Regional sensitivity of primate brain dopaminergic neurons to haloperidol: alterations following chronic treatment

[1]  A. Carlsson,et al.  EFFECT OF CHLORPROMAZINE OR HALOPERIDOL ON FORMATION OF 3METHOXYTYRAMINE AND NORMETANEPHRINE IN MOUSE BRAIN. , 2009, Acta pharmacologica et toxicologica.

[2]  R. Roth,et al.  Regulation of tyrosine hydroxylase from human pheochromocytoma, bovine adrenal and rat striatum. , 1978, Life sciences.

[3]  A Carlsson,et al.  Antipsychotic drugs, neurotransmitters, and schizophrenia. , 1978, The American journal of psychiatry.

[4]  W. Lovenberg,et al.  Haloperidol: effect of long-term treatment on rat striatal dopamine synthesis and turnover. , 1977, Science.

[5]  H. Vanpraag The significance of dopamine for the mode of action of neuroleptics and the pathogenesis of schizophrenia. , 1977 .

[6]  S. Snyder,et al.  Antischizophrenic drugs: chronic treatment elevates dopamine receptor binding in brain. , 1977, Science.

[7]  R. Roth,et al.  Dihydroxyphenylacetic acid conjugate: natural occurrence and demonstration of probenecid-induced accumulation in rat striatum, olfactory tubercles and frontal cortex. , 1977, European journal of pharmacology.

[8]  J. Hyttel Levels of HVA and DOPAC in mouse corpus striatum in the supersensitivity phase after neuroleptic treatment , 1977, Journal of neurochemistry.

[9]  J. Glowinski,et al.  Dopamine metabolism in the mesolimbic and mesocortical dopaminergic systems after single or repeated administrations of neuroleptics , 1976, Brain Research.

[10]  S H Snyder,et al.  Dopamine receptor binding predicts clinical and pharmacological potencies of antischizophrenic drugs , 1976, Science.

[11]  A. Guidotti,et al.  Effect of thioridazine, clozapine and other antipsychotics on the kinetic state of tyrosine hydroxylase and on the turnover rate of dopamine in striatum and nucleus accumbens. , 1975, The Journal of pharmacology and experimental therapeutics.

[12]  R. Roth,et al.  Dopaminergic neurons: effect of antipsychotic drugs and amphetamine on single cell activity. , 1973, The Journal of pharmacology and experimental therapeutics.

[13]  A. Faurbye The structural and biochemical basis of movement disorders in treatment with neuroleptic drugs and in extrapyramidal diseases. , 1970, Comprehensive psychiatry.

[14]  G. Sedvall,et al.  Effect of chlorpromazine on accumulation and disappearance of catecholamines formed from tyrosine-C14 in brain. , 1968, The Journal of pharmacology and experimental therapeutics.

[15]  B. Hofstee On the evaluation of the constants Vm and KM in enzyme reactions. , 1952, Science.