A model of quiescent tumour microregions for evaluating multicellular resistance to chemotherapeutic drugs
暂无分享,去创建一个
[1] H. Riedel. Models for Tumour Growth and Differentiation , 2007 .
[2] E. Lam,et al. Cell proliferation and cell death , 2006 .
[3] G. Patton,et al. Cell-cycle dependence of foamy virus vectors. , 2004, The Journal of general virology.
[4] J. Pinski,et al. A new screening system for proliferation-independent anti-cancer agents. , 2004, Cancer letters.
[5] L. Gianni,et al. Anthracyclines: Molecular Advances and Pharmacologic Developments in Antitumor Activity and Cardiotoxicity , 2004, Pharmacological Reviews.
[6] M. Jordan,et al. Microtubules as a target for anticancer drugs , 2004, Nature Reviews Cancer.
[7] Z. Siddik,et al. Cisplatin: mode of cytotoxic action and molecular basis of resistance , 2003, Oncogene.
[8] B. Kaina. DNA damage-triggered apoptosis: critical role of DNA repair, double-strand breaks, cell proliferation and signaling. , 2003, Biochemical pharmacology.
[9] J. Carroll,et al. p27Kip1 Induces Quiescence and Growth Factor Insensitivity in Tamoxifen-treated Breast Cancer Cells , 2003 .
[10] P. Johnston,et al. 5-Fluorouracil: mechanisms of action and clinical strategies , 2003, Nature Reviews Cancer.
[11] H. Nagawa,et al. Lysophosphatidic acid (LPA) enhances the metastatic potential of human colon carcinoma DLD1 cells through LPA1. , 2003, Cancer research.
[12] C. Cordon-Cardo,et al. p27 as a target for cancer therapeutics. , 2003, Cancer cell.
[13] S. Okada,et al. Mitotic index and ki-67 nuclear antigen labeling index as predictors of chemotherapy response in uterine cervical carcinoma. , 2001, Gynecologic Oncology.
[14] Chi V. Dang,et al. Hypoxia Inhibits G1/S Transition through Regulation of p27 Expression* , 2001, The Journal of Biological Chemistry.
[15] M. Shah,et al. The relevance of drug sequence in combination chemotherapy. , 2000, Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy.
[16] J. Jardillier,et al. Multicellular resistance: a paradigm for clinical resistance? , 2000, Critical reviews in oncology/hematology.
[17] J. Gerdes,et al. The Ki-67 protein: fascinating forms and an unknown function. , 2000, Experimental cell research.
[18] Johannes Gerdes,et al. The Ki‐67 protein: From the known and the unknown , 2000, Journal of cellular physiology.
[19] R. Poon,et al. Differential responses of proliferating versus quiescent cells to adriamycin. , 1999, Experimental cell research.
[20] K. Ono,et al. Potentially lethal damage repair by total and quiescent tumor cells following various DNA-damaging treatments. , 1999, Radiation medicine.
[21] D. Gewirtz,et al. A critical evaluation of the mechanisms of action proposed for the antitumor effects of the anthracycline antibiotics adriamycin and daunorubicin. , 1999, Biochemical pharmacology.
[22] J. Cheville,et al. p27kip1: a multifunctional cyclin-dependent kinase inhibitor with prognostic significance in human cancers. , 1999, The American journal of pathology.
[23] D. Siemann. The tumor microenvironment: a double-edged sword. , 1998, International journal of radiation oncology, biology, physics.
[24] G Hamilton,et al. Multicellular spheroids as an in vitro tumor model. , 1998, Cancer letters.
[25] A. Giaccia,et al. The unique physiology of solid tumors: opportunities (and problems) for cancer therapy. , 1998, Cancer research.
[26] R. Kerbel,et al. Impact of the cyclin–dependent kinase inhibitor p27Kip1 on resistance of tumor cells to anticancer agents , 1996, Nature Medicine.
[27] S. Lippard,et al. Cisplatin and DNA repair in cancer chemotherapy. , 1995, Trends in biochemical sciences.
[28] K. Franssila,et al. Do DNA ploidy and S-phase fraction in primary tumour predict the response to chemotherapy in metastatic breast cancer? , 1995, British Journal of Cancer.
[29] R W Veltri,et al. Implication of cell kinetic changes during the progression of human prostatic cancer. , 1995, Clinical cancer research : an official journal of the American Association for Cancer Research.
[30] Tony Hunter,et al. p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21 , 1994, Cell.
[31] H. Magdelenat,et al. Prognostic Value of the S‐Phase Fraction of Breast Cancers Treated by Primary Radiotherapy or Neoadjuvant Chemotherapy , 1993, Annals of the New York Academy of Sciences.
[32] T. H. van der Kwast,et al. Monoclonal antibody Ki-67 defined growth fraction in benign prostatic hyperplasia and prostatic cancer. , 1989, The Journal of urology.
[33] M. Rousset,et al. Growth-related glycogen levels of human intestine carcinoma cell lines grown in vitro and in vivo in nude mice. , 1980, Journal of the National Cancer Institute.
[34] L. V. van Putten,et al. Proliferation-dependent cytotoxicity of anticancer agents: a review. , 1975, Cancer research.
[35] Inch Wr,et al. Growth of nodular carcinomas in rodents compared with multi-cell spheroids in tissue culture. , 1970 .
[36] M. Hall,et al. Comparative efficacy of novel platinum(IV) compounds with established chemotherapeutic drugs in solid tumour models. , 2004, Biochemical pharmacology.
[37] T. Petit,et al. Comparative value of tumour grade, hormonal receptors, Ki-67, HER-2 and topoisomerase II alpha status as predictive markers in breast cancer patients treated with neoadjuvant anthracycline-based chemotherapy. , 2004, European journal of cancer.
[38] J. Carroll,et al. p27(Kip1) induces quiescence and growth factor insensitivity in tamoxifen-treated breast cancer cells. , 2003, Cancer research.
[39] Shaun Thomas,et al. Cell Proliferation and Cell Death , 2002 .
[40] M. Clayton,et al. Plateau-phase cultures: an experimental model for identifying drugs which are bioactivated within the microenvironment of solid tumours. , 1997, British Journal of Cancer.
[41] R. Jackson,et al. The problem of the quiescent cancer cell. , 1989, Advances in enzyme regulation.
[42] R. Sutherland,et al. Growth of multicell spheroids in tissue culture as a model of nodular carcinomas. , 1971, Journal of the National Cancer Institute.
[43] R. Sutherland,et al. Growth of nodular carcinomas in rodents compared with multi-cell spheroids in tissue culture. , 1970, Growth.