , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[27] Michael D. Schneider,et al. Sizing up the heart: development redux in disease. , 2003, Genes & development.
[28] Timothy E Hewett,et al. Targeted inhibition of p38 MAPK promotes hypertrophic cardiomyopathy through upregulation of calcineurin-NFAT signaling. , 2003, The Journal of clinical investigation.
[29] Attila Kovacs,et al. The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis. , 2003, The Journal of clinical investigation.
[30] C. Pham,et al. Modulation of integrins and integrin signaling molecules in the pressure-loaded murine ventricle , 2002, Histochemistry and Cell Biology.
[31] S. Cook,et al. Phenotypic Spectrum Caused by Transgenic Overexpression of Activated Akt in the Heart* , 2002, The Journal of Biological Chemistry.
[32] S. Cherry,et al. Cardiac Myocyte-Specific Excision of the &bgr;1 Integrin Gene Results in Myocardial Fibrosis and Cardiac Failure , 2002, Circulation research.
[33] E. Olson,et al. Activated glycogen synthase-3β suppresses cardiac hypertrophy in vivo , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[34] D. Kass,et al. The in vivo role of p38 MAP kinases in cardiac remodeling and restrictive cardiomyopathy , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[35] M. Crackower,et al. Temporally Regulated and Tissue-Specific Gene Manipulations in the Adult and Embryonic Heart Using a Tamoxifen-Inducible Cre Protein , 2001, Circulation research.
[36] R. Ross,et al. Integrins and the myocardium. , 2001, Genetic engineering.
[37] C. Pham,et al. Striated muscle-specific beta(1D)-integrin and FAK are involved in cardiac myocyte hypertrophic response pathway. , 2000, American journal of physiology. Heart and circulatory physiology.
[38] R. Hynes. Cell adhesion: old and new questions. , 1999, Trends in cell biology.
[39] D. Erle,et al. Integrin β Cytoplasmic Domains Differentially Bind to Cytoskeletal Proteins* , 1998, The Journal of Biological Chemistry.
[40] I. Nonaka,et al. Altered expression of the alpha7beta1 integrin in human and murine muscular dystrophies. , 1997, Journal of cell science.
[41] John Calvin Reed,et al. Integrins (alpha7beta1) in muscle function and survival. Disrupted expression in merosin-deficient congenital muscular dystrophy. , 1997, The Journal of clinical investigation.
[42] S. Chien,et al. Role of integrins in cellular responses to mechanical stress and adhesion. , 1997, Current opinion in cell biology.
[43] R. Fässler,et al. Consequences of lack of beta 1 integrin gene expression in mice. , 1995, Genes & development.
[44] R. Pedersen,et al. Deletion of beta 1 integrins in mice results in inner cell mass failure and peri-implantation lethality. , 1995, Genes & development.
[45] R. Juliano,et al. Integrin Signaling , 2005, Cancer and Metastasis Reviews.
[46] Luigi Fratta,et al. Melusin, a muscle-specific integrin β1–interacting protein, is required to prevent cardiac failure in response to chronic pressure overload , 2003, Nature Medicine.
[47] E. Olson,et al. Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo. , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[48] G. Dorn,et al. Cytoplasmic signaling pathways that regulate cardiac hypertrophy. , 2001, Annual review of physiology.
[49] E. Hoffman,et al. Mutations in the integrin alpha7 gene cause congenital myopathy. , 1998, Nature genetics.
[50] J. Sadoshima,et al. Mechanotransduction in stretch-induced hypertrophy of cardiac myocytes. , 1993, Journal of receptor research.