Oncogenic mutations and chromosomal aberrations in primary extranodal diffuse large B-cell lymphomas of the thyroid--a study of 21 cases.
暂无分享,去创建一个
G. Brabant | N. Gebauer | A. Feller | D. Rades | H. Lehnert | C. Thorns | K. Reddemann | J. Gebauer | J. Knief | V. Bernard | Janina Schemme
[1] N. Gebauer,et al. Prevalence of targetable oncogenic mutations and genomic alterations in Epstein–Barr virus-associated diffuse large B-cell lymphoma of the elderly , 2015, Leukemia & lymphoma.
[2] M. Seto,et al. Array-comparative genomic hybridization profiling of immunohistochemical subgroups of diffuse large B-cell lymphoma shows distinct genomic alterations , 2014, Cancer science.
[3] T. Petrella,et al. Multiple genetic alterations in primary cutaneous large B-cell lymphoma, leg type support a common lymphomagenesis with activated B-cell-like diffuse large B-cell lymphoma , 2014, Modern Pathology.
[4] A. Belkina,et al. The double bromodomain protein Brd2 promotes B cell expansion and mitogenesis , 2014, Journal of leukocyte biology.
[5] Suresh C. Sharma,et al. Survival and Failure Outcomes in Primary Thyroid Lymphomas: A Single Centre Experience of Combined Modality Approach , 2013, Journal of thyroid research.
[6] Xiaoli Zhao,et al. Chromosome abnormalities in diffuse large B‐cell lymphomas: analysis of 231 Chinese patients , 2013, Hematological oncology.
[7] M. Kersten,et al. High prevalence of oncogenic MYD88 and CD79B mutations in diffuse large B-cell lymphomas presenting at immune-privileged sites , 2013, Blood Cancer Journal.
[8] Jung-Ah Hwang,et al. Recent Progress of Genome Study for Anaplastic Thyroid Cancer , 2013, Genomics & informatics.
[9] N. Gebauer,et al. MicroRNA Expression and JAK2 Allele Burden in Bone Marrow Trephine Biopsies of Polycythemia Vera, Essential Thrombocythemia and Early Primary Myelofibrosis , 2013, Acta Haematologica.
[10] J. Dowell,et al. Vemurafenib: targeted inhibition of mutated BRAF for treatment of advanced melanoma and its potential in other malignancies. , 2012, Drugs.
[11] P. Sindou,et al. Differential Expression of Neurotensin and Specific Receptors, NTSR1 and NTSR2, in Normal and Malignant Human B Lymphocytes , 2012, The Journal of Immunology.
[12] M. Reed,et al. High Quality Genomic Copy Number Data from Archival Formalin-Fixed Paraffin-Embedded Leiomyosarcoma: Optimisation of Universal Linkage System Labelling , 2012, PloS one.
[13] K. Dybkær,et al. Comprehensive gene expression profiling and immunohistochemical studies support application of immunophenotypic algorithm for molecular subtype classification in diffuse large B-cell lymphoma: a report from the International DLBCL Rituximab-CHOP Consortium Program Study , 2012, Leukemia.
[14] R. Siebert,et al. Recurrent deletions of the TNFSF7 and TNFSF9 genes in 19p13.3 in diffuse large B‐cell and Burkitt lymphomas , 2012, International journal of cancer.
[15] K. Uzawa,et al. Overexpression of cell cycle regulator CDCA3 promotes oral cancer progression by enhancing cell proliferation with prevention of G1 phase arrest , 2012, BMC Cancer.
[16] S. Swerdlow,et al. THYROID CARCINOMA-ASSOCIATED GENETIC MUTATIONS ALSO OCCUR IN THYROID LYMPHOMAS , 2012, Modern Pathology.
[17] Y. Nikiforov. Molecular diagnostics of thyroid tumors. , 2011, Archives of pathology & laboratory medicine.
[18] M. Kami,et al. Clinicopathological features of 171 cases of primary thyroid lymphoma: a long‐term study involving 24 553 patients with Hashimoto’s disease , 2011, British journal of haematology.
[19] Joseph M. Connors,et al. Oncogenically active MYD88 mutations in human lymphoma , 2011, Nature.
[20] Jan Delabie,et al. Chronic active B-cell-receptor signalling in diffuse large B-cell lymphoma , 2010, Nature.
[21] S. Roman,et al. Prognosis of primary thyroid lymphoma: demographic, clinical, and pathologic predictors of survival in 1,408 cases. , 2009, Surgery.
[22] L. Staudt,et al. Chromosomal alterations detected by comparative genomic hybridization in subgroups of gene expression-defined Burkitt’s lymphoma , 2008, Haematologica.
[23] R. Domingues,et al. Comparative genomic hybridization, BRAF, RAS, RET, and oligo-array analysis in aneuploid papillary thyroid carcinomas. , 2007, Oncology reports.
[24] C. Rouveirol,et al. Computation of recurrent minimal genomic alterations from array-CGH data , 2006, Bioinform..
[25] E. Jordanova,et al. Array-based comparative genomic hybridization analysis reveals recurrent chromosomal alterations and prognostic parameters in primary cutaneous large B-cell lymphoma. , 2006, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[26] L. Staudt,et al. Diffuse large B-cell lymphoma subgroups have distinct genetic profiles that influence tumor biology and improve gene-expression-based survival prediction. , 2005, Blood.
[27] J. Pasieka,et al. Primary thyroid lymphomas , 2004, Current treatment options in oncology.
[28] R. Siebert,et al. Homogeneous immunophenotype and paucity of secondary genomic aberrations are distinctive features of endemic but not of sporadic Burkitt's lymphoma and diffuse large B‐cell lymphoma with MYC rearrangement , 2004, The Journal of pathology.
[29] Göran Roos,et al. Chromosomal Imbalances in Diffuse Large B-Cell Lymphoma Detected by Comparative Genomic Hybridization , 2002, Modern Pathology.
[30] A. Nicholson,et al. Mutations of the BRAF gene in human cancer , 2002, Nature.
[31] L. Thompson,et al. Malignant lymphoma of the thyroid gland: a clinicopathologic study of 108 cases. , 2000, The American journal of surgical pathology.
[32] J Hermans,et al. Clinical relevance of BCL2, BCL6, and MYC rearrangements in diffuse large B-cell lymphoma. , 1998, Blood.
[33] H. Gogas,et al. T-cell lymphoma in Hashimoto's thyroiditis. , 1996, Histopathology.
[34] T. Suchi,et al. Chronic thyroiditis as a risk factor of B-cell lymphoma in the thyroid gland. , 1985, Japanese journal of cancer research : Gann.
[35] H. Blomgren,et al. Cancer risks in patients with chronic lymphocytic thyroiditis. , 1985, The New England journal of medicine.
[36] Y. Oda,et al. NOTCH4 is a potential therapeutic target for triple-negative breast cancer. , 2014, Anticancer research.
[37] O. Sheils,et al. Low-level genomic instability is a feature of papillary thyroid carcinoma: an array comparative genomic hybridization study of laser capture microdissected papillary thyroid carcinoma tumors and clonal cell lines. , 2007, Archives of pathology & laboratory medicine.
[38] L. Staudt,et al. Confirmation of the molecular classification of diffuse large B-cell lymphoma by immunohistochemistry using a tissue microarray. , 2004, Blood.